Preparation and characterization of cuprous iodide nanoparticles

A new technique for the preparation of CuI nanoparticles from CuSO₄ and KI ethanol solutions has been developed. Preparation conditions were optimized through a series of experiments. Under these conditions, the yield of CuI reached 95.39%. The product was characterized and the reaction kinetics was studied. The results show that the product takes a roughly spherical shape with an average particle size of less then 50 nm. The activation energy of the formation of CuI is found to be E _a = 0.58 × 10² kJ/mol, and the preexponential factor in the Arrhenius equation is k ₀ = 7.43 × 10¹⁶ mol/(l s). The text was submitted by the authors in English.     

垂直管吸收器内泡式吸收过程的数值研究

建立垂直管吸收器管内泡式吸收过程中传热传质的数学物理模型，对其泡式吸收过程进行数值研究，获得泡式吸收方式的一些传热传质特性，为吸收器的优化设计提供一定理论指导。     

ISIS全局优化多道反演技术在墩2块岩性油藏描述中的应用

为进一步落实墩2块戴一段岩性油藏成藏规模及分布规律，针对该储层薄、横向变化快的特点，利用ISIS全局优化多道反演技术，对该构造区岩性储层进行了精细描述，较好的预测出戴一段储层平面分布规律。     

冠心病患者循环血中内皮细胞、内皮素和6－酮－前列环素F_（1a）的动态变化及意义

以循环内皮细胞（ＣＥＣ）作为血管内皮细胞（ＶＥＣ）损伤的指示物，以血浆内皮素（ＥＴ）和前列环素（ＰＧＩ２）反映ＶＥＣ的功能变化，对８５例冠心病患者与３０例正常人进行对比研究，并对５０例不稳定型心绞痛及２０例急性心肌梗塞进行了动态观察．结果表明：冠心病外周血中ＣＥＣ、ＥＴ明显升高（Ｐ＜０．００１），ＰＧＩ２明显下降（Ｐ＜０．００１），尤以不稳定型心绞痛和急性心肌梗塞更显著，与病情的严重程度相一致．提示冠心病ＶＥＣ损伤，以及它所分泌的内皮源性舒缩因子失平衡参与了冠心病的病理过程。     

聚四氢呋喃及其下游产品的开发应用

本文论述了聚四氢呋喃技术进展、氨纶弹性纤维发展现状和国内外聚四氢呋喃产能和需求。     

基于Winsock 的多线程CAMAC通信服务器软件的设计与实现

叙述了应用 Winsock技术与多线程技术实现高性能的 CAMAC通信服务器程序。介绍了整个程序的设计实现方法 ,重点介绍了网络通信服务的实现和多线程访问设备时的同步问题     

多棒高功率固体激光器动态模匹配研究

采用光学矩阵分析了多元热透镜谐振腔内模式的动态匹配特征,给出了平平腔参数调整的关系式,实验结果与理论分析相符,并在两棒连续Nd:YAG系统上得到0W～530W功率范围的稳定输出.     

Effects of chronic U50,488H treatment on binding and mechanical responses of the rat hearts

The effects of chronic injection of U50,488H (trans-3,4-dichloro-N-[2-(1-pyrrolidinyl)cyclohexyl]benzeacetamidel++ +), a selective kappa opioid agonist, on the properties of the binding sites of tritiated U69593 [(5 alpha,7 alpha,8 beta)-(-)-N-methyl-N-(7-(1-pyrrolidinyl)-1-oxaspiro (4,5)dec-8-yl)benzeneacetamide], another selective kappa opioid agonist, and mechanical responses to U50,488H of the heart were studied. Rats received injection twice a day with U50,488H for 4 days. Binding studies on the crude membrane homogenates revealed that there was no change in maximum binding, but a significant increase in Kd after the treatment, indicating that the number of kappa binding sites remained unchanged whereas the affinity of the binding sites to kappa-agonist decreased. The study on the mechanical responses to U50,488H in the isolated perfused heart preparation showed that although the agonist at 10(-6) M caused MR2266 reversible reductions in heart rate and force of contraction as well as ventricular ectopic beat in the heart of rats in the control group, its effects were absent in the U50,488H-treated group, indicating the development of tolerance to the mechanical effects of U50,488H on the heart. The results indicate that the development of tolerance to the mechanical effects of a kappa-agonist after chronic treatment with the agonist was not accompanied by down-regulation, but only a slight and significant reduction in affinity of kappa binding sites in the rat heart.