无油润滑压缩机活塞环和支承环设计

通过对填充聚四氟乙烯材料活塞环及带卸荷槽结构支承环的研究 ,确定了活塞及活塞环结构 ,建立了带卸荷槽支承环的力学分析模型 ,并将其结果用于结构尺寸的确定。在压缩机工艺指标不变的情况下 ,用该方法设计的活塞环和支承环性能优良。     

一种新的TDRSS扩频调制方式的研究

本文讨论跟踪及数字中继卫星系统（TDRSS）一种新通信体制，即并行MSK扩频系统，发射端采用长短码正交扩频的平衡调制，接收端采用一种简单解扩方式简化了并行MSK信号的解调，同时用短码全“1”引导长码同步的方法大大减小了长码的捕获时间，实验结果证明了良好的伪码同步性能。     

金刚石膜的计算机虚拟制备技术中的分子动力学模拟

综述了近年来金刚石薄膜形成过程的分子动力学（Molecular Dynamics，简称MD）模拟研究，详细地阐述了原子间相互作用势的选取，总结了不同沉积条件下MD的计算模型和几种典型情况下的模拟结果。研究表明：在原子尺度上，MD方法能较全面地提供有关膜生长的信息，对进一步了解金刚石膜形成的微观机制以及为细观层次仿真提供基本信息均具有重要意义。     

引线框架材料QFe2.5加工工艺研究

本文介绍引线框架材料的发展现状，讨论引线框架用铜合金的强化机理，童点阐述引线框架用QFe2．5铜合金带材的性能及主要生产工艺（铸锭加热、热轧、时效退火等）。     

佛山有线电视网络呼叫中心建设规划

介绍佛山有线电视网络呼叫中心基础平台的建设情况以及呼叫中心系统的发展规划,佛山有线电视呼叫中心采用先进的IP技术建立起呼叫中心的基本平台,在此基础平台上,通过建立先进的多媒体业务平台,佛山有线电视呼叫中心最终将发展成为佛山有线电视网络的服务支持平台、数据收集平台和业务销售平台。     

基于T0944的新型平板显示接口卡的设计

介绍了平板显示控制芯片TO944的特点、功能、原理，并给出了基于T0944芯片的新型平板显示接口卡的软硬件实现方案，该方案具有电路简单、高可靠性、高性价比、高显示质量等优点，可用于各种不同类型与不同尺寸的平板显示器的显示。     

Immunogenic potential of glanders and melioidosis agents

Unlike the glanders agent, the superficial structures of the melioidosis agent were demonstrated to be responsible for marked was immunosuppressive activity. Some antigenic fractions suppressing the blast transformation of lymphocytes, reducing the count of T helpers and profoundly potentiating the infection in vivo were isolated from P. pseudomallei cells. The immunogenic and immunosuppressive activities of both agents' superficial structures were studied by high performance chromatography. Antigenic complexes that were able to protect immunized laboratory animals against fatal infections and to prevent bacterial carriage due to the activation of T cells and to the bacterial activity of macrophages were identified. A composition comprising several immunogens was found to provide an additive protective action against both causative agents. Therefore, the composition may be considered to be a prototype of a molecular antipseudomonadic vaccine.     

An artificial intelligent diagnostic system on differential recognition of hematopoietic cells from microscopic images

The endothelium participates actively in homeostatic mechanisms such as the regulation of vascular tone and maintenance of a nonthrombotic environment, as well as directing biological responses such as leukocyte trafficking to inflammatory sites. Disruption of these processes leads to disease. In the antiphospholipid antibody syndrome autoantibodies provoke the endothelium to develop a prothrombotic surface. In systemic vasculitides associated with presence of antineutrophil cytoplasm antibodies, it is likely that the autoantibodies incite premature neutrophil activation, disrupted neutrophil-endothelium interactions and endothelial damage. This review considers how normal endothelial functions may be subverted in disease and how active endothelial responses may contribute to disease.