一个表示和运用非功能性需求的框架

本文分析了一个软件开发进程中表示和运用非功能性需求的综合框架，该框架的目的是开发一套技术以调整软件开发进程中的设计决策，从而强调了依据非功能需求，探索合理化的软件开发进程。     

国内外电雷管电引火元件现状及改进思路

文章分析了目前国内外电引火元件的现状，列举了国外著名火炸药公司典型的几种电引火元件结构及电火参数，提出了改进我国电引火元件的技术构思。     

节理岩体的边界元模型及其应用

边界元法广泛应用于岩石力学问题的最大障碍是岩体的非均质及其包含的大量节理断层，本文采用多子域思想结合节理断层的非线性模型较好地解决了这一难题。该法在老厂锡矿的应用较好地解释了该矿采场顶板的冒落机理。     

InGaAsP/InP有源双稳放大器的实验研究

本文报导了室温下F—P型InGaAsP/InP双异质结半导体激光放大器的双稳态效应,放大器的增益为20dB。     

排列互比法用于超精测角的研究

作者分析了我互比法现行的两种数据处理方法的缺点，提出了应用最小二乘法原理推导出合理的数据处理方法。本文还首次从理论上阐明了排列互比法测量结果中出现一次谐波系统误差的原因，提出了减少和消除这一些系统误差的方法，并给实验验证。     

无横梁高刚性轧机的结构分析

本文介绍了无横梁高刚性轧机的新颖结构，分析了该轧机高刚性及轴承长寿的原理。该轧机在现场使用中，取得了良好的效果。     

集群系统与公用网互连的仿真模型和模拟算法

在讨论ＭＰＴ１３２７信令的集群移动通信系统与国内Ｎｏ．７信令的ＰＳＴＮ网间互连，帝现两种信令配合中，研究了集群系统的仿真模型和模拟算法，同时模拟出整个集群系统的工作情况，运行结果证实了所提方案的可行性与准确性。     

红外热成象技术在股骨头坏死疾病针刺疗法中的应用

红外热成象技术可以有效地用于股骨头坏死疾病的诊断和针刺治疗效果的评价，效果优于其它常规方法。     

Synthesis and Crystal Structure of RE[CH_2(CH_2)_4CONC_4H_9]_3(NO_3)_3(RE=Dy,La)

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CBF beta-SMMHC, expressed in M4Eo AML, reduced CBF DNA-binding and inhibited the G1 to S cell cycle transition at the restriction point in myeloid and lymphoid cells

CBF beta-SMMHC is expressed from the inv(16) chromosome in M4Eo AML. Mice lacking CBF subunits or expressing the CBF beta-SMMHC or AML1-ETO oncoproteins failed to develop definitive hematopoiesis. To investigate these effects on hematopoiesis, we expressed CBF beta-SMMHC from the metallothionein promoter, in both 32D cl3 myeloid cells and Ba/F3 B-lymphoid cells. Addition of zinc increased CBF beta-SMMHC levels more than tenfold, with higher levels evident in Ba/F3 lines. Levels obtained in 32D cl3 cells were similar to those of endogenous CBF beta. Indirect immunofluorescence revealed zinc-inducible speckled, nuclear staining in Ba/F3 cells and diffuse nuclear staining in 32D cl3 cells. CBF beta-SMMHC reduced endogenous CBF DNA-binding fivefold in both cell types, increased cell generation time 1.9-fold, on average, in 32D cl3 cells and 1.5-fold in Ba/ F3 cells and decreased tritiated thymidine incorporation into DNA correspondingly. CBF beta-SMMHC increased the proportion of cells in G1 1.7-fold, on average, in 32D cl3 and Ba/F3 cells, and decreased the proportion of cells in S phase by a similar degree. CBF beta-SMMHC induced a marked increase in hypophosphorylated Rb, but did not alter IL-3 Receptor alpha or beta subunit levels. Neither apoptosis nor 32D differentiation was induced by zinc in IL-3 in these lines. Induction of CBF beta-SMMHC in 32D cl3 cells did not inhibit their differentiation to neutrophils or their expression of myeloperoxidase mRNA in G-CSF, and did not produce an eosinophilic phenotype. Additional, proliferative genetic changes in M4eo AMLs might potentiate inhibition of differentiation by CBF beta-SMMHC by allowing its increased expression.