全数控装置在轧机电动压下系统中的应用

阐述了安钢中板厂２３００ｍｍ轧机ＭＩＮＩＳＥＭＩＤ全数控电动压下系统的系统结构，并分析了全数控直流调速系统的使用效果。     

PLC与可编程终端之间的通讯

就马钢二铁厂３号高炉槽下上料系统ＰＬＣ控制系统中ＯＭＲＯＮＣ系列可编程序控制器与ＯＭＲＯＮＮＴ６００Ｍ可编程终端之间的通讯问题，介绍装置的配置，命令及功能，以及ＮＴ６００Ｍ支持工具软件的使用。     

本钢冷轧厂乳化液系统改造

针对本钢冷轧厂ＣＤＣＭ机组乳化液系统使用中，撇污装置、供给泵、磁性过滤装置存在的问题进行了改造。     

高炉过程控制新技术在瑞典律勒欧工厂的发展与应用

ＳＳＡＢＴｕｎｎｐｌａｔＡＢ在律勒欧在两座高炉（Ｍ１和Ｍ２）最近几年大力致力高过程控制新技术的开发研究，例如，安装了炉料分布曲线测量系统。该系统用在大高炉（Ｍ２）炉料分布系统的开发中，铁水沟中铁水温度的连续测量和炉缸液位探测正在评估中，使用这些技术目的在于提前给出关于高炉冶炼过程的状态信息，与此同时，铁水中硅的预测模型，软熔带位置，形状和“崩料警告”也在离线开发和测试中，这些根据新测量技术得来的新     

MXL集散系统在6000m~3n／h制氧机中的应用

ＭＸＬ集散系统在６０００ｍ~３ｎ／ｈ制氧机中的应用韶钢集团公司杨建军本文介绍日本横河公司生产的ＭＸＬ集散系统在韶钢制氧分厂６０００ｍ３ｎ／ｈ制氧机组中的应用及使用过程中应注意的问题。１系统硬件的组成本集散控制系统为日本横河公司生产的ＭＸＬ型，由ＭＯＰ?..     

关于MK—A25·5T自卸卡车制动系统的改进

分析了ＭＫ－Ａ２５．５Ｔ自卸卡车制动系统原理以及目前使用条件，存在的弊端。对该系统提出了改进，经试验运行，效果良好，经济效益十分明显。     

企业信息管理系统的建设道路

本文论述了企业计算机信息管理系统建设的必要性，所面临的问题，大中型企业ＭＩＳ规划思想，ＭＩＳ系统建设实施步骤，并根据国有企业现状和特点，重点分析了其在建设ＭＩＳ系统中可供选择方式，可能遇到的问题及其解决方法等。     

DV—4真空直读光谱仪光学校准系统的调整

安钢从美国ＢＡＩＲＤ公司引进的ＤＶ－４型真空直读光谱仪在使用中发现随着环境温度的变化，分析结果有明显漂移，尤其是Ｍｎ元素漂移现象更为严重，直接影响了炼钢化学成份的控制，通过对该仪器光学校准系统的调整。从根本上解决了这一问题。     

钢铁工业低成本CIMS的构成和发展

将钢铁企业六级自动化系统全部集成可实现全局ＣＩＭＳ，而低成本自动化进一步发展后可以实现低成本ＣＩＭＳ，即将企业关键部分集成的局部ＣＩＭＳ。实现低成本ＣＩＭＳ三级控制部分的优选系统为现场总线系统，实现三级管理部分的优选系统为客户机／服务器系统，可以通过二层计算机网络系统的集成构成企业的低成本ＣＩＭＳ。同时还对我国钢铁工业发展ＣＩＭＳ提出了看法。     

焦化厂的CIMS系统及应用研究

本语文从焦炭及炼焦化学吕生产过程的工艺特点出发，研究了大型焦化厂的ＣＩＭＳ系统及其应用，研究了以效益为目标的焦化厂ＣＩＭＳ系统的结构及软件。指出焦化厂的ＣＩＭＳ系统能否实施中产生显著的效益，其核心技术在于建立以优化的核心的分布工信息处理机制，并通过计算机网络系统来实现分布式的信息集成和信息处理。     

Modulation of mustard toxicity by tacrine

Compounds containing the chloroethyl group are potent inhibitors of DNA synthesis and cell growth. Tacrine, a choline carrier inhibitor, was found to protect both HeLa cells and rat thymocytes against the effects of nitrogen mustard. DNA synthesis was restored from 13 to 71% of the control value and cell viability restored from 27 to 57% of the control value by exposure of the cells to an equimolar concentration of tacrine immediately prior to nitrogen mustard. In contrast, tacrine was unable to significantly protect rat thymocytes against the toxic effects of sulphur mustard. These results have implications for the clinical use of nitrogen mustard.     

Intensity Modulation of Olfactory Acuity.

Acuity is fundamental to sensory systems, establishing the foundation for detectable differences in stimulus quality and consequently shaping animals' sensory capacities. In the olfactory system, which samples intrinsically high-dimensional chemical information, acuity for odor quality is measurable by means of ad hoc dimensions based on behaviorally confirmed sets of sequentially similar odorants. The authors measure olfactory acuity in mice using a rewarded forced-choice odor generalization task and show that mice exhibit greater olfactory acuity in response to higher concentration (1,0 Pa) odorants than to lower concentration (0.01 Pa) odorants. Results suggest that the dynamic modulation of sensory acuity-not necessarily its maximization-is an important component of olfactory processing and reflects the salience of odorant stimuli. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

Modulation of adrenal gap junction expression

To increase our knowledge of the role of peptide hormone stimulation in gap junction protein expression and adrenal cortical cell function, primary rat adrenal cortical cells were treated with adrenocorticotropin, and gap junction proteins were measured. Immunocytochemistry and western blot analysis were used to detect and characterize gap junction type and distribution. The gap junction protein, connexin 43 (alpha 1), was detected. Analysis of six connexin protein types did not reveal gap junction species other than alpha 1. Cells of the inner adrenal cortical zones, zonae fasciculata and reticularis, were demonstrated to have the highest number of gap junctions per cell in the adrenal gland. Adrenal cell cultures enriched for the two inner cortical adrenal zones were established and demonstrated also to express alpha 1 gap junction protein. Adrenocorticotropin (40 mUnits/ml) and dibutyryl cyclic adenosine monophosphate (1 mM) treatments increased alpha 1 gap junction protein levels and decreased cell proliferation rates in the cell cultures. The results are consistent with the hypothesis that gap junction expression can be regulated by adrenocorticotropin acting through the second messenger cyclic adenosine monophosphate. It can be suggested that gap junction expression in the adrenal gland may be under hormonal influence, and that gap junctions serve as passage for movement of molecules involved in control of cell proliferation.     

Modulation of cell calcium signals by mitochondria

It is now clearer and clearer that mitochondria play a role, and perhaps an active role, in cell calcium signalling. The fact that mitochondria can exhibit a Ca2+-induced Ca2+ release (mCICR, Ichas et al. [37]) reinforces this concept and makes the mitochondria an essential element in the relay of Ca2+ wave propagation. It must be emphasized that the modulation of cell Ca2+ signals by mitochondria depends upon their energetic status, thus making mitochondria an essential link between energy metabolism and calcium signalling inside the cell.     

Modulation of hypertensive heart disease by estrogen

During pharmaceutical production the quality of the air in manufacturing areas is monitored by volumetric samplers and settle plates. Settle plates are often assigned a secondary role, their usefulness in predicting airborne contamination of containers not being fully recognised. In this paper criticisms of settle plates are answered and the advantages of settle plates explained. It is concluded that using settle plates is the best method of quantifying the likely airborne microbial contamination of containers filled in pharmaceutical production areas.     

Modulation of macrophage activation by ammonium metavanadate

The molecular basis of the immunotoxic effect of ammonium metavanadate on signal transduction involved in macrophage activation was studied in resident peritoneal macrophages (PEM) and a murine macrophage-like cell line, J774. A fourfold elevation in cytosolic free calcium levels was observed within 10 s following lipopolysaccharide (LPS) stimulation of the non-vanadate-exposed controls both in vitro and in vivo; the levels returned to prestimulation values within 70 s. Exposure to phorbol ester (PMA) did not result in any appreciable change in cytosolic free calcium levels. Compared to untreated controls, treatment with vanadate caused a significant elevation in basal cytosolic calcium levels. Such elevation was not enhanced further by LPS. LPS stimulation of macrophages also resulted in a significant elevation of membrane-associated protein kinase C (PKC) activity, which was, however, inhibited in a dose-dependent manner by vanadate in both in vitro and in vivo studies. Exposure to PMA also resulted in a significant elevation of membrane-associated PKC activity; vanadate treatment at lower levels did not cause downregulation, indicating that vanadate at these levels interfered with the receptor-mediated events rather than the enzyme directly. Vanadate at higher exposure levels inhibited the activity even in PMA-stimulated macrophages. No significant difference occurred in cytosolic PKC activities in control macrophages; vanadate treatment at lower levels resulted in a significant elevation of cytosolic PKC activities following stimulation with LPS or PMA, indicating that vanadate might be interfering with the translocation process.