"Differential involvement of the dorsal anterior cingulate and prelimbic-infralimbic areas of the rodent prefrontal cortex in spatial working memory": Correction to Ragozzino et al. (1998).

Reports an error in "Differential involvement of the dorsal anterior cingulate and prelimbic-infralimbic areas of the rodent prefrontal cortex in spatial working memory" by Michael E. Ragozzino, Spencer Adams and Raymond P. Kesner (Behavioral Neuroscience, 1998[Apr], Vol 112[2], 293-303). Figure 1 (page 295) and Figure 4 (page 299) were printed incorrectly. The corrected figure pages and corresponding captions are provided in the erratum. (The following abstract of the original article appeared in record 1998-01023-003.) The present study examined the effects of quinolinic acid lesions of the dorsal anterior cingulate and prelimbic-infralimbic cortices on spatial working memory and spatial discrimination using go/no-go procedures. All testing occurred in a 12-arm radial maze. In a working memory task, rats were allowed to enter 12 arms for a cereal reward. Three or 4 arms were presented for a 2nd time in a session, which did not result in a reward. In a spatial discrimination task, rats had successive access to 2 different arms. One arm always contained a reward, and the other never contained a reward. Prelimbic-infralimbic lesions impaired spatial working memory but only produced a transient spatial discrimination deficit. Dorsal anterior cingulate lesions did not induce a deficit in either task. These findings suggest that the prelimbic-infralimbic cortices, but not the anterior cingulate cortex, are important in spatial working memory. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

"Entorhinal cortex lesions disrupt the transition between the use of intra- and extramaze cues for navigation in the water maze": Correction to Oswald et al. (2003).

Reports an error in "Entorhinal cortex lesions disrupt the transition between the use of intra- and extramaze cues for navigation in the water maze" by C. J. P. Oswald, D. M. Bannerman, B. K. Yee, J. N. P. Rawlins, R. C. Honey and M. Good (Behavioral Neuroscience, 2003[Jun], Vol 117[3], 588-595). The definitions "Present = intramaze landmark present during Stage 2" and "Absent = intramaze landmark absent during Stage 2" appear incorrectly in the caption to Figure 3. These terms and definitions should appear in the caption to Figure 4. (The following abstract of the original article appeared in record 2003-05069-018.) This study with rats examined the effects of excitotoxic lesions to the entorhinal cortex (EC) and hippocampus (HPC) on using extramaze and intramaze cues to navigate to a hidden platform in a water maze. HPC lesions resulted in a disruption to the use of extramaze cues, but not intramaze cues, whereas EC lesions had no effect on the use of these cues when they were encountered for the fast time. However, prior navigation training in which 1 type of cue was relevant disrupted navigation with the other type in rats with EC lesions. Results show that the EC contributes to the processing of spatial information, but that this contribution is most apparent when there is a conflict between 2 sources of navigational cues in the water maze. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

"Differential effects of posterior septal lesions on dispositional and representational memory": Correction to Thomas and Gash (1986).

Reports an error in "Differential effects of posterior septal lesions on dispositional and representational memory" by Garth J. Thomas and Don M. Gash (Behavioral Neuroscience, 1986[Oct], Vol 100[5], 712-719). A phrase was erroneously deleted from the text. In the seventh paragraph on p. 713, the second sentence should read as follows: Early in training individual differences were great, but by the end of adaptation training, individual differences were quite small and all rats responded at close to asymptotic speeds. (The following abstract of the original article appeared in record 1987-06585-001.) A distinction between 2 classes of memory has been made in terms of the sensory availability of cues at the time of making discriminations that are influenced by past experience. Three tasks objectively defining this distinction were learned in a T-maze by 36 male Long-Evans rats in 3 groups: (a) a delayed non-matching-to-sample (DNMTS) that depended on representational memory; (b) a simple sensory discrimination (SD) that depended on dispositional memory; and (c) a more difficult discrimination that also depended on dispositional memory, called the simultaneous conditional discrimination (SCD). The DNMTS and SD tasks were acquired quickly; the SCD task took many more trials. Posterior septal lesions impaired DNMTS performance but had no effect on retention of tasks that depended on dispositional memory. Results indicate that dispositional and representational memory systems have at least partially distinct anatomical substrates in the brain and that it is the representational and not the conditional aspects of the DNMTS task that are impaired by the septal lesions. (21 ref) (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

"Acquisition of a complex place task in rats with selective ibotenate lesions of hippocampal formation: Combined lesions of subiculum and entorthinal cortex versus hippocampus": Correction.

Reports an error in "Acquisition of a complex place task in rats with selective ibotenate lesions of hippocampal formation: Combined lesions of subiculum and entorhinal cortex versus hippocampus" by John-Paul Bouffard and Leonard E. Jarrard (Behavioral Neuroscience, 1988[Dec], Vol 102[6], 828-834). This article's corresponding plate appears on page 995. The information should read, "Plate A. Photomicrographs of horizontal, cresyl violet stained sections at dorsal, middle, and ventral levels of the brain for an unoperated control rat (left), an animal from the Subiculum + Entorhinal lesion group (middle), and a rat from the hippocampus group (right)." (The following abstract of the original article appeared in record 1989-28756-001.) The effects of isolating the hippocampus from its neocortical inputs and outputs by damaging the deep layers of entorhinal cortex and subiculum were compared with direct removal of the hippocampus using acquisition of a complex radial maze task. A series of eight problems (four out of eight arms being correct) were learned under either massed (45 s) or distributed (10 min) practice conditions, thus varying contextual information. Performance of rats with subiculum/entorhinal cortex lesions was similar to that of controls in all aspects of the radial maze task; whereas animals with hippocampal lesions were impaired on nearly all dependent measures. Although the effects of varying the intertrial interval were generally small, distributed practice did serve to facilitate the performance of hippocampal rats in terms of working memory. These findings are discussed as they related to recent theorizing in the area. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

"Ovarian hormones and cognition in the aged female rat: I. Long-term, but not short-term, ovariectomy enhances spatial performance": Correction to Bimonte-Nelson et al. (2003).

Reports an error in "Ovarian Hormones and Cognition in the Aged Female Rat: I. Long-Term, but Not Short-Term, Ovariectomy Enhances Spatial Performance" by Heather A. Bimonte-Nelson, Rachel S. Singleton, Christopher L. Hunter, Kimber L. Price, Alfred B. Moore and Ann-Charlotte E. Granholm (Behavioral Neuroscience, 2003[Dec], Vol 117[6], 1395-1406). The fifth sentence of the abstract reads, "Aged rats exhibited estradiol and elevated progesterone levels comparable to those of young rats." The sentence should read as follows: "Aged rats exhibited similar estradiol and elevated progesterone levels compared with those of young rats." (The following abstract of the original article appeared in record 2003-10460-025.) Although research suggests that ovariectomy (ovx) is detrimental to spatial cognition in young rats, little work has evaluated the cognitive effects of ovx in aged rats. The authors investigated the effects of ovx in aged rats using the water radial-arm maze. In Study 1, young rats and aged rats receiving ovx 1.5 months before testing outperformed aged rats receiving sham surgery or ovx 21 days before testing. In Study 2, young rats and aged rats receiving ovx 2.0 or 6.0 months before testing outperformed aged sham rats. Aged rats exhibited estradiol and elevated progesterone levels comparable to those of young rats. The findings suggest that 1.5-6.0 months, but not 21 days, of ovx improves spatial memory in aged rats. The hypothesis that long-term ovarian hormone loss is detrimental to spatial memory in aged rats was not supported. The authors hypothesize that removal of elevated progesterone levels is related to the ovx-induced cognitive enhancement. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

"Preexposure and extinction effects of lithium chloride induced taste-potentiated aversions for spatially contiguous auditory food cues in rats": Correction to Ellins and von Kluge (1987).

Reports an error in "Preexposure and extinction effects of lithium chloride induced taste-potentiated aversions for spatially contiguous auditory food cues in rats" by Stuart R. Ellins and Silvia von Kluge (Behavioral Neuroscience, 1987[Apr], Vol 101[2], 164-169). The aforementioned article contains errors in the last paragraph of the Results section, none of which change the results of the experiment. The corrected paragraph sections are provided in the erratum. (The following abstract of the original article appeared in record 1987-24095-001.) Taste potentiated illness-induced aversions for noisy food were studied in Sprague-Dawley rats. The rats ate from receptacles containing salty food and a contiguous tone produced by speakers under the food followed by lithium chloride injections. In preference tests, the rats then avoided noisy food in favor of quiet food followed by extinction and spontaneous recovery of the auditory aversion over repeated nonreinforced trials. Other rats were given either 4 or 10 days of exposure to the noisy food prior to taste-toxicosis treatment. None of these rats subsequently avoided noisy food. The importance of spatial contiguity and methodological variation in associating nongustatory food cues with illness is discussed. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

"Social influences on the selection of a protein-sufficient diet by Norway rats (Rattus norvegicus)": Correction to Beck and Galef (1989).

Reports an error in "Social influences on the selection of a protein-sufficient diet by Norway rats (Rattus norvegicus)" by Matthew Beck and Bennett G. Galef (Journal of Comparative Psychology, 1989[Jun], Vol 103[2], 132-139). There was a misstatement. On page 137, second column, second paragraph, the sentence that begins on line 7 ought to read as follows: Protected t tests revealed that subjects in the Different Food-Same Place Group gained a significantly smaller percentage of body weight than did subjects in each of the other two groups (LSD = .67, both ps 1989-31944-001.) Investigated effects of interactions between naive and knowledgeable rats (Rattus norvegicus) on selection of a nutritionally adequate diet by the naive. We found that during a 7-day test, isolated rats choosing among 4 foods, 3 of which were protein-deficient and 1 of which was protein-rich, failed to learn to prefer the protein-rich diet and lost weight. Conversely, those rats that interacted with conspecifics trained to eat the protein-rich diet developed a strong preference for that diet and thrived. The authors also found that Ss were more strongly influenced in their diet selection by the flavor of the foods eaten by conspecifics than by the locations where conspecifics fed. The results suggest that social influence may be important in development of adaptive patterns of diet choice by rats (or other dietary generalists) that need to find nutritionally adequate diets in demanding environments. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

"Snatching defeat from the jaws of victory: Self-esteem differences in the experience and anticipation of success": Correction to Wood et al (2005).

Reports an error in "Snatching Defeat From the Jaws of Victory: Self-Esteem Differences in the Experience and Anticipation of Success" by Joanne V. Wood, Sara A. Heimpel, Ian R. Newby-Clark and Michael Ross (Journal of Personality and Social Psychology, 2005[Nov], Vol 89[5], 764-780). There are typographical errors in Table 2 (certain values should not have been in bold face). The corrected table is provided in the erratum. (The following abstract of the original article appeared in record 2005-15658-009.) Successes--defined broadly as meeting important standards or receiving positive evaluations--are widely assumed to be enjoyed equally by people with high self-esteem (HSEs) and low self-esteem (LSEs). Three studies examined the contradictory hypothesis that HSEs react more favorably to success than do LSEs and that success brings about certain unfavorable consequences for LSEs. Undergraduate participants reacted to a laboratory-manipulated success (Studies 1 and 2) or imagined highly positive events in the future (Study 3). Self-esteem differences emerged in anxiety, thoughts about the self, and (in Study 3) thoughts about non-self-related aspects of the event. LSEs were more anxious than HSEs after succeeding, success improved HSEs' self-relevant thoughts but not LSEs', and LSEs focused more on success's negative aspects. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

"Self-administration of alcohol before and after a public speaking challenge by individuals with social phobia": Correction to Abrams et al. (2002).

Reports an error in "Self-administration of alcohol before and after a public speaking challenge by individuals with social phobia." by Kenneth Abrams, Matt G. Kushner, Krista Lisdahl Medina and Amanda Voight (Psychology of Addictive Behaviors, 2002[Jun], Vol 16[2], 121-128). On page 121, in the abstract, the penultimate sentence incorrectly reads, “As predicted, participants consumed more alcohol following the anxiety challenge than following the control task; however, the opposite pattern was evidenced for drinking following the 2 activities.” The sentence should read as follows: “As predicted, participants consumed more alcohol following the anxiety challenge than following the control task; however, the opposite pattern was evidenced for drinking preceding the 2 activities.” (The following abstract of the original article appeared in record 2002-01321-005.) K. Abrams, M. Kushner, K. Medina, and A. Voight (2001) showed that alcohol attenuates social anxiety symptoms in socially phobic individuals. This article examines whether social anxiety symptoms can lead to increased alcohol use in this same population. Forty-four individuals with social phobia attended 2 laboratory sessions, spaced 1 week apart, in groups of approximately 10. Participants underwent a social anxiety challenge during 1 session and a control task during the other. Half of the sample self-administered alcohol immediately before, and half immediately after, these 2 activities. As predicted, participants consumed more alcohol following the anxiety challenge than following the control task; however, the opposite pattern was evidenced for drinking following the 2 activities. These findings add to an understanding of why social phobia and alcohol problems tend to co-occur. (PsycINFO Database Record (c) 2010 APA, all rights reserved)     

"Attribution and expressed emotion in the relatives of patients with schizophrenia": Correction to Brewin, MacCarthy, Duda, and Vaughn (1991).

Reports an error in "Attribution and expressed emotion in the relatives of patients with schizophrenia" by Chris R. Brewin, Brigid MacCarthy, Karin Duda and Christine E. Vaughn (Journal of Abnormal Psychology, 1991[Nov], Vol 100[4], 546-554). An incorrect sentence was published. The sentence that ends the fourth paragraph on p. 547 ought to read: A more recent onset of illness would be expected to produce more unstable attributions, and more disturbed behavior (particularly involving violence toward the relative) would be expected to produce attributions that were more internal and personal to the patient but more external and uncontrollable as regards the relative. (The following abstract of the original article appeared in record 1992-12907-001.) Indexes of expressed emotion (EE) in 58 relatives of patients with schizophrenia were related to those relatives' spontaneously expressed causal beliefs about the illness and about related symptoms and behaviors. Relatives made attributions predominantly to factors external, universal, and uncontrollable from their own perspective, and to factors internal, universal, and uncontrollable from the patient's perspective. Low-EE relatives were similar in their attributions to emotionally overinvolved relatives. Compared with these 2 groups, critical and/or hostile relatives made more attributions to factors personal to and controllable by the patient. Subsequent analyses suggested that hostile relatives were further characterized by making more attributions to factors internal to the patient and by making attributions with fewer causal elements. (PsycINFO Database Record (c) 2010 APA, all rights reserved)