Superconductivity and magnetic interactions in Yb1.2−x Eu x Mo6S8 pseudoternary compounds

The superconducting and magnetic properties of the Yb_1.2–x Eu _x Mo₆S₈ series of compounds have been investigated by means of ac magnetic susceptibility, electrical resistivity, and upper critical magnetic fieldH _c2 measurements. The superconducting transition temperature at ambient pressure is depressed by Eu additions, until superconductivity disappears completely atx0.7. For Eu concentrationsx<0.45,H _c2 is enhanced at low temperatures and exceeds the values for pure Yb_1.2Mo₆S₈. The temperature dependence ofH _c2 for the various Yb_1.2–x Eu _x Mo₆S₈ compounds is analyzed using a multiple pair-breaking theory that includes the compensation of the applied magnetic field by the negative exchange field produced by antiferromagnetic interactions between the conduction electron spins and the Eu²⁺ spins (Jaccarino-Peter effect). Hydrostatic pressure was found to induce superconductivity in all Eu-rich compositions that were not superconducting at ambient pressure.     

On the thermal stability of xanthan gum

The thermal stability of xanthan gum in dilute aqueous solutions at 90°C is considered. The relative viscosity as a function of ageing time is discussed, and it has been found to depend on the polymer concentration and conformation as well as on the salt content. The effectiveness of a quencher demonstrates the existence of a free-radical process in the degradation. During ageing, the molecular weight first decreases by rapid random hydrolysis of the main chain and loss of the pyruvate and acetate substituents. Later, oligomers are formed corresponding to a breakdown of both the side chain and the main chain. The identification of cellodextrins in the oligomer pool demonstrates the second effect.     

Efficient Reduction of NO x by H2 Under Oxygen-Rich Conditions over Pd/TiO2 Catalysts: An in situ DRIFTS Study

The H₂/NO/O₂ reaction under lean-burn conditions has been studied by means of in situ DRIFTS, reactor measurements and temperature-programmed desorption with the aim of understanding the very different behavior of Pd/TiO₂ and Pd/Al₂O₃ catalysts. The former deliver very high NO _x conversions (70-80%) with good N₂ selectivity whereas the latter show very low activity. In addition, PdTiO₂ exhibits two distinct NO _x reduction pathways, thus greatly extending the useful temperature range. It is shown that the PdTiO₂ low-temperature channel involves adsorption and subsequent dissociation of NO on reduced (Pd⁰) metal sites. The low activity of PdAl₂O₃ is a consequence of palladium remaining in an oxidized state under reaction conditions. The high-temperature NO reduction channel found with PdTiO₂ is associated with the generation and subsequent reaction of NH _x species.     

Architectures for Fast Transcoding of H.264/AVC to Quality-Scalable SVC Streams

The scalable extension of H.264/AVC (SVC) was recently standardized, and offers scalability at a minor penalty in rate-distortion efficiency when compared to single-layer H.264/AVC coding. In SVC, a scaled version of the original video sequence can easily be extracted by dropping layers from the stream. However, most of the video content nowadays is still produced in a single-layer format. While decoding and reencoding is a possible solution to introduce scalability in the existing bitstreams, this is an approach which requires a tremendous amount of time and effort. In this paper, we show that transcoding can be used to intelligently derive scalable bitstreams from existing single-layer streams. We focus on SNR scalability, and introduce techniques that are able to create multiple quality layers in the bitstreams. We also discuss bitstream rewriting from SVC to H.264/AVC, and examine how our newly proposed architectures can benefit from the changes that were introduced for bitstream rewriting. Architectures with different rate distribution flexibility and computational complexity are discussed. Rate-distortion performance of transcoding is shown to be comparable to that of reencoding at a fraction of the time needed for the latter.     

Neutrophil apoptosis is associated with a reduction in CD16 (Fc gamma RIII) expression

Resolution of inflammation involves removal of recruited neutrophils from inflamed sites via a noninflammatory mechanism, possibly involving neutrophil apoptosis and engulfment/phagocytosis by macrophages. In this study, we describe the reduction in surface expression (> 90%) of the neutrophil molecule Fc gamma RIII (CD16) during in vitro culture at 37 degrees C, which was found to be temporally associated with the appearance of neutrophils with apoptotic morphology during in vitro culture and inhibitable by granulocyte-macrophage colony-stimulating factor (GM-CSF), which postpones apoptosis in the neutrophil. By using dual fluorescence analysis, CD16 "low" expressing neutrophils showed reduced staining with the DNA-binding dye propidium iodide, suggesting that CD16 low expressing neutrophils were apoptotic. Separation of CD16 "high" and CD16 "low" expressing neutrophils by fluorescence-activated cell sorting revealed that morphologically apoptotic cells exhibited the CD16 low phenotype. We did not observe similar marked changes in expression of other neutrophil surface molecules (including other phosphatidylinositol (PI)-linked molecules), indicating that generalized loss of surface molecules does not occur during apoptosis. We believe this to be the first reported cell type-specific membrane alteration in a surface glycoprotein associated with apoptosis, suggesting that the program of cell death in the neutrophil, in addition to morphologic and nuclear changes, includes alterations in expression of surface receptors.     

High-resolution crystal structures of human hemoglobin with mutations at tryptophan 37beta: structural basis for a high-affinity T-state,

The high-resolution X-ray structures of the deoxy forms of four recombinant hemoglobins in which Trp37(C3)beta is replaced with Tyr (betaW37Y), Ala (betaW37A), Glu (betaW37E), or Gly (betaW37G) have been refined and analyzed with superposition methods that partition mutation-induced perturbations into quaternary structure changes and tertiary structure changes. In addition, a new cross-validation statistic that is sensitive to local changes in structure (a "local Rfree" parameter) was used as an objective measure of the significance of the tertiary structure changes. No significant mutation-induced changes in tertiary structure are detected at the mutation site itself for any of the four mutants studied. Instead, disruption of the intersubunit contacts associated with Trp37(C3)beta results in (1) a change in quaternary structure at the alpha1beta2 interface, (2) alpha subunit tertiary structure changes that are centered at Asp94(G1)alpha-Pro95(G2)alpha, (3) beta subunit tertiary structure changes that are located between residues Asp99(G1)beta and Asn102(G4)beta, (4) increased mobility of the alpha subunit COOH-terminal dipeptide, and (5) shortening of the Fe-Nepsilon2His(F8) bond in the alpha and beta subunits of the betaW37G and betaW37E mutants. In each case, the magnitude of the change in a particular structural parameter increases in the order betaW37Y < betaW37A < betaW37E approximately betaW37G, which corresponds closely to the degree of functional disruption documented in the preceding papers.     

Extrarenal rhabdoid tumors of soft tissue: a clinicopathologic and immunohistochemical study of 18 cases

Rhabdoid tumor is a well-accepted clincopathologic entity among childhood renal neoplasms; similar tumors have been described in extrarenal locations. We present the clinicopathologic profile and the immunohistochemical features of a series of soft tissue rhabdoid tumors. Twenty-eight cases coded as extrarenal rhabdoid tumor (ERRT), RT, possible ERRT, and "large cell sarcoma" were retrieved from the Armed Forces Institute of Pathology soft tissue registry. The tumors were reclassified according to strict criteria by light microscopy, clinical information, immunohistochemistry, and, in some cases, electron microscopy. Soft tissue rhabdoid tumor (STRT) was defined as (1) a tumor composed of noncohesive single cells, clusters, or sheets of large tumor cells with abundant glassy eosinophilic cytoplasm, an eccentric vesicular nucleus, and an extremely large nucleolus; (2) positivity for vimentin and/or cytokeratin or other epithelial markers by immunostaining; and (3) exclusion of other tumor types with rhabdoid inclusions (melanoma, other sarcomas, carcinoma). Eighteen cases met our criteria for soft tissue rhabdoid tumors. The median patient age was 13 years (range, 6 months to 56 years). Ninety-four percent of STRT cases were positive for vimentin and 59% for pan-cytokeratin. Sixty-three percent and 60% were positive for CAM 5.2 and EMA, respectively. Seventy-nine percent stained for at least one epithelial marker; 76% stained for both vimentin and epithelial markers simultaneously. Forty-two percent stained for MSA, and 14% for CEA and SMA. CD99, synaptophysin, CD57 (Leu-7), NSE, and focal S100 protein were identified in 75%, 66%, 56%, 54%, and 31% of the STRT cases, respectively. All STRT cases examined were negative for HMB-45, chromogranin, BER-EP4, desmin, myoglobin, CD34, and GFAP. Follow-up examination in 61% of the STRT patients revealed that 64% of patients died of disease within a median follow-up interval of 19 months (range, 4 months to 5 years); 82% had metastases to lung, lymph nodes, or liver; 22% had local recurrences before metastasis; and 18% were alive without known disease status (median, 5.5 years). Soft tissue rhabdoid tumor is a highly aggressive sarcoma, predominantly of childhood. Besides having nearly consistent coexpression of vimentin and epithelial markers, STRTs show positivity for multiple neural/neuroectodermal markers that overlap with those of primitive neuroectodermal tumor.