丙烯酸接枝聚丙烯纤维的结构与性能

本文研究了用化学引发剂引发丙烯酸接枝聚丙烯纤维的某些影响因素。红外光谱分析表明，丙烯酸接枝在聚丙烯大分子链的叔碳原子上。Ｘ－射线衍射和ＤＳＣ分析指出，纤维接枝后结晶度有所增加，接枝前纤维存在的β－变体转化成了α－变体，接枝纤维的吸湿性、离子交换容量和染色性随接枝率增加而增大。接枝纤维的力学性能有所下降。     

激光辐照对冷冻精子活率和活力的影响

分别用7.48J/cm~2和14.96J/cm~2能量密度的氦氖激光辐照23份液氮冷冻前人的精液,冻贮三个月后复温查精子活率和毛细管穿透高度。结果:经7.48J/cm~2剂量的激光辐照后的精子活率和穿透高度明显提高,分别平均提高10.18％和8.32mm,与未受辐照组比较差异显著(P<0.05)经14.96J/cm~2辐照组,活率较未照组降低。实验结果说明适当剂量的激光辐照能够增强精子对冷冻的耐受力,提高精子的复活率和活动力。     

空拉铜管“缩径”现象研究

揭示了空拉铜管时的“缩径”现象,分析了产生这种现象的原因以及各种工艺条件对其影响的规律。     

影响碲镉汞晶体电子迁移率的主要因素

通过把迁移率的实测值与影响ＨｇＣｄＴｅ晶体电子迁移率的主要散射机构进行对比，得出结论：位错是ＨｇＣｄＴｅ晶体低温电子迁移率降低的主要原因。     

Electron microscopic observations and DNA chain fragmentation studies on apoptosis in bone tumor cells induced by ~(153)Sm-EDTMP

ＥｌｅｃｔｒｏｎｍｉｃｒｏｓｃｏｐｉｃｏｂｓｅｒｖａｔｉｏｎｓａｎｄＤＮＡｃｈａｉｎｆｒａｇｍｅｎｔａｔｉｏｎｓｔｕｄｉｅｓｏｎａｐｏｐｔｏｓｉｓｉｎｂｏｎｅｔｕｍｏｒｃｅｌｓｉｎｄｕｃｅｄｂｙ１５３ＳｍＥＤＴＭＰＺｈｕＳｈｏｕＰｅｎｇ，Ｘｉａ...     

Up-regulation of CIR1/CROC1 expression upon cell immortalization and in tumor-derived human cell lines

Inactivation of 5-HT1A and [3H]5-HT binding sites by N-Ethoxycarbonyl-2-ethoxy-1, 2-dihydroquinoline (EEDQ) was studied in regions of rat brain. After exposure to EEDQ (4 mg/kg body wt.) for 7 days, it is observed that the density of 5-HT1 receptor sites was decreased by nearly 20% in both cortex and hippocampus. The decrease, however, in 5-HT1A sites was more significant (70%) in both the regions. The affinity of [3H]5-HT to 5-HT1 sites was decreased significantly in both cortex and hippocampus after exposure to EEDQ, without affecting the Kd of 5-HT1A sites. Displacement studies suggested that EEDQ has high affinity to 5-HT1 sites with a Ki of 42.9+/-2.4 nM. After exposure neither basal nor 5-HT stimulated adenylyl cyclase activity was changed in cortex. The results of this study suggest that EEDQ decreases the density of 5-HT1 and 5-HT1A receptor sites but does not cause functional downregulation of these sites in rat brain.     

布尔函数的对偶性和线性点

本文主要讨论了部分自对偶和部分反自对偶函数、部分线性函数以及具有线性点的函数的性质。     

Finite element analysis of local damage and post-failure behavior for strain-softening solid

A finite element analysis of the viscoplastic-softening model with the evolution of local damage for strain softening material is presented. An effective damage matrix is introduced to consider the influence of isotropic damage on different types of micro-cracking by different states of stress. The localized strain mode with local damage occurs in the post-failure regime of deformation. The computational techniques for tracing the post-failure path of the softening response are discussed with several examples.