Modelling and controller design of an isolated diesel enginepermanent magnet synchronous generator

A method to analyze the steady-state performance of a stand-alone permanent magnet synchronous generator driven by a diesel engine is presented. The proposed method is based on equivalent d-q circuits and the phasor diagram of such a generator under steady-state conditions. A fixed capacitor-thyristor controlled reactor scheme is used to regulate the generator terminal voltage by controlling the thyristor ignition angle. Furthermore the overall system dynamics are modelled in terms of state variables and control inputs. Based on a reduced order linearized model, digital optimal state and output feedback controllers are designed by minimising a quadratic performance index using the dynamic programming technique. The objective of the controller is to maintain the load voltage and frequency constant under varying load conditions. The controller's effectiveness is assessed by examining the closed-loop system response to sudden load changes     

Assembly of hepatic gap junctions. Topography and distribution of connexin 32 in intracellular and plasma membranes determined using sequence-specific antibodies

The subcellular distribution in rat liver and the topography in intracellular and plasma membranes of connexin 32, a major protein component of gap junctions, was studied using sequence-specific anti-peptide antibodies generated to extracellular and intracellular domains of the protein. The distribution of connexin 32 in liver analyzed using SDS-polyacrylamide gel electrophoresis and Western blotting showed the relative protein levels in the subcellular fractions to be: lateral plasma membranes > Golgi membranes > sinusoidal plasma membranes > lysosomes. Low amounts of connexin 32 were detected in microsomes, endosomes, and bile canalicular plasma membranes. Six highly conserved cysteine residues are located in the amino acid sequences comprising the two extracellular loops of all connexins thus far isolated, and these loops are positioned to extend the channel in the lipid bilayers across the intercellular region of the gap junction. In the present work, the intramolecular disulfide bonds linking the extracellular loops in gap junctions were shown to be present in connexins located in plasma membranes, Golgi, and a microsomal fraction, and it was concluded that the disulfide linkages were formed in the endoplasmic reticulum. In addition, immature configurations of connexin 32, probably occurring during membrane insertion, were detected in liver microsomal fractions. The results contribute to charting of the biogenetic routes followed by connexins in hepatocytes and the general mechanisms of gap junction assembly.     

Priority-based joint EDF–RM scheduling algorithm for individual real-time task on distributed systems

The Journal of Supercomputing - Multiple tasks arrive in the distributed systems that can be executed in either parallel or sequential manner. Before the execution, tasks are scheduled prioritywise...     

Improving security of the Internet of Things via RF fingerprinting based device identification system

Neural Computing and Applications - Security is one of the primary concerns when designing wireless networks. Along detecting user identity, it is also important to detect the devices at the...     

RFID-based interactive multimedia system for the children

In this paper, we present an interactive edutainment system for the children that leverages multimedia and RFID technologies in a seamless manner. The proposed system allows children to learn about new objects/entities by tapping on physical objects through a specially designed RFID-Bluetooth based Tangible User Interface (TUI) tool. The output of the system is delivered as a set of appropriate multimedia representations related to the objects being tapped. The TUI uses RFID technology for object identification and Bluetooth communication to transmit data to the computer where the system??s software is running. We incorporated our system in three games that allow children of different ages to benefit from the system??s functionalities and encourage them to interact with it.     

Clones: what is that smell?

Clones are generally considered bad programming practice in software engineering folklore. They are identified as a bad smell?(Fowler et?al. 1999) and a major contributor to project maintenance difficulties. Clones inherently cause code bloat, thus increasing project size and maintenance costs. In this work, we try to validate the conventional wisdom empirically to see whether cloning makes code more defect prone. This paper analyses the relationship between cloning and defect proneness. For the four medium to large open source projects that we studied, we find that, first, the great majority of bugs are not significantly associated with clones. Second, we find that clones may be less defect prone than non-cloned code. Third, we find little evidence that clones with more copies are actually more error prone. Fourth, we find little evidence to support the claim that clone groups that span more than one file or directory are more defect prone than collocated clones. Finally, we find that developers do not need to put a disproportionately higher effort to fix clone dense bugs. Our findings do not support the claim that clones are really a “bad smell”?(Fowler et?al. 1999). Perhaps we can clone, and breathe easily, at the same?time.