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Effects of leukotriene D on the airways in asthma 总被引:2,自引:0,他引:2
M Griffin JW Weiss AG Leitch ER McFadden EJ Corey KF Austen JM Drazen 《Canadian Metallurgical Quarterly》1983,308(8):436-439
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Drazen Jurisic Neven Mijat George S. Moschytz 《International Journal of Circuit Theory and Applications》2013,41(1):15-32
A new and straightforward design procedure for simple canonical topologies of allpole, active‐RC, low‐selectivity band‐pass (BP) filters, with low sensitivity to component tolerances is presented. The procedure is primarily intended for discrete‐component, low‐power filter applications using just one amplifier for relatively high‐order filters. The design procedure starts out with an ‘optimized’ low‐pass (LP) prototype filter, yielding an ‘optimized’ BP filter, whereby the wealth of ‘optimized’ single‐amplifier LP filter designs can be exploited. Using a so‐called ‘lossy’ LP–BP transformation, closed‐form design equations for the design of second‐ to eighth‐order, single‐amplifier BP filters are presented. The low sensitivity, low power consumption, and low noise features of the resulting circuits, as well as the influence of the finite gain‐bandwidth product and component spread, are demonstrated for the case of a fourth‐order filter example. The optimized single‐opamp fourth‐order filter is compared with other designs, such as the cascade of optimized Biquads. Using PSpice with a TL081 opamp model, the filter performance is simulated and the results compared and verified with measurements of a discrete‐component breadboard filter using 1% resistors, 1% capacitors, and a TL081 opamp. Copyright © 2011 John Wiley & Sons, Ltd. 相似文献
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N Noviski JP Brewer WA Skornik SJ Galli JM Drazen TR Martin 《Canadian Metallurgical Quarterly》1999,86(1):202-210
Exposure to ambient ozone (O3) is associated with increased exacerbations of asthma. We sought to determine whether mast cell degranulation is induced by in vivo exposure to O3 in mice and whether mast cells play an essential role in the development of pulmonary pathophysiological alterations induced by O3. For this we exposed mast cell-deficient WBB6F1-kitW/kitW-v (kitW/kitW-v) mice and the congenic normal WBB6F1 (+/+) mice to air or to 1 or 3 parts/million O3 for 4 h and studied them at different intervals from 4 to 72 h later. We found evidence of O3-induced cutaneous, as well as bronchial, mast cell degranulation. Polymorphonuclear cell influx into the pulmonary parenchyma was observed after exposure to 1 part/milllion O3 only in mice that possessed mast cells. Airway hyperresponsiveness to intravenous methacholine measured in vivo under pentobarbital anesthesia was observed in both kitW/kitW-v and +/+ mice after exposure to O3. Thus, although mast cells are activated in vivo by O3 and participate in O3-induced polymorphonuclear cell infiltration into the pulmonary parenchyma, they do not participate detectably in the development of O3-induced airway hyperresponsiveness in mice. 相似文献
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A comparison was made between the use of data processing in hospitals and Health Maintenance Organizations (HMOs). Findings indicate that the HMO and hospital data processing markets are currently quite distinct. As expected, the functions automated reflect the uses of the different care delivery systems. The hardware and software vendors to these markets are also different. A high percentage (50%) of HMOs are using software they developed in-house. In this respect, current HMO data processing is similar to the hospitals of a decade ago. The hospital market (for hardware and total systems) is highly concentrated. A few firms have a combined market share of over 50% and there are a large number of firms with very small shares. In the HMO market, there is a high concentration ratio for hardware but not for software or systems. HMOs and hospitals are almost uniformly satisfied with their hardware. Satisfaction with most applications is between 80 and 90% but there is a need, at least within HMOs, for improvements in support of the software supplied. 相似文献
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Shyam B. Karki Varaporn Treemaneekarn Drazen Ostovic Maneesh Nerurkar Örn Almarsson 《Drug development and industrial pharmacy》2013,39(3):327-334
ABSTRACTThe chemical degradation of N-(glutaryl-hyp-ala-ser-cyclohexylglycyl-gln-ser-leu)-doxorubicin (henceforth referred to as doxorubicin peptide conjugate 1) was studied in buffered aqueous solution. The pH-rate profile of degradation shows that the doxorubicin conjugate is most stable between pH 5 and 6. The dependence of log kobsd on pH in acidic medium is characteristic of specific acid-catalysis of the sugar hemiaminal of 1 (as in the case of doxorubicin). Isolation of degradates and structural determination shows that the degradation at lower pH values yields the water-insoluble aglycone doxorubicinone, supporting the mechanism of acid-catalyzed loss of the amino sugar. At pH higher than 5, a more complicated degradation pattern is observed, including the loss of the amino sugar and the aromatization of the saturated ring to give 7,8-dehydro-9,10-desacetyldoxorubicinone as one of the major products. Around the pH of maximum stability in solution, the rate of degradation of 1 is significantly greater than that for doxorubicin, which rules out the formulation of a room temperature solution product with a sufficiently long shelflife for market use. Design of a stable lyophilized formulation for sterile reconstitution based on the physicochemical properties of 1 is described. 相似文献