基于CPSO-BP神经网络的风电并网暂态电压稳定评估

针对目前传统方法难以快速、准确判断风电并网后系统暂态电压稳定性的问题,提出了一种基于CPSO-BP组合的风电并网暂态电压稳定评估方法。首先采用混沌理论对粒子群算法的不足进行改善,应用改进后的算法对神经网络的初始权值和阈值进行优化,然后利用系统故障前后采集的传统物理量和风电场相关的物理量作为BP神经网络输入特征量进行监督学习,最后将训练得到的模型应用于风电并网系统的暂态电压稳定评估中。利用英格兰10机39节点系统标准算例进行风电并网仿真分析,结果证明了所提方法的有效性。     

Conventional and Unconventional Mechanisms by which Exocytosis Proteins Oversee β-cell Function and Protection

Type 2 diabetes (T2D) is one of the prominent causes of morbidity and mortality in the United States and beyond, reaching global pandemic proportions. One hallmark of T2D is dysfunctional glucose-stimulated insulin secretion from the pancreatic β-cell. Insulin is secreted via the recruitment of insulin secretory granules to the plasma membrane, where the soluble N-ethylmaleimide-sensitive factor attachment protein receptors (SNAREs) and SNARE regulators work together to dock the secretory granules and release insulin into the circulation. SNARE proteins and their regulators include the Syntaxins, SNAPs, Sec1/Munc18, VAMPs, and double C2-domain proteins. Recent studies using genomics, proteomics, and biochemical approaches have linked deficiencies of exocytosis proteins with the onset and progression of T2D. Promising results are also emerging wherein restoration or enhancement of certain exocytosis proteins to β-cells improves whole-body glucose homeostasis, enhances β-cell function, and surprisingly, protection of β-cell mass. Intriguingly, overexpression and knockout studies have revealed novel functions of certain exocytosis proteins, like Syntaxin 4, suggesting that exocytosis proteins can impact a variety of pathways, including inflammatory signaling and aging. In this review, we present the conventional and unconventional functions of β-cell exocytosis proteins in normal physiology and T2D and describe how these insights might improve clinical care for T2D.     

Utilizing an Animal Model to Identify Brain Neurodegeneration-Related Biomarkers in Aging

Glycine N-methyltransferase (GNMT) regulates S-adenosylmethionine (SAMe), a methyl donor in methylation. Over-expressed SAMe may cause neurogenic capacity reduction and memory impairment. GNMT knockout mice (GNMT-KO) was applied as an experimental model to evaluate its effect on neurons. In this study, proteins from brain tissues were studied using proteomic approaches, Haemotoxylin and Eosin staining, immunohistochemistry, Western blotting, and ingenuity pathway analysis. The expression of Receptor-interacting protein 1(RIPK1) and Caspase 3 were up-regulated and activity-dependent neuroprotective protein (ADNP) was down-regulated in GNMT-KO mice regardless of the age. Besides, proteins related to neuropathology, such as excitatory amino acid transporter 2, calcium/calmodulin-dependent protein kinase type II subunit alpha, and Cu-Zn superoxide dismutase were found only in the group of aged wild-type mice; 4-aminobutyrate amino transferase, limbic system-associated membrane protein, sodium- and chloride-dependent GABA transporter 3 and ProSAAS were found only in the group of young GNMT-KO mice and are related to function of neurons; serum albumin and Rho GDP dissociation inhibitor 1 were found only in the group of aged GNMT-KO mice and are connected to neurodegenerative disorders. With proteomic analyses, a pathway involving Gonadotropin-releasing hormone (GnRH) signal was found to be associated with aging. The GnRH pathway could provide additional information on the mechanism of aging and non-aging related neurodegeneration, and these protein markers may be served in developing future therapeutic treatments to ameliorate aging and prevent diseases.     

Bionic Design and Simulation Analysis of Energy-Efficient and Vibration-Damping Walking Mechanism

节能减振步行机构的仿生设计及仿真分析

张锐¹,庞浩¹,何远^1,2,韩佃雷¹,文立阁³,江磊⁴,李建桥¹

（1. 吉林大学 工程仿生教育部重点实验室,长春 130022;

2. 长春长光睿视光电技术有限责任公司,长春 130102;

3. 吉林大学 机械与航天工程学院,长春 130022;

4. 中国北方车辆研究所,北京 100072）

摘要：非洲鸵鸟能以60 km/h的速度持续奔跑30分钟,其后肢结构具有优良的节能、减振性能。为实现腿式机器人高效节能、缓冲减振的腿部机构的设计,根据鸵鸟跗骨间关节的被动回弹特性以及腿部的输出刚度可变特性,结合鸵鸟后肢结构的比例和尺寸,设计出了仿生刚-柔复合腿式机器人单腿结构。并且利用ADAMS软件模拟了仿生机械腿的运动。通过运动仿真分析,研究了内弹簧刚度系数在一定范围内的变化对机身质心垂直加速度以及电机功耗的影响,获得最佳的内弹簧刚度系数为200 N/mm,并进一步验证了内外弹簧机制能够有效地降低机械腿的能耗。仿真结果表明,采用内外弹簧机制能够有效降低电机约72.49%能耗。

关键词：工程仿生;仿生机械腿;被动回弹特性;刚-柔复合结构;节能减振

     

淹没深度对三自由度波能浮子获能的影响

为明确浮子淹没深度对三自由度浮子装置获能的影响,提出漂浮式与淹没式两种浮子模型,计算并比较在不同淹没深度和波浪条件下浮子的获能特性。浮子模型具有纵荡、垂荡与纵摇3个运动自由度。浮子选用直立圆柱体浮子。淹没深度分别设置为其高度的0.5、1.0、1.5与2.0倍,质量相同。漂浮式浮子模型作为对照,基于线性势流理论与边界元方法,建立浮子水动力学模型,计算浮子水动力学参数及运动响应,并采用线性能量转换(power take-off, PTO)系统阻尼,计算并对比浮子在最优PTO阻尼下的平均输出功率,分析淹没深度对浮子获能的影响。研究结果表明,增大淹没深度可以减小浮子的波浪激励力和平均输出功率。对于三自由度浮子,漂浮浮子在低频波浪下获能特性较好,而具有较小淹没深度的浮子在高频波浪下性能更优。     

深海带电插拔连接器力学特性分析

以水下7 000 m工况的深海带电插拔连接器为研究对象,旨在获得湿插拔连接器在深海双重高压下的受力特性和插拔过程中的动密封性能。介绍连接器的插头结构与压力补偿技术;推导压力补偿器工作容积的计算公式;运用Ansys Workbench建立插头内壳体组件与插孔组件的动密封有限元模型,对内壳体、压力平衡膜及内胆进行强度计算;分析由连接器插拔引起的平衡膜内外瞬时压差对膜的应力分布及变形情况;讨论密封件径向压缩量、密封接触面摩擦因数、插拔速度对插拔过程中动密封性能的影响。结果表明:插头插合时,平衡膜外表面应力基本不变,其变形减小;拔出时,平衡膜外表面的应力及变形都逐渐增大。密封件之间的接触压力受密封接触面过盈配合量的影响明显,增大过盈配合量可显著提高动密封区域的接触压力及密封性能,同时也会加剧密封件失效的可能性,密封接触面的摩擦因数、插拔速度对接触压力的影响则较小。     

振荡翼改进运动模型的能量捕获性能分析

为了提高振荡水翼的水动力性能和能量捕获效率,基于传统简谐运动模型提出改进运动模型,通过引入俯仰运动系数k,推导改进后俯仰运动规律的一般形式,利用Fluent软件建立水翼的二维模型,综合分析半主动振荡模型下不同俯仰运动系数、折算频率和俯仰振幅等运动参数对水翼能量捕获性能的影响。结果表明:与传统简谐运动模型相比,改进运动模型能够使水翼升力系数在较长时间段内维持较大数值,提高水翼的升力系数和捕能效率;在相同的折算频率和俯仰振幅下,减小俯仰运动系数会增大俯仰角转动的角速度,进而增大俯仰运动所消耗的能量;不同俯仰振幅下对应不同的最优折算频率,折算频率越低水翼的捕能性能越不稳定,俯仰振幅越大,转动俯仰角所消耗的能量越多。在给定参数条件下,水翼的能量转化效率可超过40%。     

高强度结构钢的韧性启裂和稳态扩展

研究了具有一定韧度的高强度结构钢裂纹早期扩展阻力与材料的微观组织结构、应力状态、变形历史和应变特性之间的关系。结果表明:裂纹扩展阻力(指J_R阻力曲线中的J_i和dJ/da)主要决定于裂纹尖端的塑性约束程度和应力应变场中消耗的弹性能和塑性功。这与三轴应力状态函数σ_mσ~(1/2)有关。材料的流变曲线描述了形变过程中的变形历史和应变特性,有明显的阶段性,其中第Ⅱ阶段的变形历史和应变特性制约着裂纹的启裂过程,第Ⅲ阶段的变形历史和应变特性制约着裂纹的扩展过程。应变硬化率dσ/dε可以敏感地反映出材料内部的应力分布和微观断裂机制。     

EFFECT OF AGING ON HYDROGEN EMBRITTLEMENT SUSCEPTIBILITY OF STEEL Cr21Ni6Mn9N

Effects of 650℃ aging for 1—1000 h on structure and hydrogen embrittlement susceptibility(HES)of steel Cr21Ni6Mn9N have been investigated.The results show that M_(23)C_6 typecarbide precipitates at grain boundaries and Cr-depletive region appears beside them duringaging.The precipitates grow and connect each other as the aging time prolongs.Meanwhile,the degree of Cr-depletion aggravates first and then recovers gradually while the aging time isvery long,i.e.,1000 h.The HES of the steel increases with increasing aging time but does notreduce with the recovery of Cr content at the Cr-depletive region.That implies that the ex-isting of carbides at grain boundaries might be the main reason which promotes the HES ofsteel during aging.