Ein numerisches Verfahren zur Berechnung magnetischer Felder,insbesondere in Anordnungen mit Permanentmagneten

Übersicht Es wird ein numerisches Verfahren zur Berechnung zweidimensionaler, bzw. rotationssymmetrischer Magnetfelder in Anordnungen mit Eisenteilgebieten, Lufträumen, stromdurchflossenen Wicklungen und Permanentmagneten mittels digitaler Rechenmaschinen beschrieben.

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Contents A numerical method to calculate with digital computers twodimensional or rotationssymmetric magnet fields in devices with iron, air, currents and permanentmagnets is described.

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Anschrift des Verfassers Dr. Ing.K. Reichert, CH-5430 Wettingen, Rebhaldenstr. 10. Bis 31. 3. 1968 wissenschaftlicher Mitarbeiter am Institut für Elektrische Maschinen der Universität Stuttgart (Technische Hochschule). Direktor: Prof. Dr. Ing. habil.H. Hess, z. Z. in Fa. AG Brown, Boveri u. Cie., Baden.     

Kinetik der Polyphenylensulfid-Synthese

The kinetics of the polycondensation of Na₂S · 3H₂O and 1,4-dichlorobenzene to poly(phenylene sulfide) in 1-methyl-2-pyrrolidone as solvent was studied in a differential scanning calorimeter at temperatures from 220°C to 260°C. Stainless steel high-pressure capsules of 30 μl volume were used as micro reactors. The reaction is highly exothermic. At 220°C the enthalpy of reaction reaches a value of 180 kJ/mol. The polycondensation is characterized by an autocatalytic behaviour. The kinetics can be modelled by a second order equation with respect to the stoichiometrically charged monomers and a propagation rate constant being dependent on conversion. The activation energy of the polycondensation is 95 kJ/mol.     

New Frontiers on ER Stress Modulation: Are TRP Channels the Leading Actors?

The endoplasmic reticulum (ER) is a dynamic structure, playing multiple roles including calcium storage, protein synthesis and lipid metabolism. During cellular stress, variations in ER homeostasis and its functioning occur. This condition is referred as ER stress and generates a cascade of signaling events termed unfolded protein response (UPR), activated as adaptative response to mitigate the ER stress condition. In this regard, calcium levels play a pivotal role in ER homeostasis and therefore in cell fate regulation since calcium signaling is implicated in a plethora of physiological processes, but also in disease conditions such as neurodegeneration, cancer and metabolic disorders. A large body of emerging evidence highlighted the functional role of TRP channels and their ability to promote cell survival or death depending on endoplasmic reticulum stress resolution, making them an attractive target. Thus, in this review we focused on the TRP channels’ correlation to UPR-mediated ER stress in disease pathogenesis, providing an overview of their implication in the activation of this cellular response.