纱线分析用精密分级系统

现代质量保证体系中,任何一次纱线贸易都要求出具检测报告,这是供应商获得产品质量保证或认证的珏要方式。Uster Classimat Quantum不仅可用于检测纱线质量、确定粗细节以及异纤数量,为贸易服务;还可作为精密的分级系统,帮助用户确定并建立自己的纱线质量标准。     

浆状炸药、乳化炸药和重铵油炸药的实验室对比性研究

一、引言重量比为94.4/5.6的多孔粒状硝铵和燃料油混成的铵油炸药是露天矿开采最常用的炸药,主要是因为它的成本低。但这种炸药也有局限性,它必须在干燥的条件下或密封在袋中使用。此外,为了在现场混装,必须采用粒度分布较窄的硝铵颗粒,才有良好的流散性,其结果炸药的密度低,一般在0.80～0.85克/厘米~3的范围。在大直径炮孔中的能量输出约为903卡/克。这就意味着,尽管铵油炸药的重量威力尚好,但容积威力只是中常。为了减轻这些缺陷,近几年作了     

Designing metallic glasses with optimal combinations of glass-forming ability and mechanical properties

It is a long-standing challenge to search for metallic glasses(MGs)with optimal combinations of glassforming ability(GFA),strength and toughness in the vast compositional space.By taking into account both recently developed ellipse criterion and temperature-based GFA criterion,here we established quantitative correlations among compositions,elastic constants,GFA and mechanical properties of MGs,which enable to predict the GFA,fracture strength and fracture surface simultaneously in advance once the compositions of MGs are determined.Experimental data confirm the validity of this approach in prediction.Finally,a strategy for designing MGs with optimal combinations of strength,toughness and GFA is proposed,which allows for high-throughput discovering glass formers with excellent mechanical properties.     

若昂·帕谢科住宅,圣米格尔岛,亚述尔群岛,葡萄牙

基地是一片由石墙环绕的牧场,被分成了3个不同标高的平台。这栋住宅就建在平台的交界处:车库与入口条带平行;起居室、餐厅和厨房与之相邻,坐落在朝西的平台上,可以望见破碎的峡谷直至远方奔腾入海的山色;卧室则被放置在更高一级的平台上。这3个部分被一个单坡的屋顶联接起来。屋顶在东面升高的部分容纳了一个夹层,并向前挑出观景台,它将视线又转回到了入口一端,并且为下方的宅门提供了庇护。□Theplace,apasturesurroundedbystonewalls,isdividedinthreedifferentlevelledplatforms.Thehousefoundsitsplacewheretheplatformsmeet:alignedwi…     

进化的建筑——采访柯里斯·威尔金森和吉姆·艾尔

与柯里斯·威尔金森和吉姆·艾尔的访谈围绕9个问题展开,从对“进化的建筑”的解释,到他们对设计理论和方法的阐述,以及时下建筑的发展方向的泛泛讨论,直至他们对中国的一些看法,比较全面地展现了威尔金森·艾尔的设计世界。     

生态景观设计40年

本文简要总结了英国生态景观设计的历史,阐述了在景观设计中如何纳入生态设计原则。通过对位于爱丁堡郊区里卡顿的一所新大学校园的规划与建设的案例研究,阐明了生态景观理论在景观设计中的实践。     

基于X射线光谱仪分析环锭纺与集聚纺纱线织物丝光后的结晶度研究

在相同的纤维混合与工艺参数下分别制得环锭纺与集聚纺纱线织物,并在不同温度、氢氧化钠质量分数、浸渍时间及织物弹性伸长率条件下进行丝光处理。采用Mat-Lab计算分析软件对织物试样的X射线光谱进行分析,得到丝光作用的最佳条件。     

Glutathione-S-transferase M1 regulation of diesel exhaust particle-induced pro-inflammatory mediator expression in normal human bronchial epithelial cells

ABSTRACT: BACKGROUND: Diesel exhaust particles (DEP) contribute substantially to ambient particulate matter (PM) air pollution in urban areas. Inhalation of PM has been associated with increased incidence of lung disease in susceptible populations. We have demonstrated that the glutathione Stransferase M1 (GSTM1) null genotype could aggravate DEP-induced airway inflammation in human subjects. Given the critical role airway epithelial cells play in the pathogenesis of airway inflammation, we established the GSTM1 deficiency condition in primary bronchial epithelial cells from human volunteers with GSTM1 sufficient genotype (GSTM1+) using GSTM1 shRNA to determine whether GSTM1 deficiency could exaggerate DEP-induced expression of interleukin-8 (IL-8) and IL-1beta proteins. Furthermore, the mechanisms underlying GSTM1 regulation of DEP-induced IL-8 and IL-1beta expression were also investigated. METHODS: IL-8 and IL-1beta protein levels were measured using enzyme-linked immunosorbent assay. GSTM1 deficiency in primary human bronchial epithelial cells was achieved using lentiviral GSTM1 shRNA particles and verified using real-time polymerase chain reaction and immunoblotting. Intracellular reactive oxygen species (ROS) production was evaluated using flow cytometry. Phosphorylation of protein kinases was detected using immunoblotting. RESULTS: Exposure of primary human bronchial epithelial cells (GSTM1+) to 25-100 mug/ml DEP for 24 h significantly increased IL-8 and IL-1beta protein expression. Knockdown of GSTM1 in these cells further elevated DEP-induced IL-8 and IL-1beta expression, implying that GSTM1 deficiency aggravated DEP-induced pro-inflammatory response. DEP stimulation induced the phosphorylation of extracellular signal-regulated kinase (ERK) and Akt, the downstream kinase of phosphoinositide 3-kinase (PI3K), in GSTM1+ bronchial epithelial cells. Pharmacological inhibition of ERK kinase and PI3K activity blocked DEP-induced IL-8 and IL-1beta expression. DEP-induced ERK and Akt phosphorylation could be increased by GSTM1 knockdown. In addition, pretreatment of HBEC with the antioxidant N-acetyl cysteine significantly inhibited DEP-induced ERK and Akt phosphorylation, and subsequent IL-8 and IL-beta expression. CONCLUSION: GSTM1 regulates DEP-induced IL-8 and IL-1beta expression in primary human bronchial epithelial cells by modulation of ROS, ERK and Akt signaling.