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101.
研究了运动目标检测与跟踪的DSP(DigitalSignalProcessor)实现算法,以形心跟踪算法为整个处理系统的核心。采用目标形心跟踪算法,通过目标分割阶段的目标标记,如目标面积、周长、形心位置等信息的提取建立目标跟踪波门,实现目标的连续跟踪,并将此算法移植到sEED—VPM642硬件平台,实验结果表明能够达到预定目标。此外,为了克服形心算法的准确性和实时性缺陷,采用粒子滤波对算法进行必要的扩展,从MATLAB的仿真结果看,除个别采样点存在误差较大的情况,真实值曲线与粒子滤波跟踪曲线拟合较好。 相似文献
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使用无源时差(TDOA)定位技术确定无人机等小型辐射源目标的位置是当前研究的热点,针对时差定位算法较为复杂的实际情况,推导了时差双曲线的几何解,并提出了一种基于自适应无迹粒子滤波(AUPF)技术的移动目标定位跟踪方法。通过仿真对该方法在不同场景的应用效果进行了验证,进一步比较分析了算法的定位精度。结果表明,基于自适应无迹粒子滤波的时差几何定位跟踪算法可以在多种情况下较好地拟合出目标真实运动轨迹,实现对运动目标的定位跟踪,同时拥有更低的定位误差和更高的轨迹包容度,使用该方法可以显著提高对非合作移动辐射源目标的位置估计性能。 相似文献
107.
Sinuo Liu Xiaokun Wang Xiaojuan Ban Yanrui Xu Jing Zhou Jií Kosinka Alexandru C. Telea 《Computer Graphics Forum》2021,40(1):54-67
A major issue in smoothed particle hydrodynamics (SPH) approaches is the numerical dissipation during the projection process, especially under coarse discretizations. High‐frequency details, such as turbulence and vortices, are smoothed out, leading to unrealistic results. To address this issue, we introduce a vorticity refinement (VR) solver for SPH fluids with negligible computational overhead. In this method, the numerical dissipation of the vorticity field is recovered by the difference between the theoretical and the actual vorticity, so as to enhance turbulence details. Instead of solving the Biot‐Savart integrals, a stream function, which is easier and more efficient to solve, is used to relate the vorticity field to the velocity field. We obtain turbulence effects of different intensity levels by changing an adjustable parameter. Since the vorticity field is enhanced according to the curl field, our method can not only amplify existing vortices, but also capture additional turbulence. Our VR solver is straightforward to implement and can be easily integrated into existing SPH methods. 相似文献
108.
为有效求解逆向物流车辆路径(VRPSPD)模型,本文提出一种基于种群多样性的自适应PSO算法(SDAPSO)。在SDAPSO运行时,根据种群多样性,自适应地对种群中运行较差的粒子进行扰动操作,提升这些粒子向最优解收敛的能力;同时,对全局最优粒子进行概率扰动,以增加种群的多样性。标准检测函数的仿真结果表明SDAPSO算法是对基本PSO算法的有效改进。在对VRPSPD模型求解中,通过与其它粒子群算法相比,表明SDAPSO是求解该类问题的一种有效方法。 相似文献
109.
N. Z. Mehdizadeh M. Lamontagne C. Moreau S. Chandra J. Mostaghimi 《Journal of Thermal Spray Technology》2005,14(3):354-361
Plasma-sprayed molten molybdenum particles (∼40 μm in diameter) were photographed impinging at high velocity (∼140 m/s) on
a glass substrate at room temperature. An optical sensor detected thermal radiation emitted by a droplet as it approached
the substrate and activated a time delay unit. After a selected time interval, an Nd:YAG laser was triggered, emitting a 5
ns pulse that provided illumination for a charge-coupled device (CCD) camera to photograph the impacting droplet through a
long-range microscope. By varying the delay before pulsing the laser, different stages of droplet deformation were recorded.
Impacting droplets spread into a thin circular film that ruptured and broke into small fragments. An optical detector recording
thermal radiation from the impacting droplet gave a signal that increased as the droplet spread out, reached a maximum when
the liquid film began to rupture, and decreased as portions of the droplet recoiled because of surface tension and then flew
out of view of the photodetector. 相似文献
110.
The voltage-gated proton channel, Hv1, also termed VSOP, was discovered in 2006. It has long been suggested that proton transport through voltage-gated proton channels regulate reactive oxygen species (ROS) production in phagocytes by counteracting the charge imbalance caused by the activation of NADPH oxidase. Discovery of Hv1/VSOP not only confirmed this process in phagocytes, but also led to the elucidation of novel functions in phagocytes. The compensation of charge by Hv1/VSOP sustains ROS production and is also crucial for promoting Ca2+ influx at the plasma membrane. In addition, proton extrusion into neutrophil phagosomes by Hv1/VSOP is necessary to maintain neutral phagosomal pH for the effective killing of bacteria. Contrary to the function of Hv1/VSOP as a positive regulator for ROS generation, it has been revealed that Hv1/VSOP also acts to inhibit ROS production in neutrophils. Hv1/VSOP inhibits hypochlorous acid production by regulating degranulation, leading to reduced inflammation upon fungal infection, and suppresses the activation of extracellular signal-regulated kinase (ERK) signaling by inhibiting ROS production. Thus, Hv1/VSOP is a two-way player regulating ROS production. Here, we review the functions of Hv1/VSOP in neutrophils and discuss future perspectives. 相似文献