Oxidation of Ti3SiC2 at 1000°C in Air

Oxidation of polycrystalline Ti₃SiC₂ was investigated at 1000°C by XRD and SEM. Results show that a thin SiO₂-rich scale forms after short-term oxidation, but it cannot prevent further oxidation effectively. The TiO₂ content increased with increasing time and covered the whole surface eventually. For the long-term oxidation, the oxide scales are layered, the outer layer is comprised of pure TiO₂, while the inner layer is a mixture of TiO₂ and SiO₂. The position of the pre-formed SiO₂ layer indicates that it can serve as an initial marker for the subsequent oxidation.     

Computational thermodynamics and microstructure simulations to understand the role of grain boundary phase in Nd-Fe-B hard magnets

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CO oxidation on non-alloyed Pt and Ru electrocatalysts prepared by the polygonal barrel-sputtering method

The CO oxidation on non-alloyed Pt and Ru electrocatalysts prepared by the polygonal barrel-sputtering method was investigated. Samples were prepared by sputtering Pt and Ru separately at room temperature. From the X-ray diffraction (XRD) measurement of the sample prepared on a SiO₂ glass plate, it was found that the sputtered metals are non-alloyed. Subsequently, the non-alloyed Pt and Ru electrocatalysts were prepared by the polygonal barrel-sputtering method using carbon powder as a support. The XRD patterns of these samples showed a single and very broad peak supporting the hypothesis of the non-alloyed Pt and Ru. No separate Ru reflections were visible, which could be attributed to Ru particle sizes smaller than 4 nm, as obtained by transmission electron microscopy (TEM). CO oxidation on the non-alloyed Pt and Ru electrocatalysts were evaluated by CO stripping voltammetry. This measurement revealed that the lower peak potential of CO oxidation varies, depending on the Pt content and the sputtering order. In addition, it was assumed that the CO oxidation reaction site for non-alloyed Pt and Ru electrocatalyst has a limited area including direct contact sites between the Pt and Ru particles.     

Low-crosstalk 10-GHz-spaced 512-channel arrayed-waveguide gratingmulti/demultiplexer fabricated on a 4-in wafer

We report a 10-GHz-spaced 512-channel arrayed-waveguide grating (AWG) multi/demultiplexer fabricated on a 4-in wafer. We achieved this by folding the 7-cm-long slab waveguides of a conventionally configured AWG, so that the whole configuration was contained in the wafer and then attached reflecting mirrors at the folded parts. Phase compensation of the fabricated AWG was performed by means of ultraviolet irradiation through a metal mask     

SiC/SiC composite heater for IFMIF

As the part of Japan/EU Broader Approach (BA) program for fusion, International Fusion Materials Irradiation Facility/Engineering Validation and Engineering Design Activities (IFMIF/EVEDA) project is on-going. Where, High Flux Test Module (HFTM) design aiming up to 1000 °C has been intensively investigated. Innovative SiC/SiC heater is currently potential and promising option. The weak temperature dependence of the SiC/SiC up to 1000 °C was interpreted to suggest the important role of grain boundaries and fiber–matrix interphase. To control the heater properties, variety of fabrication conditions, mainly on SiC nano-powders and green-sheet, were selected and tested. The heater performance was reasonably controlled and the results of heater performance and underlying microstructure behavior are provided.     

Comparison of ultrafast half-Fourier single-shot turbo spin-echo sequence with turbo spin-echo sequences for T2-weighted imaging of the female pelvis

1. This investigation was undertaken to compare pre- and postjunctional receptors involved in the responses of the canine mesenteric and pulmonary arteries to angiotensin II. 2. In the mesenteric artery, angiotensin II caused an enhancement of tritium overflow evoked by electrical stimulation (EC30% = 5 nM), the maximal effect representing an increase by about 45%. Postjunctionally, angiotensin II caused concentration-dependent contractions (pD2 = 8.57). Saralasin antagonized both pre- and postjunctional effects of angiotensin II, but it was more potent at post- than at prejunctional level (pA2 of 9.51 and 8.15, respectively), while losartan antagonized exclusively the postjunctional effects of angiotensin II (pA = 8.15). PD123319 had no antagonist effect either pre- or postjunctionally. 3. In the pulmonary artery, angiotensin II also caused an enhancement of the electrically-evoked tritium overflow (EC30% = 1.54 nM), its maximal effect increasing tritium overflow by about 80%. Postjunctionally, angiotensin II caused contractile responses (pD2 = 8.52). As in the mesenteric artery, saralasin antagonized angiotensin II effects at both pre- and postjunctional level and it was more potent postjunctionally (pA2 of 9.58 and 8.10, respectively). Losartan antagonized only the postjunctional effects of angiotensin II (pA2 = 7.96) and PD123319 was ineffective. 4. It is concluded that in both vessels: (1) pre- and postjunctional receptors belong to a different subtype, since they are differently antagonized by the same antagonists; (2) postjunctional receptors belong to AT1 subtype, since they are blocked by losartan but not by AT2 antagonists; (3) prejunctional receptors apparently belong to neither AT1 or AT2 subtype since they are blocked by neither AT1 nor AT2 antagonists.     

Statins Show Anti-Atherosclerotic Effects by Improving Endothelial Cell Function in a Kawasaki Disease-like Vasculitis Mouse Model

Kawasaki disease (KD) is an acute inflammatory syndrome of unknown etiology that is complicated by cardiovascular sequelae. Chronic inflammation (vasculitis) due to KD might cause vascular cellular senescence and vascular endothelial cell damage, and is a potential cause of atherosclerosis in young adults. This study examined the effect of KD and HMG-CoA inhibitors (statins) on vascular cellular senescence and vascular endothelial cells. Candida albicans water-soluble fraction (CAWS) was administered intraperitoneally to 5-week-old male apolipoprotein E-deficient (ApoE−) mice to induce KD-like vasculitis. The mice were then divided into three groups: control, CAWS, and CAWS+statin groups. Ten weeks after injection, the mice were sacrificed and whole aortic tissue specimens were collected. Endothelial nitric oxide synthase (eNOS) expression in the ascending aortic intima epithelium was evaluated using immunostaining. In addition, eNOS expression and levels of cellular senescence markers were measured in RNA and proteins extracted from whole aortic tissue. KD-like vasculitis impaired vascular endothelial cells that produce eNOS, which maintains vascular homeostasis, and promoted macrophage infiltration into the tissue. Statins also restored vascular endothelial cell function by promoting eNOS expression. Statins may be used to prevent secondary cardiovascular events during the chronic phase of KD.