Ca和Sr对AZ91D镁合金低周疲劳性能的影响

研究了合金元素Ca、Sr复合添加对金属型铸造镁合金AZ91D的低周疲劳性能的影响。通过运用控制总应变幅0.05 mm～0.25 mm范围内,在应力比R=-1条件下施加拉-压载荷,测试不同状态下合金的低周疲劳性能,并利用光学电子显微镜观察了材料在不同状态下的显微组织及低周疲劳断口特征。结果表明:Ca、Sr元素的添加能细化铸态AZ91D镁合金的晶粒,复合加入0.4%Ca和0.3%Sr,可以提高镁合金的低周疲劳性能。     

高温烧结羟基磷灰石靶的细胞相容性

为研究高温烧结羟基磷灰石靶的细胞相容性,把纯度为99%的羟基磷灰石粉末压制成圆盘形靶,在氩气中在800℃高温下烧结成型。用X射线衍射仪和X射线光电子能谱仪对羟基磷灰石靶的结构组成进行检测。用四氮唑盐比色法对羟基磷灰石靶的细胞相容性进行检测。检测结果表明：纯度为99%的羟基磷灰石粉末在压制成型和800℃高温烧结的过程中化学组成没有发生变化,羟基磷灰石靶中的平均粒径为78.2nm、结晶度为89.4%、Ca/P比值为1.592,具有很好的细胞相容性。     

烧结温度对羟基磷灰石靶的影响

为研究烧结温度对羟基磷灰石靶的影响规律,把纯度为99%的羟基磷灰石粉末压制成圆盘形靶后,在氩气中分别在600℃、700℃和800℃高温下进行烧结。用X射线衍射仪和X射线光电子能谱仪对未烧结和不同温度下烧结的羟基磷灰石靶进行检测。检测结果表明：烧结温度对羟基磷灰石靶的结晶度和组成元素没有显著影响。但是随着烧结温度的升高,羟基磷灰石靶的平均粒径和钙磷比值都增大。800℃高温烧结的羟基磷灰石靶将具有更好的生物相容性、生物活性和稳定性。     

Thermodynamic Evaluation of Inclusions Formation and Behaviour in Steels during Casting and Solidification

For demanding applications high steel cleanliness and strictly controlled inclusions are required. Primary inclusions are formed during steel treatments in the ladle. Most of these are removed to the ladle slag or on the lining. However, the rest of the inclusions still remain through the successive process stages, and additionally new inclusions are formed during casting and solidification due to decreasing thermodynamic solubility of oxygen in the steel at lower temperatures, reactions with surrounding slag, refractory materials and eventual contact with air. Inclusions formation and transformation are simulated by thermodynamic calculations in the steel/inclusions/slag system taking into account the solidification phenomena. In this paper inclusions in Si/Mn‐deoxidised steel and Al‐deoxidised Ca‐treated steels are contributed. Calculations are compared with experimental results from steel plants.     

微量Ca对Cu-Ca合金显微组织和性能的影响

在ZG-0025型的真空中频炉中制备了Ca质量分数分别为0.19％,0.56％,0.69％,0.87％,1.31％的Cu-Ca合金试样；采用光学显微镜、X射线衍射仪、HV-1000A显微硬度计和涡流导电仪分析了不同Ca含量的Cu-Ca合金显微组织和性能.结果表明:随Ca含量的增加,Cu5Ca数量增多,合金导电率下降,显微硬度增大.     

钙灰中CaO含量降低原因分析探究

冶炼用钙灰在存放期间,由于暴露在空气中,钙灰堆表面钙灰与空气接触,吸收空气中的水分消化从而影响钙灰的质量。简要分析钙灰中CaO含量降低原因。     

CaF2对Mg-12%Li-1%Al合金组织和性能的影响

试验熔制了Mg-12%Li-1%Al合金,研究了添加少量的CaF2对合金组织和性能的影响。通过显微观察可知,被还原元素Ca对显微组织中的β相(bcc)有细化作用,Ca主要富集在晶界处,合金中的晶界网格为Ca和Al的富集物,为Al2Ca化合物。显微组织细化机理可以认为,由于Ca在晶界处的吸附作用极大的抑制了晶粒的长大而使其细化。室温下进行轧制板材拉伸测试,结果表明,置换出来的元素Ca可以提高合金的强度和塑性。     

Effect of silica fume on cement hydration and temperature rise of concrete in tropical environment

Investigated herein is the effect of temperature on heat development in cement pastes and concretes with and without silica fume cured at relatively high temperatures often encountered in tropical environment. With an initial temperature of 30°C, adiabatic temperature rise of the concrete with 8% silica fume as cement replacement was similar to that of the control Portland cement concrete up to about 18 h. After 24 h, however, the temperature of the silica fume concrete was lower than that of the control concrete. Since the concrete with 8% silica fume had a higher 28-day compressive strength (72.5 MPa) than the control concrete without silica fume (59.2 MPa), the concrete with silica fume is likely to have a lower temperature rise as compared with the control concrete of equivalent 28-day strength by reducing cementitious materials content with the same water content. The extent of heat evolution in the silica fume pastes was generally greater at lower temperatures of 20–50°C, but less at 65°C than in the control paste. At the relatively high curing temperatures, the degree of cement hydration in the paste with silica fume was lower than that in the control cement paste at early ages. However, the pozzolanic reaction started even before 24 h after water was added.     

Ca3Co4O9陶瓷的固相法制备及分析

本实验采用固相合成法成功地制备了Ca3Co4O9热电材料。通过对样品的显微结构与物相组成等进行分析研究,优化了制备工艺。实验结果表明:由固相法制得的Ca3Co4O9为取向无规则的片状组织;适当提高煅烧温度、延长保温时间有利于片状组织的生长。     

Site-directed mutagenesis of a calcium binding site modifies specifically the different biochemical properties of annexin I

All the functions of annexins in vitro as well as in vivo aremediated and probably regulated by calcium. We have used recombinantannexin I, synthesized by Escherichia coli, and we have performedsite-directed mutagenesis. We have mutated the endonexin foldof domain 2 that binds calcium. Mutations were performed inthis domain of the molecule because it perfectly matches thecalcium binding consensus sequence. The two glycines of thisfold were mutated into glutamic acid. The helix content andthe stability of the mutants are identical to those of the wild-type,suggesting that the mutations did not drastically affect thestructure of the protein. The two mutants showed modified calciumbinding affinities. However, the calcium binding affinity ofthe G131E mutant was far more altered than that of the G129Emutant. Furthermore, other biochemical properties of these mutantswere modified to different extents. The binding to phospholipidwas not seriously affected, whereas the selfassociation waslost by the G131E mutant. In the same way, liposome aggregationis conserved, but modified, while the calcium affinity measuredby equilibrium dialysis is dramatically altered.