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21.
Wenying Wei Zhen Wang Yanhua Yin Jinyu Han Wen Xu 《Korean Journal of Chemical Engineering》2008,25(3):562-567
Composition Group Vector Space (CGVS) method for estimating melting and boiling point T
m
, T
b
of organic compound has been proposed, and the principle of this method has been elucidated. The models for estimating T
m
, T
b
have been established and the numerical values of relative parameters have been presented. The average percentage deviations
of T
m
, T
b
estimation are 7.53 and 1.58, respectively, which show that the present method demonstrates significant improvement in applicability
to predict the above properties, compared to conventional group methods. 相似文献
22.
介绍在Windows XP家庭版下成功安装与配置ⅡS和ASP.NET的方法。 相似文献
23.
24.
周冰毅 《有色金属(冶炼部分)》1996,(6):14-17
亚铜离子氨浸法作为大洋多金属结核处理方法之一,一直被认为是一个很有前途的冶金处理工艺,是催化化学还原技术在冶金生产中的具体应用,具有流程简单,能耗低,浸出选择性好,产品结构灵活,市场适应能力强,浸出试剂腐蚀性与毒性小,环境污染小等优点。 相似文献
25.
BID: a novel BH3 domain-only death agonist 总被引:1,自引:0,他引:1
K Wang XM Yin DT Chao CL Milliman SJ Korsmeyer 《Canadian Metallurgical Quarterly》1996,10(22):2859-2869
The BCL-2 family of proteins consists of both antagonists (e.g., BCL-2) and agonists (e.g., BAX) that regulate apoptosis and compete through dimerization. The BH1 and BH2 domains of BCL-2 are required to heterodimerize with BAX and to repress cell death; conversely, the BH3 domain of BAX is required to heterodimerize with BCL-2 and to promote cell death. To extend this pathway, we used interactive cloning to identify Bid, which encodes a novel death agonist that heterodimerizes with either agonists (BAX) or antagonists (BCL-2). BID possesses only the BH3 domain, lacks a carboxy-terminal signal-anchor segment, and is found in both cytosolic and membrane locations. BID counters the protective effect of BCL-2. Moreover, expression of BID, without another death stimulus, induces ICE-like proteases and apoptosis. Mutagenesis revealed that an intact BH3 domain of BID was required to bind the BH1 domain of either BCL-2 or BAX. A BH3 mutant of BID that still heterodimerized with BCL-2 failed to promote apoptosis, dissociating these activities. In contrast, the only BID BH3 mutant that retained death promoting activity interacted with BAX, but not BCL-2. This BH3-only molecule supports BH3 as a death domain and favors a model in which BID represents a death ligand for the membrane-bound receptor BAX. 相似文献
26.
27.
高职教育高等数学教学的改革 总被引:1,自引:0,他引:1
随着高职教育的迅速发展,本文针对高职数学教学现状,结合实际分别对教学思想、教学内容、教学方法和教学手段、教学形式以及考试方式等方面的改革进行了探讨。 相似文献
28.
29.
We have previously shown that Stu2p is a microtubule-binding protein and a component of the Saccharomyces cerevisiae spindle pole body (SPB). Here we report the identification of Spc72p, a protein that interacts with Stu2p. Stu2p and Spc72p associate in the two-hybrid system and can be coimmunoprecipitated from yeast extracts. Stu2p and Spc72p also interact with themselves, suggesting the possibility of a multimeric Stu2p-Spc72p complex. Spc72p is an essential component of the SPB and is able to associate with a preexisting SPB, indicating that there is a dynamic exchange between soluble and SPB forms of Spc72p. Unlike Stu2p, Spc72p does not bind microtubules in vitro, and was not observed to localize along microtubules in vivo. A temperature-sensitive spc72 mutation causes defects in SPB morphology. In addition, most spc72 mutant cells lack cytoplasmic microtubules; the few cytoplasmic microtubules that are observed are excessively long, and some of these are unattached to the SPB. spc72 cells are able to duplicate and separate their SPBs to form a bipolar spindle, but spindle elongation and chromosome segregation rarely occur. The chromosome segregation block does not arrest the cell cycle; instead, spc72 cells undergo cytokinesis, producing aploid cells and polyploid cells that contain multiple SPBs. 相似文献
30.