G神经网络函数映射能力的构造性证明

该文研究了G神经网络的函数映射能力,给出了前馈G神经网络映射任意G型多项式的构造性证明。采用该文的方法映射同一个多项式,所用的神经元数目可少至以往方法的2/(n+1),其中n是G型多项式的次数。     

基于ASP的教师评价系统的设计与实现

给出了一种可自定义评价指标的教师评价系统的统计数学模型,并采用了ASP技术来实现这个评价系统。     

An approach for detecting the commonality and specialty between scientific publications and patents

Scientometrics - Scientific publications and patents are usually viewed as respective proxies of scientific research and technical development. There is considerable effort spent towards...     

基于链路状态数据库的数据中心网络异常检测算法

目前IDC数据中心内部由于网络攻击或网络配置等原因路由会经常变化,然而由于缺乏有效的监控软件,路由异常、路由抖动难以发现,故障难以定位.数据中心业务出现网络故障时无法确认故障点导致修复时间延长、用户体验降低和运营收入减少等问题.分析了当前主流数据中心的网络架构、通信协议和路由计算原理,提出了一种基于链路状态数据库(link state database, LSDB)的数据中心网络异常检测方法LSAP,该方法通过搜集LSDB,使用改进路由算法计算全网路由形成路由择域信息库(routing information base, RIB),根据LSDB快照和RIB快照比对准确关联链路变化和路由变化,发现链路异常、路由异常,能够定位故障.LSAP基于大数据分析平台实时计算路由表,能够实现秒级处理上亿条路由信息,满足当前数据中心对于分析速率的要求.通过在数据中心网络中部署试用,LSAP能够快速发现拓扑变化、复原路由表,统计分析所有路由变化,先于业务发现路由异常、路由攻击,且对网络改动很少,被动搜集数据不影响网络自身稳定性,适用对稳定性要求较高的数据中心部署.     

中国科学院光电技术研究所的变形反射镜研究进展

变形反射镜是自适应光学系统的核心部件,也是开展自适应光学技术研究的首要研究对象。本文首先介绍了中国科学院光电技术研究所从事自适应光学特别是变形反射镜技术研究的历史背景,简述了光电所变形反射镜技术早期的发展脉络。然后介绍了光电所研制的变形反射镜在我国历代惯性约束聚变系统中的应用情况,也介绍了在天文光学观测领域典型的多单元变形镜技术及应用成果,随后还介绍了应用于生物医学等领域的紧凑型变形反射镜的发展情况和研究现状。最后介绍了光电所在变形反射镜技术新方向的研究情况。

     

发动机用K439B镍基高温合金光纤激光自熔焊性能分析

采用激光自熔焊方法连接K439B焊接头,开展焊接头组织结构转变并对其力学特性进行了测试,分析焊接速率对其焊缝微观组织及焊缝变形程度的影响。研究结果表明:随着焊接速率提高,K439B焊接头获得热输入低,靠近焊缝中心部以等轴晶为主柱状晶,基本都是出现在熔合线区域,形成更细的焊缝组织,并引起焊缝硬度的明显提升。随着焊接速率增加,K439B焊接头拉伸强度和伸长率均表现出先增加后减小,最大值发生在焊接速率4 m/min时,此时拉伸强度和伸长率分别为852 MPa和44.5%。对焊接速率4 m/min下K439B焊接头在焊缝内形成了连续分布的胞晶、柱状晶、等轴晶。K439B焊接头形成了平整的表面区域,存在许多韧窝结构,主要表现为穿晶断裂的特点,焊缝端部发生了明显变形。     

超重质原油井下密闭取样工艺研究及其在塔河油田的应用

塔河油田12区奥陶系油藏原油密度大,属于超重质原油;原油动力粘度高,流动性能极差;地层压力高、地层温度高;主要采用掺稀生产,掺稀点深度一般为5500m 左右。通过超重质原油井下密闭取样工艺的研究,对改进高压物性取样器、研制加热转样装置、优化取样工艺,首次成功于5950m 取得合格的高压物性样品,首次成功转样并成功进行高压物性分析,创造了高压物性取样点压力最高、粘度最高、温度最高、深度最大4项国内纪录,形成了一整套超重质原油井下密闭取样工艺技术。     

流量阶跃变化时供暖房间内温度场的数值模拟

两位通断式调节是一种计量供热系统控制模式,与欧洲供热系统控制模式不同的是,其温控阀只有启、闭两种状态.用数值模拟方法研究了该调控模式对室内热环境的影响规律.     

Weakly Binding Molecules-Based Fast Charging Li-Ion Batteries

Fast charging of Li-ion batteries (LIBs) beyond standard 0.3 C (charged in 3.3 h) are desperately pursued but hindered by sluggish desolvation kinetics of ethylene carbonate-based traditional electrolyte, and Li-plating and dendrites growth at graphite anode and fire hazard. Herein, a new class of weakly binding all linear molecules-based nonflammable electrolyte (WNLE) is reported, comprising 1 m LiPF₆ in ethyl methyl carbonate and 2,2,2-trifluoroethyl acetate with additives for 10–20 times faster charging LIBs than traditional ones. The critical benefits of WNLE are 44% lower viscosity, 62% higher Li⁺ diffusion coefficient, 20% higher Li⁺ transference number, and 17% lower desolvation energy, which promotes diffusion kinetics and desolvation kinetics of Li⁺ in the vicinity of graphite anode enabling dendrites-free LIB, along with nonflammability. Under 3 C (charged in 20 min), WNLE-based industrial 800 mAh graphite//LiNi_0.8Mn_0.1Co_0.1O₂ (high active mass 13 mg cm⁻²) Li-ion pouch battery achieves outstanding 700 cycles, delivering 82% capacity retention and high Coulombic efficiencies ≈100%. Robust solid electrolyte interphase layers formed at the anode and cathode mitigate interfacial failures, making fast charge to 7 C and longer cycle-life. This new class of electrolyte formulation is a promising solution and a new opportunity to realize safe and long operation of fast-charging LIBs for practical applications.     

The Iridoid Glycoside Loganin Modulates Autophagic Flux Following Chronic Constriction Injury-Induced Neuropathic Pain

Autophagy facilitates the degradation of organelles and cytoplasmic proteins in a lysosome-dependent manner. It also plays a crucial role in cell damage. Whether loganin affects autophagy in chronic constriction injury (CCI)-induced neuropathic pain remains unclear. We investigated the neuroprotective effect of loganin on the autophagic–lysosomal pathway in the rat CCI model. Sprague–Dawley rats were divided into sham, CCI, sham + loganin, and CCI + loganin. Loganin (5 mg/kg/day) was intraperitoneally injected once daily, and rats were sacrificed on day 7 after CCI. This study focused on the mechanism by which loganin modulates autophagic flux after CCI. CCI enhanced the autophagic marker LC3B-II in the ipsilateral spinal cord. The ubiquitin-binding protein p62 binds to LC3B-II and integrates into autophagosomes, which are degraded by autophagy. CCI caused the accumulation of p62, indicating the interruption of autophagosome turnover. Loganin significantly attenuated the expression of Beclin-1, LC3B-II, and p62. Double immunofluorescence staining was used to confirm that LC3B-II and p62 were reduced by loganin in the spinal microglia and astrocytes. Loganin also lessened the CCI-increased colocalization of both proteins. Enhanced lysosome-associated membrane protein 2 (LAMP2) and pro-cathepsin D (pro-CTSD) in CCI rats were also attenuated by loganin, suggesting that loganin improves impaired lysosomal function and autophagic flux. Loganin also attenuated the CCI-increased apoptosis protein Bax and cleaved caspase-3. Loganin prevents CCI-induced neuropathic pain, which could be attributed to the regulation of neuroinflammation, neuronal autophagy, and associated cell death. These data suggest autophagy could be a potential target for preventing neuropathic pain.