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101.
广水市位于湖北省东北部,地处大别山与桐柏山交汇处南麓,地跨长江、淮河两大流域,国土面积2647km^2,是湖北省38个山区县市之一。 相似文献
102.
深大露天矿的间断向半连续开采工艺过渡时,合理深度及半连续开采工艺的移设步距是两个重要的开采参数,对矿山的经济效果影响巨大,本文首次提出采用组合台阶单位体的开采来分析确定这两个重要的参数,得到的结论在霍林河煤矿的应用中取得了满意的结果,对其它类似矿山具有应用价值。 相似文献
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采用稀土氧化物和无机化合物的复合物(不简称RI-复合物)作为搪瓷釉浆悬浮剂,使之全部或大部分代替粘土,进行了一系列试验,已获得成功,突破了长期来搪瓷工业离不开粘土的传统工艺,并实现了工业化生产。 相似文献
105.
本文从理论上推导出了桩柱式采金船每帮挖方量计算的数学模型并,可广泛地用于设计与生产中,从而提高采金船设计的准确性,以及对生产矿山进行矿量、回收率和实收品位等采选指标收测的可靠性。 相似文献
106.
新一代银行大型机网络接入技术研究 总被引:1,自引:0,他引:1
银行大型机网络接入技术的转型是当前一个非常重要的研究与应用课题。文章在成功实现银行大型机网络接入技术转型的基础上,分析了相关新技术,解决了转型中的关键问题,提出并实现了一种稳定高效的银行大型机网络接入技术方案,对银行大型机网络接入技术的转型具有指导意义,对银行信息化技术研究具有重要参考价值。 相似文献
107.
Hart D. Novosel D. Calero F. Udren E. LiFeng Yang 《Power Delivery, IEEE Transactions on》1996,11(3):1274-1284
This paper describes the testing of a new power system numerical relaying concept. The new technique is applied to distance relaying to offer improved reliability and security of the relay, and improve the speed of relay operation. To validate the algorithm's performance, the scheme was analyzed under a variety of test conditions and compared with conventional relay concepts. This paper presents the results of the testing and shows that the new relaying scheme operated with a high level of security and dependability for all of the test cases. The tests show improvements that the new scheme can offer in comparison to the conventional schemes 相似文献
108.
In the present study, we examined the effect of the thromboxane/prostaglandin endoperoxide analogue U46619 on proliferation and hypertrophy in cultured rat vascular smooth muscle cells and the roles of protein kinase C and transforming growth factor-beta (TGF-beta) in the mediation of the hypertrophic response to U46619. Since an increase in basic fibroblast growth factor (bFGF) was previously shown to mediate the hypertrophic response to U46619, we also assessed the relationship between bFGF and TGF-beta in the expression of U46619 actions. U46619 increased [35S]methionine incorporation into protein and protein content of vascular smooth muscle cells but had no effect on cell number. A role for TGF-beta was supported by the following observations: (1) exogenous human TGF-beta 1 increased protein synthesis; (2) antibody to TGF-beta blocked both TGF-beta- and U46619-induced increases in protein content; (3) U46619 increased active and total TGF-beta bioactivities; and (4) the actions of U46619 on protein content and TGF-beta bioactivity were blocked by the thromboxane/prostaglandin endoperoxide receptor antagonist SQ 29,548. Previous observations had demonstrated a role for bFGF in the expression of U46619 actions on protein synthesis. Results of the present study suggest that TGF-beta and bFGF interact in mediating the protein synthetic response to U46619. First, the concentration of exogenous TGF-beta (10 pmol/L) alone required to produce a protein synthetic response equivalent to that induced by U46619 was much higher than the concentration of endogenous active TGF-beta that accumulated in the media in response to U46619 (0.7 pmol/L). Second, bFGF (20 ng/mL) increased total TGF-beta bioactivity and stimulated protein synthesis. The hyper-trophic response to bFGF was blocked by anti-TGF-beta. The ability of U46619 and bFGF to increase protein synthesis and protein content in vascular smooth muscle cells was associated with TGF-beta-induced suppression of proliferation, as evidenced by the ability of antibody to TGF-beta to enhance U46619- and bFGF-induced increases in [3H]thymidine incorporation into DNA. Results of the present study also supported a role for protein kinase C in the expression of U46619 and bFGF actions. U46619 increased protein kinase C activity in the particulate fraction of vascular smooth muscle cells. Moreover, the protein kinase C inhibitors GF109203X and staurosporine blocked U46619- and bFGF-induced increases in protein synthesis as well as active and total TGF-beta bioactivities. By contrast, the protein kinase C inhibitors did not prevent the increases in protein synthesis induced by exogenous TGF-beta. The results demonstrate that thromboxane/prostaglandin endoperoxide signals increased TGF-beta bioactivity via protein kinase C. Increases in both bFGF and TGF-beta are required for an optimal hypertrophic response to U46619. The hypertrophic response to TGF-beta occurs through a protein kinase C-independent pathway. 相似文献
109.
110.