SO3和C3A对水泥膨胀性能的影响

本文研究了ＳＯ３和Ｃ３Ａ对水泥浆体膨胀的影响。对于某一较早的龄期，当ＳＯ３掺量超过某一数值时，水泥浆体的膨胀与ＳＯ３掺量关系不大，而提高水泥的Ｃ３Ａ含量则能显著地增加其膨胀。本文还探讨了ＳＯ３和Ｃ３Ａ影响水泥浆体膨胀的机理。     

HYSIM软件模拟天然气加工工艺流程的研究(Ⅰ)——系统中无循环物流

本文结合大庆油田天然气矿场加工实际生产装置,讨论了应用HYSIM烃类工艺模拟软件对含有不确定组分的体系进行流程模拟计算的特点、塔设备严格模拟计算过程中输入规定的选择、以及多股流换热器和吸附脱水工艺的模拟计算方法。     

光学全交叉网络与SW榕树（F＝S＝2）网络拓扑等价的多样性

本文对有关互连网络拓扑等价的分析方法作了简单评述。并采用互连网络拓扑等价的图分析法分析了自由空间光学互连全交叉网络与ＳＷ榕树网络的拓扑等价及其多样性，拓展了全交叉网络在数字光计算、光子交换及多处理机系统的潜在应用。     

激光诱导H_2S催化裂解制备H_2的研究

在室温常压静态条件下研究了以Ｖ２Ｏ５／ＴｉＯ２为催化剂，紫外脉冲激光诱导Ｈ２Ｓ催化分解为Ｈ２和Ｓ的反应及其机理。考察了产氢量与激光不同照射条件之间的关系。     

多层气敏薄膜表面吸附及界面扩散的研究

制备了ＳｎＯ３／ＺｎＯ及ＺｎＯ／ＳｎＯ３多层结构的气敏薄膜，用能谱结合氩离子刻蚀的方法及Ｘ射线衍射法，对薄膜的表面吸附。膜间的相互与组成等进行了研究，结果表明：薄膜表面存在少量的吸附多层膜中锌的扩散远比锡一个模型，对吸附现象作了初步的解释，讨论了造成锌锡扩散的原因。     

GaAs FET辐射退化研究

低噪声ＧａＡｓ ＦＥＴ和功率ＧａＡｓ ＦＥＴ经中子辐射都未发生致命性的通或断失效，直流偏置伏态下，其失效模式仅仅表现为源漏饱和电流ＩＤＳＳ的退化降低，据此提出了ＩＤＳＳ退化失效与快中子注量φｎ之间的解析关键式ｌｎｙ＝ａ＋ｂｌｎφ。     

统筹城乡发展全面推进城乡一体化

两年前,义乌市委、市政府认真贯彻党的十六大精神,顺应广大群众的呼声,按照统筹城乡发展的思路,编制了《义乌市城乡一体化行动纲要》,在全市全面部署开展城乡一体化行动,已取得初步成效,赢得了社会各界的赞誉。     

N-Methyl-D-aspartate attenuates opioid receptor-mediated G protein activation and this process involves protein kinase C

The effects of N-methyl-D-aspartate (NMDA) on opioid receptor-mediated G protein activation were explored in neuroblastoma X glioma hybrid (NG108-15) cells. Treatment of the cells with NMDA resulted in a remarkable attenuation of [35S]guanosine-5'-O-(3-thio)triphosphate binding stimulated by [D-Pen2,D-Pen5]-enkephalin (DPDPE), a delta-opioid receptor agonist. The effects of NMDA were dose and time dependent with an IC50 value of 5 nM and could be blocked by NMDA receptor antagonists. After NMDA treatment, the DPDPE dose-response curve shifted to the right (EC50 value increased approximately 7-fold, from 6 to 40 nM), and the maximal response induced by DPDPE was reduced by approximately 60%. The effects of NMDA were reversible, and the DPDPE response could recover within 60 min. The functional responses of delta-, mu-, and kappa-opioid receptors in primarily cultured neurons also were attenuated significantly by NMDA treatment. The inhibitory effects of NMDA on opioid receptor-mediated G protein activation could be blocked by coadministration of the protein kinase C (PKC) inhibitors or by elimination of the extracellular Ca2+. Correspondingly, NMDA treatment of NG108 cells significantly elevated cellular PKC activity and stimulated Gialpha2 phosphorylation. Transient transfection into NG108-15 cells of the wild-type Gialpha2 and a mutated Gialpha2 (Ser144Ala) resulted in a 2-fold increase in DPDPE-stimulated G protein activation. The DPDPE responses were greatly inhibited by NMDA treatment in the wild-type Gialpha2-transfected cells but much less affected in the mutant Gialpha2-transfected cells. In summary, NMDA attenuates opioid receptor/G protein coupling, and this process requires activation of PKC.