Adult age differences in task switching.

Age differences in 2 components of task-set switching speed were investigated in 118 adults aged 20 to 80 years using task-set homogeneous (e.g., AAAA…) and task-set heterogeneous (e.g., AABBAABB…) blocks. General switch costs were defined as latency differences between heterogeneous and homogeneous blocks, whereas specific switch costs were defined as differences between switch and nonswitch trials within heterogeneous blocks. Both types of costs generalized over verbal, figural, and numeric stimulus materials; were more highly correlated to fluid than to crystallized abilities; and were not eliminated after 6 sessions of practice, indicating that they reflect basic and domain-general aspects of cognitive control. Most important, age-associated increments in costs were significantly greater for general than for specific switch costs, suggesting that the ability to efficiently maintain and coordinate 2 alternating task sets in working memory instead of 1 is more negatively affected by advancing age than the ability to execute the task switch itself. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Aging and symptoms of anxiety and depression: Structural invariance of the tripartite model.

Negative affect measures were evaluated in a cross-sectional community sample of adults aged 18-93 (N = 335) to examine the structure of neuroticism, anxiety, and depressive symptoms in young, middle, and older adult cohorts. Structural equation modeling was used to contrast 3 nested models: a 1-factor general distress model; a 2-factor high negative-low positive affect model; and a 3-factor "tripartite model" reflecting a higher order Negative Affect factor that is common to depression and anxiety problems and 2 lower order factors, Low Positive Affect (mostly specific to depression) and Arousal (specific to anxiety/panic). As expected, the tripartite model fit best for all age groups. Further, multigroup analyses indicated age invariance for the tripartite model, suggesting the model can be effectively applied with older populations. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Thirty years of research on race differences in cognitive ability.

The culture-only (0% genetic-100% environmental) and the hereditarian (50% genetic-50% environmental) models of the causes of mean Black-White differences in cognitive ability are compared and contrasted across 10 categories of evidence: the worldwide distribution of test scores, g factor of mental ability, heritability, brain size and cognitive ability, transracial adoption, racial admixture, regression, related life-history traits, human origins research, and hypothesized environmental variables. The new evidence reviewed here points to some genetic component in Black-White differences in mean IQ. The implication for public policy is that the discrimination model (i.e., Black-White differences in socially valued outcomes will be equal barring discrimination) must be tempered by a distributional model (i.e., Black-White outcomes reflect underlying group characteristics). (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Using confirmatory factor analysis to understand executive control in preschool children: I. Latent structure.

Although many tasks have been developed recently to study executive control in the preschool years, the constructs that underlie performance on these tasks are poorly understood. In particular, it is unclear whether executive control is composed of multiple, separable cognitive abilities (e.g., inhibition and working memory) or whether it is unitary in nature. A sample of 243 normally developing children between 2.3 and 6 years of age completed a battery of age-appropriate executive control tasks. Confirmatory factor analysis was used to compare multiple models of executive control empirically. A single-factor, general model was sufficient to account for the data. Furthermore, the fit of the unitary model was invariant across subgroups of children divided by socioeconomic status or sex. Girls displayed a higher level of latent executive control than boys, and children of higher and lower socioeconomic status did not differ in level. In typically developing preschool children, tasks conceptualized as indexes of working memory and inhibitory control in fact measured a single cognitive ability, despite surface differences between task characteristics. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

A structural equation modeling approach to the multiple memory systems question.

Used linear structural equation modeling to test 2 models of declarative memory. A unitary model was compared with the semantic-episodic theory proposed by E. Tulving (e.g., 1972, 1983). The data consisted of performance scores of 300 Ss on 14 cognitive tests. The results of 2 sets of analyses suggest rejection of the unitary model but not the 2-factor model. Moreover, nested comparisons of the models revealed that the 2-factor model provided a significantly better fit to data than did the unitary model. Results support a division of declarative memory into memory for episodic information and memory for facts. This finding is discussed in terms of (1) different underlying memory systems mediating performance on semantic and episodic tests and (2) differences in the retrieval strategies that are required for accessing semantic and episodic information. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

The cognitive reserve hypothesis: A longitudinal examination of age-associated declines in reasoning and processing speed.

The term cognitive reserve is frequently used to refer to the ubiquitous finding that, during later life, those higher in experiential resources (e.g., education, knowledge) exhibit higher levels of cognitive function. This observation may be the result of either experiential resources playing protective roles with respect to the cognitive declines associated with aging or the persistence of differences in functioning that have existed since earlier adulthood. These possibilities were examined by applying accelerated longitudinal structural equation (growth curve) models to 5-year reasoning and speed data from the no-contact control group (N = 690; age 65–89 years at baseline) of the Advanced Cognitive Training for Independent and Vital Elderly study. Vocabulary knowledge and years of education, as markers of cognitive reserve, were related to levels of cognitive functioning but unrelated to rates of cognitive change, both before and after the (negative) relations between levels and rates were controlled for. These results suggest that cognitive reserve reflects the persistence of earlier differences in cognitive functioning rather than differential rates of age-associated cognitive declines. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Primary aging, secondary aging, and intelligence.

The distinction between primary aging, representing innate maturational processes, and secondary aging, representing the effects of environment and disease (E. W. Busse, 1969), was used to develop a model for the assessment of factors that are associated with age-related individual differences in intelligence. Intelligence was measured by performance on a number of tests that measure cognitive abilities known to decline with age. In a hierarchical multiple regression analysis, primary aging and education but not health explained a significant portion of the variance in fluid intelligence. Chronological age had a residual effect over and above that of primary and secondary aging, indicating that there was remaining age-associated variance unaccounted for in the proposed model. The results suggest that the model of primary and secondary aging is a valid means of operationalizing chronological age. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Selection and socialization of risky drinking during the college transition: The importance of microenvironments associated with specific living units.

Risky drinking among college students differs as a function of living types, with living at Greek houses as a major risk factor. Both self-selection based on prior drinking and socialization through living environments have been shown to account for this association. However, it is not clear whether selection and socialization processes occur as a function of specific living units within living types. Multilevel models using a prospective sample of incoming college students (N = 2,392) demonstrated that (1) precollege drinking based selection into specific living units occurred within both fraternity houses and residence halls (beyond selection into the Greek system in general) and (2) socialization of extremely risky drinking among certain fraternity houses was greater than other houses (beyond greater socialization of living at fraternity houses than residence halls in general). Living unit-level precollege correlates (i.e., college attendance motives and cigarette use) and college correlates (i.e., peer drinking norms and alcohol availability) accounted for most of the selection and socialization effects. These findings highlight the importance of micro-environments associated with specific living units in risky drinking during the college transition. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Testing model nesting and equivalence.

When using existing technology, it can be hard or impossible to determine whether two structural equation models that are being considered may be nested. There is also no routine technology for evaluating whether two very different structural models may be equivalent. A simple nesting and equivalence testing (NET) procedure is proposed that uses random sample and model-reproduced moment matrices to evaluate both model nesting and equivalence. The analysis is “local” rather than “global” in nature, but its use with simulation or bootstrapping can imply global conclusions. Two standard applications of NET are to verify whether or not two proposed models are equivalent and whether a baseline model used in an incremental fit index is appropriately nested. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Distinguishing general and specific personality disorder features and implications for substance dependence comorbidity.

Clinical and population-based samples show high comorbidity between Substance Use Disorders (SUDs) and Axis II Personality Disorders (PDs). However, Axis II disorders are frequently comorbid with each other, and existing research has generally failed to distinguish the extent to which SUD/PD comorbidity is general or specific with respect to both specific types of PDs and specific types of SUDs. We sought to determine whether ostensibly specific comorbid substance dependence-Axis II diagnoses (e.g., alcohol use dependence and borderline personality disorder) are reflective of more pervasive or general personality pathology or whether the comorbidity is specific to individual PDs. Face-to-face interview data from Wave 1 and Wave 2 of the National Epidemiologic Survey on Alcohol and Related Conditions were analyzed. Participants included 34,653 adults living in households in the United States. We used hierarchical factor models to statistically partition general and specific personality disorder dimensions while simultaneously testing for specific PD-substance dependence relations. Results indicated that substance dependence-Axis II comorbidity is characterized by general (pervasive) pathology and by Cluster B PD pathology over and above the relationship to the general PD factor. Further, these relations between PD factors and substance dependence diagnoses appeared to largely account for the comorbidity among substance dependence diagnoses in the younger but not older participants. Our findings suggest that a failure to consider the general PD factor, which we interpret as reflecting interpersonal dysfunction, can lead to potential mischaracterizations of the nature of certain PD and SUD comorbidities. (PsycINFO Database Record (c) 2011 APA, all rights reserved)     

Within-Occasion Intraindividual Variability and Preclinical Diagnostic Status: Is Intraindividual Variability an Indicator of Mild Cognitive Impairment?

Intraindividual variability in cognitive test performance has the potential to be a good marker of preclinical Alzheimer's disease status (S. C. Li & U. Lindenberger, 1999). Using cross-sectional community data from 2,317 individuals aged 60-64 years, the authors of this study found that variability was greater in individuals who met criteria for mild cognitive impairment or aging-associated cognitive decline but not for age-associated memory impairment. Higher variability was associated with lower education and a non-English-speaking background. In contrast to previous findings, variability in this study did not contribute uniquely to meeting criteria for mild cognitive impairment. The reasons for the differences may reside in the authors' method of estimating mean independent variability, the use of an occasion-specific measure, or the relatively younger age of the participants. Follow-up of the cohort in 4 years will yield data on the prospective validity of variability as a risk factor for impairment. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Correction to Rushton and Jensen (2005).

Reports an error in the original article by J. Philippe Rushton and Arthur R. Jensen (Psychology, Public Policy, and Law, 2005[Jun], Vol 11[2], pp. 235-294). An equation on p. 271 was incorrect. In the note to Table 5 on p. 273, a similar equation was incorrect. Finally, on p. 274, the last line of the first paragraph was incorrect. Corrections are published here. (The following abstract of this article originally appeared in record 2005-03637-001.) The culture-only (0% genetic-100% environmental) and the hereditarian (50% genetic-50% environmental) models of the causes of mean Black-White differences in cognitive ability are compared and contrasted across 10 categories of evidence: the worldwide distribution of test scores, g factor of mental ability, heritability, brain size and cognitive ability, transracial adoption, racial admixture, regression, related life-history traits, human origins research, and hypothesized environmental variables. The new evidence reviewed here points to some genetic component in Black-White differences in mean IQ. The implication for public policy is that the discrimination model (i.e., Black-White differences in socially valued outcomes will be equal barring discrimination) must be tempered by a distributional model (i.e., Black-White outcomes reflect underlying group characteristics). (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

A Test of the Tripartite Model of Depression and Anxiety in Older Adult Psychiatric Outpatients.

This study examined the tripartite model of depression and anxiety in 131 psychiatric outpatients, ages 55-87. Confirmatory factor analyses revealed that a 3-factor model provided an adequate fit to the observed data, that the 3-factor model was empirically superior to 1- or 2-factor models, and that the 3-factor structure obtained in the current sample of older adult outpatients converged with that obtained on a separate, younger 'sample. Negative affect was significantly related to depression and anxiety symptoms and syndromes, and positive affect was more highly related to depression than anxiety symptoms and syndromes. Ways for taking into account possible age-associated differences in emotion in older adults and thus improving the conceptual model of anxiety and depression are briefly noted. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Cognitive and sensory declines in old age: Gauging the evidence for a common cause.

Resource accounts of behavioral aging postulate that age-associated impairments within and across intellectual and sensory domains reflect, in part, a common set of senescent alterations in the neurochemistry and neuroanatomy of the aging brain. Hence, these accounts predict sizeable correlations of between-person differences in rates of decline, both within and across intellectual and sensory domains. The authors examined reliability-adjusted variances and covariances in longitudinal change for 8 cognitive measures and for close visual acuity, distant visual acuity, and hearing in 516 participants in the Berlin Aging Study (ages 70 to 103 years at 1st measurement). Up to 6 longitudinal measurements were distributed over up to 13 years. Individual differences in rates of cognitive decline were highly correlated, with a single factor accounting for 60% of the variance in cognitive change. This amount increased to 65% when controlling for age at first measurement, distance to death, and risk of dementia. Contrary to expectations, the correlations between cognitive and sensory declines were only moderate in size, underscoring the need to delineate both domain-general and function-specific mechanisms of behavioral senescence. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Cognitive processes as predictors of word recognition and reading comprehension in learning-disabled and skilled readers: Revisiting the specificity hypothesis.

Examines how cognitive processes interrelate as well as predict learning-disabled (LD) readers' word recognition and reading comprehension performance. Correlations between phonological, orthographic, semantic, metacognitive, and working memory measures with reading performance were examined in LD and skilled readers (aged 8–12 yrs). LD Ss were deficient on all cognitive processes compared with skilled Ss, but these differences do not reflect IQ scores. Reading ability group differences emerged on a component composed primarily of working memory measures (referred to as "g") as well as unique components, suggesting that these differences emerge on both general and specific (modular) processes. G best predicts reading comprehension for both groups, and phonological awareness best predicts skilled Ss' pseudoword reading, whereas g best predicts LD Ss' pseudoword performance. Overall, LD Ss' information processing difficulties were described within a general working memory model that views such children as having difficulty accessing and coordinating both general and specific processes. Results suggest that the cognitive processes that contribute to reading deficits are best understood in the context of their combination with other operations rather than in isolation. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

On the specificity of face cognition compared with general cognitive functioning across adult age.

Face cognition is considered a specific human ability, clearly differentiable from general cognitive functioning. Its specificity is primarily supported by cognitive-experimental and neuroimaging research, but recently also from an individual differences perspective. However, no comprehensive behavioral data are available, which would allow estimating lifespan changes of the covariance structure of face-cognition abilities and general cognitive functioning as well as age-differences in face cognition after accounting for interindividual variability in general cognition. The present study aimed to fill this gap. In an age-heterogeneous (18–82 years) sample of 448 adults, we found no factorial dedifferentiation between face cognition and general cognition. Age-related differences in face memory were still salient after taking into account changes in general cognitive functioning. Face cognition thus remains a specific human ability compared with general cognition, even until old age. We discuss implications for models of cognitive aging and suggest that it is necessary to include more explicitly special social abilities in those models. (PsycINFO Database Record (c) 2011 APA, all rights reserved)     

Biological rhythms as organization and information

The purpose of the current study was to describe four models of cognitive deficit and to outline the statistical hypotheses underlying each model. The four models of cognitive deficit were (a) specific deficit; (b) subgroup deficit; (c) a syndrome dissociation model; and (d) a global function dissociation model. Neuropsychological data are analyzed to examine each of these four models in a sample of mild Multiple Sclerosis (MS) patients. The results suggest that for these subjects and tests, the specific deficit model best fits the data. The results are reviewed initially in the context of MS. There follows a consideration of statistical caveats and finally, general applications of the proposed procedures.     

Cognitive controls and psychopathology.

Studied 90 psychiatric inpatients (nonpsychotic, nonschizophrenic psychotic, schizophrenic) and 35 normal controls to (a) determine whether some of the traditional cognitive controls could be isolated in these patients and (b) evaluate whether various diagnostic groups differed from each other and from normals in terms of these congitive controls. Ss were administered a battery of cognitive control tasks (e.g., the Phillips Scale of Premorbid Adjustment, WAIS, and Rorschach tests). With age, socioeconomic status, and verbal IQ as covariates, the factor structure for the total group was similar to that reported in other studies of cognitive control organization in the normal population. Schizophrenic and other psychiatric patients were not characterized by distinctly different organizations of cognitive controls. Although patients showed specific areas of cognitive dysfunctions, these seemed to be related to degree of disorganization. (41 ref) (PsycINFO Database Record (c) 2010 APA, all rights reserved)