IEEE1394技术在视频采集与传输中的应用

数字视频采集与传输所使用的普通串口技术普遍存在成本较高、体积较大、速度较低等问题,而IEEE1394技术则以多方面的优势成为一种较为理想的视频传输方案.通过IEEE1394接口,可以用摄录设备来播放和采集视频,也可以将计算机上编辑好的视频下载录制到录像带或其他媒体上.     

结合功率控制的时-空多用户检测器

针对CDMA系统中抗干扰技术,提出一种结合多用户检测、波束成形和功率控制的算法,对接收信号在时域和空间上进行联合处理,能大大抑制干扰,提高系统容量。     

宽工作电压范围单电源CMOS误差放大器

采用0.8μm标准数字CMOS工艺(VTN0=0.836V,VTP0=0.930V),设计并流片验证了具有宽工作电压范围(3～6V),可作SOC系统动态电源管理芯片内部误差放大器应用的单电源CMOS运算放大器。该误差放大器芯核同时具有适合低电压工作,并对工艺参数变化不敏感的优点。对于相同的负载情况,在3V的工作电压下,开环电压增益AD=83.1dB,单位增益带宽GB=2.4MHz,相位裕量Φ=85.2°,电源抑制比PSRR=154.0dB,转换速率Sr=2.2V/μs;在6V工作电压下,AD=85.1dB,GB=2.4MHz,Φ=85.4°,PSRR=145.3dB,Sr=3.4V/μs。     

基于有限状态的全周期序列混沌映射的研究

在有限长度的数字计算机中，混沌迭代序列是一个有限周期序列，其相关性能远远低于理想序列。基于skew tent映射，提出一种离散化的有限状态混沌映射，它允许精确的数值计算，没有舍入误差，通过选择合适的参数，映射能产生全周期长度的序列。仿真实验表明，归一化的序列平均周期对计算机精度不敏感，生成的二进制序列具有良好的相关性能。     

MRC分集接收的MQAM在Nakagami信道上的性能分析

Nakagami信道是一种适应性较强的信道模型，MQAM(M-ary quadrature amplitude modulation)是一种频谱利用率较高的调制方式。在存在多径影响的无线通信环境中，最大比合并(maximum ratio combining)方式的分集接收是克服衰落的有效方法。本文利用适用范围更广的Nakagami信道模型，在多径衰落的情况下对最大比合并分集接收的MQAM系统误符号率性能作了分析。仿真结果对分析实际信道下具体系统的性能具有一定的参考价值。     

射频电缆组件组装工艺浅谈

近年来国内外射频电缆组件的发展很快,射频电缆组件的性能不断提高,对射频电缆组件组装工艺也提出了更高的要求.射频电缆组件组装过程多是手工操作,产品的一致性及性能很难控制,要求有良好的工艺保证,为此介绍了射频电缆组件组装工艺,包括电缆的裁剪及剥皮、内导体的连接、外导体的连接和电气性能的测试;探讨了在组装过程中应注意的问题,以及针对内导体焊接易出现虚焊问题进行研究.     

Mechanical Model on Brittle Crack Initiation and Propagation

A mechanical model is presented in this paper to describe the initiating and propagating of brittle cracks. Two criteria have been deduced from the model to determine the effects of such factors as local stress state, surface and grain boundary energies as well as local grain boundary orientation on the initiating and propagating of both intergranular and transgranular brittle cracks. By which the role of adding B in Ni3Al base alloys to improve their ductility,temper and hydrogen embrittlements in steels and other alloys as well as the random feature of brittle crack initiation could be explained     

Effect of hygiene measures, water sanitation and oral rehydration therapy on diarrhea in children less than five years old in the south of Ivory Coast

BACKGROUND: Gastric sucrose permeability is a noninvasive marker that reliably increases in association with gastrointestinal injury due to use of nonsteroidal antiinflammatory drugs. Despite the effect of Helicobacter pylori infection on the gastric mucosa, in a previous study we were unable to demonstrate that H. pylori infection was associated with abnormal gastric sucrose permeability. Our goal in this study was to explore further whether H. pylori infection changed gastric permeability; therefore, we evaluated the effect of treatment of H. pylori infection on gastric permeability to sucrose and the relation of sucrose permeability to density of polymorphonuclear leukocytes. MATERIALS AND METHODS: Five hundred milliliters of a solution containing 100 gm of sucrose was ingested by the subject at bedtime. Overnight urine was collected and assayed for sucrose by high-performance liquid chromatography. Sucrose permeability was assessed both before and approximately 4 weeks after anti-H. pylori therapy. RESULTS: Seventeen asymptomatic H. pylori-infected volunteers participated; 8 were cured. Sucrose permeability was in the range commonly found in normal controls both before and after anti-H. pylori therapy (mean excretion, 76.3 mg; range, 13-171 mg). Gastric sucrose permeability correlated with the density of polymorphonulcear cell infiltration of the mucosa. Cure of the H. pylori infection was associated with a small but significant decrease in sucrose permeability (98.8 +/- 18 mg to 51.7 +/- 9.8 mg (p = .01). Sucrose permeability was greater in those with a high density of mucosal polymorphonuclear cells compared to those with lower scores (119.5 +/- 4 vs 71.4 +/- 13 for those with scores > or = 5 compared to scores < or = 4; p = .023). Failed therapy resulted in an increase in the mucosal density of polymorphonuclear infiltration and sucrose permeability (56.4 +/- 13 mg-99.7 +/- 19 mg pretreatment vs posttreatment, respectively; p = .031). CONCLUSION: H. pylori gastritis causes a small but measurable increase in gastric permeability to sucrose that may reflect epithelial transmigration of neutrophils.