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81.
针对目前爆轰试验中使用的机械密封射频连接器存在的问题.设计了基于玻璃封接技术的新型密封射频连接器结构,并进行了射频反射补偿计算和强度分析.本项研究为爆轰条件下新型密封射频连接器的研制提供了设计依据. 相似文献
82.
83.
文章从四个方面阐述了我院电子科学与技术专业在培养学生科技创新能力方面所实施的多方位培养模式. 相似文献
84.
介绍了平板显示控制芯片TO944的特点、功能、原理,并给出了基于T0944芯片的新型平板显示接口卡的软硬件实现方案,该方案具有电路简单、高可靠性、高性价比、高显示质量等优点,可用于各种不同类型与不同尺寸的平板显示器的显示。 相似文献
85.
The Bisphosphonate Clodronate Modifying Hydroxyapatite Bioceramics for Bone Scaffold 总被引:3,自引:0,他引:3
LUO En HU Jing WEI Shicheng HE Gang XIAO Lin ZHAO Qiang 《武汉理工大学学报(材料科学英文版)》2005,20(B12):98-101
To investigate the efficiency of clodronate modifying HA bioceramics,and to evaluate the effect of clodronate modifying HA bioceramics on the cells in vitro,clodronate modified the porous HA bioceramics for bone scaffold by chelation .The outermost layer of the specimens was analyzd by XPS and FI-IR ,The depth profile was investigated by the argon-ion sputtering method.The cell culture test was conducted using MC3T3-E1 osteoblastic cells,The cells were inoculated and cultured on the scaffolds.Morphological observation of the cells,MTT test and ALP activity test evaluated the cell attachment ,proliferation and activity on the scaffolds.The cell culture test in cell quantity and morphology indicated active proliferation of the cells on the scaffolds.The ALP activity of the cells cultured for 3d and 7d on clodronate-HA bioceramics was slightly higher than that on HA bioceramics ,but the difference was not signifcant,This result indicated that clodronate-HA bioeramics had favorable cytocompatibility to be used as bone scaffold with potential ability to improve asteogensis. 相似文献
86.
The behaviour of hydrogen permeation and diffusion in amorphous alloy Ni68Cr7Si8B14Fe3 hasbeen investigated by an ultrahigh vacuum gas permeation technique. A comparison experimentwas carried out between the as-quenched and annealed States (400℃/2h) of the amorphousalloy. The results show that, for both states of the amorphous alloy in the temperature rangeof 200~350℃, the diffusivity and permeability of hydrogen are in agreement with Arrheniusrelationship, there does not exist H-trapping effect, and the activation energies of diffusion andpermeation almost keep the same. 相似文献
87.
88.
II Kalachev AN Baídus' OA Ivanova EA Ganina IA Boldyrev EA Svetoch 《Canadian Metallurgical Quarterly》1997,(6):32-37
Unlike the glanders agent, the superficial structures of the melioidosis agent were demonstrated to be responsible for marked was immunosuppressive activity. Some antigenic fractions suppressing the blast transformation of lymphocytes, reducing the count of T helpers and profoundly potentiating the infection in vivo were isolated from P. pseudomallei cells. The immunogenic and immunosuppressive activities of both agents' superficial structures were studied by high performance chromatography. Antigenic complexes that were able to protect immunized laboratory animals against fatal infections and to prevent bacterial carriage due to the activation of T cells and to the bacterial activity of macrophages were identified. A composition comprising several immunogens was found to provide an additive protective action against both causative agents. Therefore, the composition may be considered to be a prototype of a molecular antipseudomonadic vaccine. 相似文献
89.
从冷却曲线上相变段倾角最小原则出发,研究了新型激冷剂的配比与热分析法检测铸铁成分精度的关系,实验结果表明,能使相变段倾角最小的成分配比,也能提高对铸铁成分的热分析和检测精度。 相似文献
90.
M Beksa? MS Beksa? VB Tipi HA Duru MU Karakás AN Cakar 《Canadian Metallurgical Quarterly》1997,30(3):145-150
The endothelium participates actively in homeostatic mechanisms such as the regulation of vascular tone and maintenance of a nonthrombotic environment, as well as directing biological responses such as leukocyte trafficking to inflammatory sites. Disruption of these processes leads to disease. In the antiphospholipid antibody syndrome autoantibodies provoke the endothelium to develop a prothrombotic surface. In systemic vasculitides associated with presence of antineutrophil cytoplasm antibodies, it is likely that the autoantibodies incite premature neutrophil activation, disrupted neutrophil-endothelium interactions and endothelial damage. This review considers how normal endothelial functions may be subverted in disease and how active endothelial responses may contribute to disease. 相似文献