用热分析法研究硝化棉的熔化过程

用ＭＤＳＣ，显微镜温台熔点测定仪和固体原位反应池／快速扫描傅立叶变换红外光谱联用装置考察了硝化棉的熔化过程。ＭＤＳＣ曲线表明吸热过程是可逆的，它系由ＮＣ和部分分解凝聚相产物的混合物的固液相变所引起。由ＮＣ的ＭＤＳＣ曲线得到的熔点，熔化含，熔化熵，分解含和分解反应热温熵分别为４７６．８４Ｋ，２０５．６Ｊ．ｇ＾－１，０．４３１２Ｊ．ｇ＾－１．Ｋ＾－１，－２４７５．０Ｊ．ｇ＾－１，－５．２４２Ｊ．ｇ＾－     

BID: a novel BH3 domain-only death agonist

The BCL-2 family of proteins consists of both antagonists (e.g., BCL-2) and agonists (e.g., BAX) that regulate apoptosis and compete through dimerization. The BH1 and BH2 domains of BCL-2 are required to heterodimerize with BAX and to repress cell death; conversely, the BH3 domain of BAX is required to heterodimerize with BCL-2 and to promote cell death. To extend this pathway, we used interactive cloning to identify Bid, which encodes a novel death agonist that heterodimerizes with either agonists (BAX) or antagonists (BCL-2). BID possesses only the BH3 domain, lacks a carboxy-terminal signal-anchor segment, and is found in both cytosolic and membrane locations. BID counters the protective effect of BCL-2. Moreover, expression of BID, without another death stimulus, induces ICE-like proteases and apoptosis. Mutagenesis revealed that an intact BH3 domain of BID was required to bind the BH1 domain of either BCL-2 or BAX. A BH3 mutant of BID that still heterodimerized with BCL-2 failed to promote apoptosis, dissociating these activities. In contrast, the only BID BH3 mutant that retained death promoting activity interacted with BAX, but not BCL-2. This BH3-only molecule supports BH3 as a death domain and favors a model in which BID represents a death ligand for the membrane-bound receptor BAX.     

地层测试资料在实际应用中若干问题浅析

华北油田冀中地区在十几年实践过程中，陆续开展了200多层次地层测试（或三开抽汲）及该试油层常规抽汲试油工作。通过对1300次测试资料特别是对200层复试资料的分析对比，认为测试资料对自喷层和中高产非自喷井是准确的，可以不必进行三开抽汲。对井壁深部污染、低渗透和特低渗透层还存在一些偏差，为解决探井中一些疑难问题，需要配合抽汲。     

首钢6H3C单机架可逆式薄板冷轧机组技术特点

首钢6H3C单机架可逆式冷轧机组是目前世界上第一套通过中间辊交叉进行板型控制的轧机．也是世界上第一套投入工业生产的6H3C轧机。本文介绍了该机组的工艺、设备参数，技术特点，电气设备及自动化控制及生产状况，并提出了该机组可能存在的问题。     

Acadesine extends the window of protection afforded by ischaemic preconditioning in conscious rabbits

OBJECTIVE: Ischaemic preconditioning protects myocardium from infarction if the reperfusion interval between the brief and prolonged ischaemic intervals is less than 1 h. In anaesthetised rabbits acadesine (5-amino-4-imidazolecarboxamide riboside, AICAR), an adenosine enhancer which increases tissue adenosine during ischaemia, prolongs the window of protection to 2 h. The aim of this study was to try to determine the maximum extension of this window of protection, using chronically instrumented, unsedated rabbits. METHODS: Rabbits were instrumented with a balloon occluder around a major branch of the left coronary artery for reversible coronary occlusion. Five to seven days after surgery all animals underwent a 30 min coronary occlusion. Animals were randomised to one of seven groups: (1) No additional treatment (control); (2) Ischaemic preconditioning with 5 min regional ischaemia followed by 10 min reperfusion before the 30 min coronary occlusion; (3) and (4) Ischaemic preconditioning followed by 2 or 4 h of reperfusion before the 30 min occlusion, respectively; (5) Treatment with acadesine (2.5 mg.kg-1.min-1 intravenously for 5 min and then 0.5 mg.kg-1.min-1 beginning 45 min before and continuing until 30 min after release of the 30 min occlusion) without ischaemic preconditioning; (6) and (7) Treatment with the higher dose of acadesine for 5 min beginning 35 min before the 5 min ischaemic period, and then the lower dose continuing until 30 min after release of the 30 min coronary occlusion in rabbits with 4 or 6 h reperfusion intervals, respectively. RESULTS: Rabbits with ischaemic preconditioning with 10 min reperfusion preceding the 30 min coronary occlusion (group 2) had only 5.6(SEM 1.1)% infarction of the ischaemic zone. Ischaemic preconditioning followed by 2 h reperfusion (group 3) offered continued protection [18.2(2.2)% infarction] as compared to control animals [37.7(2.6)% infarction]. However, protection waned if ischaemic preconditioning was followed by 4 h reperfusion (group 4) [36.7(3.0)% infarction]. Additionally, treatment with acadesine alone did not modify infarct size (group 7) [39.5(4.0)%], but acadesine largely restored the protection of ischaemic preconditioning despite a 4 h reperfusion interval (group 5) [20.4(3.0)% infarction, P < 0.01 v control]. However, when reperfusion was extended to 6 h (group 6) acadesine could no longer restore protection [36.2(0.9)% infarction]. CONCLUSIONS: The protection afforded by a 5 min ischaemic preconditioning period lasts from 2 to 4 h in the awake, unsedated rabbit, and acadesine can extend the duration of this window of protection to at least 4 h but not to 6 h.     

等离子体聚合有机膜的离子注入改性

以苯胺为单体在等离子体条件下进行聚合，测量所合成的有机膜经１００ｋｅＶ、５×１０１５ｃｍ￣（－２）Ａｒ￣＋注入后电阻率的变化，并用ＦＴＩＲ、ＸＰＳ和核分析方法对离子注入引起的结构和成分改变进行了探讨。结果表明，在等离子体环境下，聚合膜内仍含有大量环结构。由于Ａｒ￣＋的注入，聚合膜表面损伤而碳化，使注入层内的体电阻率降低１１个数量级。     

基于损失自注意力机制的立体匹配算法研究

针对现有的立体匹配算法在阴影、物体边缘和光照反射等区域匹配困难且存在大量错误结果的问题,设计了一种可拆卸的损失自注意力网络（loss self-attention net,LSAnet）查找图像中的匹配困难区域。LSAnet的网络各层相互稠密连接,应用了空洞卷积来扩大感受野,并以立体匹配算法生成的损失分布为标签,能够动态地进行有监督训练,最终生成匹配困难区域掩膜辅助立体匹配网络进行更好的优化;同时,改进了立体匹配网络中经典的特征匹配代价卷结构,降低了后续3D卷积的计算负荷,提高了匹配效率。实验结果表明,该算法相比于基准算法精度更高,并且可以提高算法对于匹配困难区域的鲁棒性。     

基于UAV发动机点火系统的EMI抑制研究

UAV集电子设备于一身,其最大射频电磁干扰(EMI)源自发动机点火系统,它不同程度地影响着机载计算机、自动驾驶仪等以及不同任务载荷的正常工作;通过对某UAV发动机研制的电容放电式点火系统(CDI)的电路和布局描述,研讨了屏蔽对其二次回路影响的机理,合理确定了工程路径,并研究出一种阻尼-屏蔽法实施在CDI中,降低点火系统EMI近20dB,同时不影响其可靠性;本研究的成功,使长期困扰CDI的EMI深入抑制取得了进步。     

基于单片机的多功能充电器的设计与实现

本课题主要研究基于单片机的多功能充电器,解决对不同电池的快速充电。通过对ATmega8的深入研究,利用它的高性能,设计完全实现了电池充电器设计的功能。本设计可以通过按键切换,对的密封铅酸电池(SLA)和镍镉电池(NiCd)进行充电,并通过LCD清楚地看到其充电状态。同时利用放大电路和Atmega8本身的AD转换器显示对电池电压和电流的实时测量,以较好的控制充电过程,保护电池。     

废胶粉改性再生粗骨料沥青混合料性能试验研究

基于将建筑垃圾与废旧橡胶循环使用的目的,研究分别以0％,50％和100％取代率的再生粗骨料替换沥青混合料中10mm～20mm的天然粗骨料后沥青混合料的性能试验,并进一步在不同再生粗骨料取代率的沥青混合料中以干法掺入20％,30％的废胶粉及以湿法掺入20％的废胶粉,研究不同废胶粉掺量及干掺、湿掺废胶粉后再生粗骨料沥青混合料的马歇尔及车辙试验.结果表明,当废胶粉掺量为0％时,随着再生粗骨料取代率的增多,马歇尔稳定度和动稳定度先增多后减小,流值增多,马歇尔模数和残留稳定度减小,而不论是干掺还是湿掺,马歇尔稳定度降低,流值增多,马歇尔模数和残留稳定度降低,但是湿法工艺降低程度低,动稳定度先增多后减小.因此,沥青混合料中建议再生粗骨料取代率低于50％,而废胶粉以低于20％的掺量湿法掺入.