面向制造业的RFID复杂事件处理

在自动数据采集中,如何维持无线射频识别(RFID)技术对海量数据的处理效率极为重要。针对RFID数据的特点以及目前RFID数据处理方法的不足,提出一种基于复杂事件处理的RFID数据处理模型,并提供相关的定义,阐述其功能模块及解决方案。该模型实现了从大量的RFID原始数据中提取出对应用程序有意义的事件。最后以面向制造业的RFID应用为例对基于复杂事件处理技术的RFID数据处理模型进行了研究并说明了其应用优势。     

电解金属锰合格液中锰镁的萃取分离研究

利用P507-磺化煤油体系对电解金属锰合格液中的锰镁组分进行了萃取研究。分别对硫酸铵的浓度,P507的体积分数,皂化率,相比以及反萃工艺中的各个参数进行了探讨。结果表明:在硫酸铵浓度为78.00 g/L,P507体积分数为30%,皂化率为20%,相比为1∶1等条件下,镁的萃取率达到48.57%,锰的萃取率达到了75.00%;反萃过程中镁的最高反萃率为45.84%,但锰的反萃率均为零。该研究为电解金属锰合格液中锰镁的萃取分离提供了必要的理论依据,对我国电解金属锰生产工艺的完善有较大的促进作用。     

Quantification by flow cytometry of chromosome-17 deletions in Smith-Magenis syndrome patients

BACKGROUND: Previous studies have suggested varying molecular weights for mast cell derived tumour necrosis factor alpha (TNF alpha ) and little data exist upon the factors which may regulate the control of this cytokine in these cells. OBJECTIVE: To determine the molecular weight of canine mastocytoma-derived TNF alpha, to determine whether it is pre-formed within the granule and whether is expression could be up-regulated by stem cell factor (SCF). METHODS: Molecular sizing was assessed by immunoblot. The cellular localization of the TNF alpha was determined by immunocytochemistry before and after stimulation by A23187 and passive sensitization. Subcellular localization was performed by immunogold immunocytochemistry. Changes in the level of mastocytoma mRNA for TNF alpha in response to stimulation with SCF or fibroblast conditioned media for up to 12 weeks were studied using Northern analysis and changes in the level of TNF alpha protein expression on immunoblot and immunocytochemistry. RESULTS: Mast cells contained authentic 17 kDa TNF alpha as identified by immunoblotting. Immunocytochemical studies demonstrated preformed TNF alpha which was released by stimulation with antigen after passive sensitization, or by the calcium ionophore A23187. Further confirmation of the preformed nature of this TNF alpha was provided by immunogold electron microscopy which localized this cytokine to the granule of the inactive mast cell. Northern blotting revealed a constitutive message for TNF alpha, which increased in response to fibroblast conditioned media (FCM) and to recombinant human SCF. Immunocytochemical studies of mast cells cultured long-term with FCM or with recombinant SCF showed increased expression of TNF alpha over the course of 12 weeks incubation with these stimuli. CONCLUSION: Mastocytoma derived-TNF alpha is a preformed, granule-associated 17 kDa cytokine which is released on stimulation with A23187 or passive sensitization. It is up-regulated by stem cell factor and by FCM over the course of 12 weeks.     

多层建筑物的室内场强预测模型的探讨

随着移动通信的发展,室内覆盖问题变得越来越突出起来。利用几何光学理论对同一楼层的传播环境下无线电波的传播进行物理建模;对多楼层传播环境,给出经验性的衰减因子模型,为工程实践中多层建筑物的室内场强预测和移动网络规划提供参考。     

FPGA实现红外焦平面的非均匀性校正

红外焦平面阵列(IRFPA)普遍存在着响应度的非均匀性问题，极大的限制了凝视红外系统的探测性能，本文先分析了红外焦平面阵列非均匀性的成因，然后讨论了非均匀性校正的方法，最后针对热释电红外焦平面非均匀性产生的特点，提出了用FPGA实现其非均匀性校正的方案。     

DDL—Ⅲ张力计与国产SKSD—B系统的挂接

国产ＳＫＳＤ－Ｂ数控生产测井地面系统是装载在原吉尔哈特公司生产的ＤＤＬ－Ⅲ型上的ＤＤＬ－Ⅲ无配套的井口张力系统，文章主要介绍将ＤＤＬ－Ⅲ张力计挂接到ＳＫＳＤ－Ｂ上，使张力信息传输到ＳＫＳＤ－Ｂ地面系统并记录下来，供有关人员观察井下仪器在井内的运行及遇阻，遇卡情况，通过室内测试和现场试验，其结果较好地反映了实际情况，说明挂接是成功的。     

G＊DAPS4—BU充电仪的研制

针对G DAPS 4数字地震仪充电机存在的问题 ,研制成功G DAPS4 BU充电仪。通过开关电源和逻辑控制电路 ,采用脉冲方式进行充电 ,并设计了充放电自动转换装置 ,解决了恒流充电的实际问题 ,消除了“记忆效应”的不良影响。充电性能与原充电机相同 ,同时具有结构简单、合理 ,降低了生产成本 ,解决了生产难题 ,具有较高的实用价值     

Panniculectomy: a useful technique for the obese patient undergoing gynecological surgery

BACKGROUND: The inner medullary collecting duct (IMCD) is composed of at least two functionally and morphologically distinct segments, the initial (IMCDi) and the terminal (IMCDt) portions. However, most studies of receptor signaling have been performed on cells obtained from the entire inner medulla. The purpose of this study was to determine whether the patterns of receptor-activated cAMP accumulation were different between these segments. METHODS: We measured cAMP accumulation stimulated by vasopressin and isoproterenol, and the effect of epinephrine in freshly dissected IMCDi and IMCDt segments cultured and IMCDi and IMCDt cells in primary culture. RESULTS: The maximum response to vasopressin was twofold higher in fresh IMCDt verus IMCDi (P < 0.05), however, it increased in cultured IMCDi by 40% verus fresh cells with no change in the response in fresh verus cultured IMCDt. The maximum response to isoproterenol was small in fresh cells but increased by five- and sixfold, respectively, in cultured IMCDi and IMCDt cells. alpha 2-Adrenoceptor stimulation almost completely inhibited both vasopressin and isoproterenol-stimulated cAMP accumulations in fresh IMCDi and IMCDt cells, but only partially inhibited either accumulation by 34 to 49% in cultured cells. CONCLUSIONS: (1) IMCDi and IMCDt cells are both subject to vasopressin and alpha 2- and beta-adrenergic regulation of adenylyl cyclase activity; (2) the relative influence of beta-adrenergic, alpha 2-adrenergic and V2 receptors to affect cAMP accumulation is altered in primary culture versus freshly dissected IMCD segments, suggesting that caution must be exercised in the extrapolation of data from cultured IMCD cells to in vivo models.     

Nephrotoxic potential of 2-amino-5-chlorophenol and 4-amino-3-chlorophenol in Fischer 344 rats: comparisons with 2- and 4-chloroaniline and 2- and 4-aminophenol

Nephrotoxicity occurs following intraperitoneal (i.p.) administration of 2-chloroaniline or 4-chloroaniline hydrochloride to Fischer 344 rats, but the nephrotoxicant chemical species and mechanism of nephrotoxicity are unknown. The purpose of this study was to evaluate the in vivo and in vitro nephrotoxic potential of 2-amino-5-chlorophenol and 4-amino-3-chlorophenol, metabolites of 4-chloroaniline and 2-chloroaniline. A comparison was also made between the nephrotoxic potential of the aminochlorophenols and the corresponding aminophenols to examine the effect of adding a chloride group on the nephrotoxic potential of the animophenols. Male Fischer 344 rats (4/group) were given an i.p. injection of a chloroaniline or aminochlorophenol hydrochloride (1.5 mmol/kg), and aminophenol (1.0 or 1.5 mmol/kg), or vehicle, and renal function monitored at 24 and 48 h. Both aminochlorophenols induced smaller and fewer renal effects that the parent chloroanilenes in vivo. Also, 4-aminophenol was markedly more potent as a nephrotoxicant that 4-amino-3-chlorophenol, while 2-aminophenol and 2-amino-5-chlorophenol induced only mild change in renal function. In vitro, the phenolic compounds reduce p-aminohippurate accumulation by renal cortical slices at bath concentrations of 0.01 mM, while a bath concentration of 0.50 mM or greater was required for the chloroanilines. However, all compounds reduced tetraethylammonium accumulation at bath concentrations of 0.1-0.5 mM or greater. These results indicate that extrarenally-produced aminochlorophenol metabolites do not contribute to the mechanism of chloroaniline nephrotoxicity. Also, the reduced nephrotoxic potential of 4-amino-3-chlorophenol compared to 4-aminophenol could result from an altered ability of the aminochlorophenol to redox cycle or form conjugates.