UVA防护的重要性及其市场发展

本文阐述了UVA防护的重要性，UVA防晒剂的发展及其配方应用。同时简单介绍了国内外有关UVA防护的评价方法以及法规管理现状。     

防晒化妆品长波紫外线防护效果的仪器评价法

综述了对化妆品防晒效果的评价方法,重点介绍了仪器法对UVA防晒效果评价的研究进展,简述了UVA防晒效果的标识方法,所使用仪器的类型及样品的制备方法.并展望了未来UVA防晒效果评价方法的发展方向.     

防晒化妆品及其未来发展

介绍了紫外线对人体的危害，重点介绍了防晒剂的分类及防晒化妆品的未来发展趋势。     

2009年主要防晒市场及发展趋势

从防晒产品、美黑产品以及晒后修复产品几个方面分析了全球防晒产品市场。同时,分别对美国、欧洲以及拉丁美洲地区的综合市场进行了分析。指出了防晒产品未来的发展趋势。     

发展向好的防晒品市场

消费者对过度日晒的危险性越来越关注,对于紫外线曝晒的认识开始向着对行业有利的方向转变。论述了防晒剂的起源、发展以及市场趋势。指出防晒剂对皮肤健康及美容的贡献也变得越来越重要。     

聚氨酯防水涂料及其市场发展概况

本文论述了聚氨酯防水涂料的产量、市场应用概况,以及其开发动向,品种多样化,结构功能化,生产工艺规模化、系列化、标准化。     

表面活性剂对皮肤的不良反应、评价体系及防护

表面活性剂广泛应用于护肤品和皮肤清洁类产品,但频繁使用或不当接触表面活性剂可导致皮肤刺激、敏感或屏障功能损伤,引起皮肤不良反应.为了皮肤的安全,本文针对表面活性剂作用于皮肤的结构和成分,梳理其作用于皮肤的途径,基于该途径和皮肤症状两个维度建立评价体系,并对减缓表面活性剂刺激的功效成分及其作用原理进行分析总结,为抗敏抗刺...     

间苯二酚的生产、市场及发展

介绍了间苯二酚的合成方法及用途,并对其市场情况进行了分析和预测,建议开发或引进间二异丙苯氧化法生产间苯二酚技术,把间苯二酚的生产推向一个新水平。     

防晒化妆品市场发展新趋势——UVA防护及PA防晒标准相关探讨

国内防晒品发展趋势 随着消费者生活水平的提高，对防晒知识的进一步认识，大多数消费者对防晒产品的购买期提前。另外，消费者以前固有的“只有天热时、太阳大时才注意防晒”的意识逐渐被全天候深层防晒护理所取代。     

橡胶防护蜡的作用机理及影响其防护性能的因素

结合橡胶防护蜡的作用机理说明橡胶制品环境温度、石蜡化学组成、硫化胶交联度、石蜡内部迁移、胶料配合剂和石蜡浓度是直接影响防护蜡性能的因素。指出改性防护蜡是今后橡胶防护蜡的一个发展方向。     

紫外线的防护

对紫外线防护的深入研究进行了综述。描述了有关专业术语，紫外线吸收和屏蔽剂的性能、应用范围、安全性问题、在法规上的禁忌和配方使用，展望了防晒剂未来的发展趋势。     

Preparation and property of poly(ethylene terephthalate) fibers providing ultraviolet radiation protection

Interest in protection against solar ultraviolet radiation (UVR) among the general public in the world has been increasing steadily. Poly(ethylene terephthalate) (PET) was blended with UVR‐protection agents and was spun into modified fibers to provide the property of UVR protection. Investigation of this property using a UV spectrophotometer showed that the modified PET fabrics could be resistant to UVR more than 90% in the UV‐B band. The treatment of aqueous alkali on the surface of the fibers to improve the comfortable feel had little influence on the property of UVR protection. Scanning electron microscopy was employed to observe the surface morphology of the fibers. Also, the modified fibers had good heat insulation property and the mechanical properties of the fibers were measured. © 2003 Wiley Periodicals, Inc. J Appl Polym Sci 88: 1180–1185, 2003     

紫外线对皮肤的光辐射损伤与防护研究

研究了阳光紫外线对皮肤造成的各种光辐损伤，探讨了如何采取积极有效的防护措施，各类防晒剂的应用状况，以及防晒化妆品的发展趋势，并对此进行了详细的论述。     

游泳运动对皮肤及头发的影响与防护

游泳是一项日趋普及的运动项目,而游泳对人体皮肤及头发的影响也越来越引起了人们的关注.综述了游泳运动过程中游泳池中化学物质和紫外线辐射对人体皮肤及头发的影响,并提出了相应的防护对策,进而阐明了开发游泳运动专用化妆品的必要性.     

紫外辐射对皮肤角化细胞的影响及其防护的研究

利用紫外线照射人皮肤角化细胞，采用流式细胞技术观察细胞 进程和细胞凋亡情况，并研究了人参皂甙单体Rg-1和Rb-1对紫外辐射的保护.作用。结果表明一定剂量的紫外线可明显诱导人皮肤角化细胞S期阻滞及细胞凋亡，人参皂甙单体Rg-1和Rb-1能够释放S期阻滞，抑制细胞凋亡，对细胞的紫外辐射损伤具有不同程度的保护作用，为开发相关的抗紫外辐射护肤产品提供理论依据。     

Inflammatory Molecules Associated with Ultraviolet Radiation-Mediated Skin Aging

Skin is the largest and most complex organ in the human body comprised of multiple layers with different types of cells. Different kinds of environmental stressors, for example, ultraviolet radiation (UVR), temperature, air pollutants, smoking, and diet, accelerate skin aging by stimulating inflammatory molecules. Skin aging caused by UVR is characterized by loss of elasticity, fine lines, wrinkles, reduced epidermal and dermal components, increased epidermal permeability, delayed wound healing, and approximately 90% of skin aging. These external factors can cause aging through reactive oxygen species (ROS)-mediated inflammation, as well as aged skin is a source of circulatory inflammatory molecules which accelerate skin aging and cause aging-related diseases. This review article focuses on the inflammatory pathways associated with UVR-mediated skin aging.     

防晒化妆品UVA区效果评价方法的研究

UVA能够穿透表皮直接损伤真皮层，使皮肤老化，纤维变性，诱发皮肤癌，如何评价防晒化妆品在UVA区的防护效果，国内尚缺乏检测方法。文章介绍了体内实验及体外实验的评价方法，体内实验采用MX16黑色素－红斑－指数仪测试人的皮肤受到UVA照射后即时变成的一次性的灰我或棕色的光化学反应。这种着色称为黑化。体外实验为紫外分光光度计测试320nm-400nm的吸光度值。两个方法显示了较好的相关性，体外方法具有操作简便，快速和重复性好等特点，可以代替代内方法。     

Type I Interferons Enhance the Repair of Ultraviolet Radiation-Induced DNA Damage and Regulate Cutaneous Immune Suppression

Type I interferons (IFNs) are important enhancers of immune responses which are downregulated in human cancers, including skin cancer. Solar ultraviolet (UV) B radiation is a proven environmental carcinogen, and its exposure contributes to the high prevalence of skin cancer. The carcinogenic effects of UV light can be attributed to the formation of cyclobutane pyrimidine dimers (CPD) and errors in the repair and replication of DNA. Treatment with a single dose of UVB (100 mJ/cm²) upregulated IFNα and IFNβ in the skin of C57BL/6 mice. IFNα and IFNβ were predominantly produced by CD11b+ cells. In mice lacking the type I IFN receptor 1 (IFNAR1), the repair of CPD following cutaneous exposure to a single dose of UVB (100 mJ/cm²) was decreased. UVB induced the expression of the DNA repair gene xeroderma pigmentosum A (XPA) in wild-type (WT) mice. In contrast, such treatment in IFNAR1 (IFNAR1-/-) mice downregulated XPA. A local UVB regimen consisting of UVB radiation (150 mJ/cm²) for 4 days followed by sensitization with hapten 2,4, dinitrofluorobenzene (DNFB) resulted in significant suppression of immune responses in both WT and IFNAR1-/- mice. However, there were significantly higher CD4+CD25+Foxp3+ regulatory T-cells in the draining lymph nodes of IFNAR1-/- mice in comparison to WT mice. Overall, our studies reveal a previously unknown action of type I IFNs in the repair of photodamage and the prevention of UVB-induced immune suppression.