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81.
Current therapy does not cure the majority of patients with B cell non-Hodgkin's lymphoma (NHL) and further intensification does not benefit the patient. Therefore, new approaches are necessary. Immunotherapy has become again a major interest as a new treatment modality for B cell lymphoma since the discovery that the lymphoma specific Id can be presented to antigen-specific T cells. Vaccination of the tumour-bearing host is one of the major strategies to induce a T cell mediated anti-tumour immunity in vivo. For B cell lymphomas the lymphoma specific Id can be used as a tumour-specific antigen to stimulate T cells. Alternatively, the malignant B cells can be modified to become efficient antigen presenting cells (APCs) and present peptides from their own tumour-specific antigens to the autologous T cells. Currently explored and future vaccination strategies for B cell lymphoma will be discussed here. 相似文献
82.
Human Myt1 is a cell cycle-regulated kinase that inhibits Cdc2 but not Cdk2 activity 总被引:1,自引:0,他引:1
Activation of the Cdc2.cyclin B kinase is a pivotal step of mitotic initiation. This step is mediated principally by the dephosphorylation of residues threonine 14 (Thr14) and tyrosine 15 (Tyr15) on the Cdc2 catalytic subunit. In several organisms homologs of the Wee1 kinase have been shown to be the major activity responsible for phosphorylating the Tyr15 inhibitory site. A membrane-bound kinase capable of phosphorylating residue Thr14, the Myt1 kinase, has been identified in the frog Xenopus laevis and more recently in human. In this study, we have examined the substrate specificity and cell cycle regulation of the human Myt1 kinase. We find that human Myt1 phosphorylates and inactivates Cdc2-containing cyclin complexes but not complexes containing Cdk2 or Cdk4. Analysis of endogenous Myt1 demonstrates that it remains membrane-bound throughout the cell cycle, but its kinase activity decreased during M phase arrest, when Myt1 became hyperphosphorylated. Further, Cdc2. cyclin B1 was capable of phosphorylating Myt1 in vitro, but this phosphorylation did not affect Myt1 kinase activity. These findings suggest that human Myt1 is negatively regulated by an M phase-activated kinase and that Myt1 inhibits mitosis due to its specificity for Cdc2.cyclin complexes. 相似文献
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M. Saqib I. Weiss G. M. Mehrotra E. Clevenger A. G. Jackson H. A. Lipsitt 《Metallurgical and Materials Transactions A》1991,22(8):1722-1728
Ti-43Al (atomic percent) alloy containing a dispersion of 7 vol pct TiB2 particles was exposed to various thermal treatments to determine the stability of TiB2 in an ⇌2 + β-phase matrix. No new phases were detected at the particle/matrix interfaces even after thermal exposure at 1473 K for
7 days. The absence of an Al peak in the energy dispersive X-ray analysis system (EDS) spectra from TiB2 particles chemically extracted from the specimens aged at 1473 K for 7 days indicated no diffusion of Al from the matrix
to the particles. These results indicate that TiB2 is stable in an α2+ β matrix at 1473 K.
E. Clevenger, formerly Undergraduate Student, Department of Mechanical and Materials Engineering, Wright State University 相似文献
86.
Ravi Mehrotra 《Journal of Low Temperature Physics》1987,68(3-4):161-168
The shear viscosity of a classical two-dimensional (2D) electron liquid is estimated by adapting the theory of Kirkwood, Buff, and Green for three dimensions to two dimensions. It is found to be large enough so that shear modes, if not overdamped by other scattering mechanisms, should be able to propagate through the electron liquid above a minimum temperature-dependent frequency, which is a small fraction of the highest frequency in the corresponding 2D electron solid. 相似文献
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Rósa Jónsdóttir Margrét Bragadóttir GudmundurÖRN Arnarson 《Journal of food science》2005,70(7):c433-c440
The stability of microencapsulated fish oil was studied during storage at 4 °C for up to 20 wk. Different coating mixtures consisting of gelatin or caseinate in blends with carbohydrates (sucrose, lactose, maltodextrin) were investigated. Oxidative stability of the microencapsulated fish oil was monitored by analysis of volatile compounds using gas chromatography olfactometry (GC‐O) or GC flame ionization (GC‐FID) (SPME‐HS‐GC/O or GC/ FID and HS‐GC/MS), Oxipres test, thiobarbituric acid‐reactive substances (TBARS), and sensory analysis. Coating mixture of caseinate and lactose showed slightly better stability than the sucrose and maltodextrin caseinate mixtures. Combination of fish gelatin and maltodextrin did not show as good oxidative stability as the coating blend of caseinate, lactose, and lecithin. Hexanal, 2‐nonenal and 2,4‐decadienals were selected as quality indicators to monitor the lipid oxidation during storage of the samples. SPME‐GC‐O analysis of these indicators showed that they were representative for the oxidation occurring in the microencapsulated fish oil. SPME‐GC‐FID analysis was sensitive enough to detect oxidative changes during storage. Oxidative stability test, TBARS results, and sensory analysis were in agreement with the SPME, indicating that SPME (polydimethylsiloxane/divinylbenzene [PDMS/ DVB] fiber) can be a useful tool for rapid analysis of lipid oxidation in microencapsulated fish oil. 相似文献
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