基于深度学习模型的无线传感器网络数据融合算法

为了在无线传感器网络中提高数据融合性能,基于深度学习模型,提出一种将层叠自动编码器(SAE)和分簇协议相结合的数据融合算法SAEMDA,该算法在各个簇内构建特征提取分类模型SAEM,通过SAEM对节点数据进行特征提取和分类,之后将同类特征融合并发送给汇聚节点。SAEM的训练既可以采用离线有监督学习也可以采用在线无监督学习。仿真实验表明:和BPFDA,SOFMDA算法相比,SAEMDA在网络能耗大致相当的情况下能将数据融合正确率提高最多7.5%。     

认知增强型无线传感器节点设计

为了提高无线传感器节点及其网络的吞吐量和频谱利用率,从节点设计的角度,引入认知方法增强节点的频谱感知能力,设计了基于低功耗高速率处理器的多射频接口认知增强无线传感器节点.节点采用STR911系列的ARM9微处理器,具有4个射频接口,覆盖了ISM频段和ZigBee频段.实验结果表明,相比基于Atmega128处理器的节点,该节点具有更强的认知能力,吞吐量提高了68.41%,平均信道感知时延缩短了1.4782ms;相比于CSMA/CA方法,通信时延缩短了11.86%,链路层控制方案能够有效避免干扰对节点间通信的影响.     

多路径DSR和AODV路由协议性能研究

在支持移动节点和自组织网络的路由协议中,最典型的单径路由协议是DSR和AODV协议,对它们进行扩展分别得到相应的多径路由协议。通过仿真比较分析这2种多径路由协议在混合无线Mesh网络和移动AdHoc网络中的性能。结果显示,混合无线Mesh网络中的这2种多径路由协议具有分组传输率高、端到端的平均延时和路由开销小等优点。以DSR协议为基础扩展得到的多径路由协议,在高度动态的网络环境中,性能得到了改善,能广泛应用于移动性强的环境中。     

Structural Abnormalities of the Optic Nerve and Retina in Huntington’s Disease Pre-Clinical and Clinical Settings

Huntington’s disease (HD) is a fatal neurodegenerative disorder caused by a polyglutamine expansion in the huntingtin protein. HD-related pathological remodelling has been reported in HD mouse models and HD carriers. In this study, we studied structural abnormalities in the optic nerve by employing Spectral Domain Optical Coherence Tomography (SD-OCT) in pre-symptomatic HD carriers of Caucasian origin. Transmission Electron Microscopy (TEM) was used to investigate ultrastructural changes in the optic nerve of the well-established R6/2 mouse model at the symptomatic stage of the disease. We found that pre-symptomatic HD carriers displayed a significant reduction in the retinal nerve fibre layer (RNFL) thickness, including specific quadrants: superior, inferior and temporal, but not nasal. There were no other significant irregularities in the GCC layer, at the macula level and in the optic disc morphology. The ultrastructural analysis of the optic nerve in R6/2 mice revealed a significant thinning of the myelin sheaths, with a lamellar separation of the myelin, and a presence of myelonoid bodies. We also found a significant reduction in the thickness of myelin sheaths in peripheral nerves within the choroids area. Those ultrastructural abnormalities were also observed in HD photoreceptor cells that contained severely damaged membrane disks, with evident vacuolisation and swelling. Moreover, the outer segment of retinal layers showed a progressive disintegration. Our study explored structural changes of the optic nerve in pre- and clinical settings and opens new avenues for the potential development of biomarkers that would be of great interest in HD gene therapies.     

无线认知网络概念与实例研究

提出了一种新的、以认知能力为基础的无线网络,称为无线认知网络。对无线认知网络进行了定义,对认知实体、认知进程和认知环境进行了分析和讨论,提出了无线认知网络的实体、网络、认知和控制4个关联层面,对无线认知网络的特点进行了阐述,并给出了技术上的挑战。在此基础上,从基础设施、层次结构和节点性质3个方面对无线认知网络进行了分类研究,在给出最小跨层设计原则与信息交换方法的基础上,提出了由基础层、网络适配层、应用开发环境层和应用逻辑层组成的4层软件体系结构。最后结合无线认知网络目前的惟一实例-认知无线电网络,对无线认知网络的实例化问题进行了讨论。     

用Visual Basic语言实现模拟绘图功能

本文介绍在Visual Basic语言编程环境中,利用鼠标mousedown、mousemove和mouseup事件,建立一个可供写字或绘图的窗口,将其编译成在桌面上可执行的文件,代替教学用粉笔写字或绘图.     

A Novel Mouse Model of TGFβ2-Induced Ocular Hypertension Using Lentiviral Gene Delivery

Glaucoma is a multifactorial disease leading to irreversible blindness. Primary open-angle glaucoma (POAG) is the most common form and is associated with the elevation of intraocular pressure (IOP). Reduced aqueous humor (AH) outflow due to trabecular meshwork (TM) dysfunction is responsible for IOP elevation in POAG. Extracellular matrix (ECM) accumulation, actin cytoskeletal reorganization, and stiffening of the TM are associated with increased outflow resistance. Transforming growth factor (TGF) β2, a profibrotic cytokine, is known to play an important role in the development of ocular hypertension (OHT) in POAG. An appropriate mouse model is critical in understanding the underlying molecular mechanism of TGFβ2-induced OHT. To achieve this, TM can be targeted with recombinant viral vectors to express a gene of interest. Lentiviruses (LV) are known for their tropism towards TM with stable transgene expression and low immunogenicity. We, therefore, developed a novel mouse model of IOP elevation using LV gene transfer of active human TGFβ2 in the TM. We developed an LV vector-encoding active hTGFβ2^C226,228S under the control of a cytomegalovirus (CMV) promoter. Adult C57BL/6J mice were injected intravitreally with LV expressing null or hTGFβ2^C226,228S. We observed a significant increase in IOP 3 weeks post-injection compared to control eyes with an average delta change of 3.3 mmHg. IOP stayed elevated up to 7 weeks post-injection, which correlated with a significant drop in the AH outflow facility (40.36%). Increased expression of active TGFβ2 was observed in both AH and anterior segment samples of injected mice. The morphological assessment of the mouse TM region via hematoxylin and eosin (H&E) staining and direct ophthalmoscopy examination revealed no visible signs of inflammation or other ocular abnormalities in the injected eyes. Furthermore, transduction of primary human TM cells with LV_hTGFβ2^C226,228S exhibited alterations in actin cytoskeleton structures, including the formation of F-actin stress fibers and crossed-linked actin networks (CLANs), which are signature arrangements of actin cytoskeleton observed in the stiffer fibrotic-like TM. Our study demonstrated a mouse model of sustained IOP elevation via lentiviral gene delivery of active hTGFβ2^C226,228S that induces TM dysfunction and outflow resistance.     

无线局域网吞吐量优化算法

针对无线局域网吞吐量较小且稳定性较差的问题,分析影响无线网络吞吐量性能的因素,提出无线局域网吞吐量优化算法,采用马尔科夫链理论建立吞吐量计算模型,运用动态调整竞争窗口、优化数据帧长度、改进退避时间的策略。NS2仿真结果表明,该算法能有效提高无线局域网的吞吐量。     

无线传感器网络自适应紧急上报与兴趣命令协议

针对无线传感器网络的节能要求,提出了自适应断链恢复、事件源数据融合、信道预约等新技术,进而提出了一种新的MAC层网络协议。该协议由紧急上报和兴趣命令驱动数据传输,适用于大规模数据采集与环境监测。与其他同类型协议比较的仿真结果表明,该协议具有更好的节能效果和更短的延时。     

矿井无线通信技术及其发展趋势

分析了目前矿井使用的无线安全调度和应急通信系统,讨论了其系统结构和特点,并根据矿井安全生产的现状,指出了矿井无线安全调度和应急通信系统的发展趋势。