A Comparison of Normal Approximation Rules for Attribute Control Charts

Control charts, known for more than 80 years, have been important tools for business and industrial manufactures. Among many different types of control charts, the attribute control chart (np‐chart or p‐chart) is one of the most popular methods to monitor the number of observed defects in products, such as semiconductor chips, automobile engines, and loan applications. The attribute control chart requires that the sample size n is sufficiently large and the defect rate p is not too small so that the normal approximation to the binomial works well. Some rules for the required values for n and p are available in the textbooks of quality control and mathematical statistics. However, these rules are considerably different, and hence, it is less clear which rule is most appropriate in practical applications. In this paper, we perform a comparison of five frequently used rules for n and p required for the normal approximation to the binomial. With this result, we also refine the existing rules to develop a new rule that has a reliable performance. Datasets are analyzed for illustration. Copyright © 2013 John Wiley & Sons, Ltd.     

Isotropic magnetization response of electrodeposited nanocrystalline Ni–W alloy nanowire arrays

Isotropic magnetization response was demonstrated in electrodeposited nanocrystalline Ni–15 % W alloy nanowire arrays, which can be applied to nanoscale magnetic field sensors. The Ni–W alloy nanowire arrays were electrochemically synthesized on a nanochannel template electrode from an aqueous electrolytic solution. X-ray and electron diffraction patterns revealed that Ni–15 % W alloy deposits were composed of ultrafine crystal grains with a supersaturated solid solution phase. The magnetization of the Ni–15 % W alloy thin films reached saturation at around 2.5 kOe in a perpendicular direction to the film plane, whereas the pure Ni thin films hardly magnetized in the perpendicular direction. On the contrary, Ni–15 % W alloy nanowire arrays were easily magnetized, and reach saturation at around 1.0 kOe, even in a perpendicular direction to the array film plane that corresponds to the long-axis direction of the alloy nanowires.     

Preparation of novel zeolite-containing hierarchical two-layered catalysts with large mesopores by gel skeletal reinforcement and their reactivities in catalytic cracking of n-dodecane

Journal of Porous Materials - In order to crack hydrocarbons with a large molecular weight, it is necessary to diffuse those substrates deeply into the inside of a catalyst. Catalysts having the...     

Cardioprotective Effects of a Nonsteroidal Mineralocorticoid Receptor Blocker,Esaxerenone, in Dahl Salt-Sensitive Hypertensive Rats

We investigated the effects of esaxerenone, a novel, nonsteroidal, and selective mineralocorticoid receptor blocker, on cardiac function in Dahl salt-sensitive (DSS) rats. We provided 6-week-old DSS rats a high-salt diet (HSD, 8% NaCl). Following six weeks of HSD feeding (establishment of cardiac hypertrophy), we divided the animals into the following two groups: HSD or HSD + esaxerenone (0.001%, w/w). In survival study, all HSD-fed animals died by 24 weeks of age, whereas the esaxerenone-treated HSD-fed animals showed significantly improved survival. We used the same protocol with a separate set of animals to evaluate the cardiac function by echocardiography after four weeks of treatment. The results showed that HSD-fed animals developed cardiac dysfunction as evidenced by reduced stroke volume, ejection fraction, and cardiac output. Importantly, esaxerenone treatment decreased the worsening of cardiac dysfunction concomitant with a significantly reduced level of systolic blood pressure. In addition, treatment with esaxerenone in HSD-fed DSS rats caused a reduced level of cardiac remodeling as well as fibrosis. Furthermore, inflammation and oxidative stress were significantly reduced. These data indicate that esaxerenone has the potential to mitigate cardiac dysfunction in salt-induced myocardial injury in rats.