低温烧结SrTiO_3晶界势垒高度的研究

本文利用双Ｓｃｈｏｔｔｋｙ势垒模型依据ＳｒＴｉＯ３晶界层电容器材料的Ｒ～Ｔ特性和室温下的Ｉ～Ｖ特性获得了晶界势垒高度随温度变化的关系．结果表明，势垒高度随温度的升高而增加．这一结果与用各不同温度下的Ｉ～Ｖ特性拟合所得到的势垒值基本相符．     

大豆蛋白添加量与成膜环境对大豆分离蛋白膜的影响

以大豆分离蛋白(SPI)为原料,通过调整SPI和甘油(GLY)的比例来制备不同配方的可食性SPI包装膜。通过观察薄膜色泽、柔软性等表观情况,测定其机械性能(抗拉强度和断裂伸长率)和阻隔性能(阻湿性和阻氧性),并比较了其性能。结果表明:影响SPI膜性能的主要因素为SPI添加量、SPI与增塑剂的质量比、干燥条件;在pH=9条件下,SPI质量为5.66 g,SPI与GLY质量比为6∶1.5,以自然晾干和烘箱干燥结合法进行干燥时,膜的性能最优。     

Shadow method for measuring the average electron density in an ionized gas flow induced by a high-frequency barrier discharge

The electron density distribution in an ionized gas flow induced by a high-frequency barrier discharge has been investigated by an optical method. It has been shown that ion acceleration occurs mainly near the minima of the electron density, and the extremes in the temperature distribution are in antiphase with the corresponding extremes of the electron density. Translated from Inzhenerno-Fizicheskii Zhurnal, Vol. 82, No. 2, pp. 364–370, March–April, 2009.     

"一带一路"战略下西亚北非技术性贸易措施分析及对策研究

文章对1995年到2016年间的西亚北非主要贸易国家TBT/SPS通报进行统计分析,研究其TBT/SPS通报的发展趋势及特点,最后重点进行对策研究,以最大限度保护我国出口企业合法权益,合理规避技术性贸易壁垒,促进中国对西亚北非贸易地区的出口.     

Grafting Silane onto Silicate Glass Surface Treated by DBD in Air

Dielectric barrier discharge plasma in air was used to modify glass surface to induce the graft of silane onto the treated surface to increase the possibility of biomolecule immobilization. The plasma treated glass had been characterized by scanning electron microscopy （SEM）, Fourier transform infrared attenuated total reflection spectroscopy, X-ray photoelectron spectroscopy （XPS） and surface water contact angle measurement. The validity of grafting silane onto glass surface was approved by the analysis of water contact angle measurement, SEM and XPS. The grafted silane content was measured by visible absorption spectroscopy using acid Orange-7. It is shown that the grafting density of silane for glass samples increases significantly after plasma treatment.     

Plant Responses to Herbivory,Wounding, and Infection

Plants have various self-defense mechanisms against biotic attacks, involving both physical and chemical barriers. Physical barriers include spines, trichomes, and cuticle layers, whereas chemical barriers include secondary metabolites (SMs) and volatile organic compounds (VOCs). Complex interactions between plants and herbivores occur. Plant responses to insect herbivory begin with the perception of physical stimuli, chemical compounds (orally secreted by insects and herbivore-induced VOCs) during feeding. Plant cell membranes then generate ion fluxes that create differences in plasma membrane potential (Vm), which provokes the initiation of signal transduction, the activation of various hormones (e.g., jasmonic acid, salicylic acid, and ethylene), and the release of VOCs and SMs. This review of recent studies of plant–herbivore–infection interactions focuses on early and late plant responses, including physical barriers, signal transduction, SM production as well as epigenetic regulation, and phytohormone responses.     

I2-Imidazoline Ligand CR4056 Improves Memory,Increases ApoE Expression and Reduces BBB Leakage in 5xFAD Mice

Recent evidence suggests that I2-imidazoline ligands have neuroprotective properties in animal models of neurodegeneration, such as Alzheimer’s disease (AD). We recently demonstrated that the I2-ligand BU224 reversed memory impairments in AD transgenic mice and this effect was not because of reductions in amyloid-β (Aβ) deposition. In this study, our aim was to determine the therapeutic potential of the powerful analgesic I2-imidazoline ligand CR4056 in the 5xFAD model of AD, since this ligand has been proven to be safely tolerated in humans. Sub-chronic oral administration of CR4056 (30 mg/kg for 10 days) led to an improvement in recognition memory in 6-month-old 5xFAD mice, but not in wild-type littermates, without affecting Aβ levels or deposition. Our results also revealed a change in the profile of microglia by CR4056, resulting in a suppression of pro-inflammatory activated microglia, but increased the density of astrocytes and the expression of ApoE, which is mainly produced by these glial cells. In addition, CR4056 restored fibrinogen extravasation, affecting the distribution of markers of astrocytic end feet in blood vessels. Therefore, these results suggest that CR4056 protects against Aβ-mediated neuroinflammation and vascular damage, and offers therapeutic potential at any stage of AD.     

Melatonin and the Brain–Heart Crosstalk in Neurocritically Ill Patients—From Molecular Action to Clinical Practice

Brain injury, especially traumatic brain injury (TBI), may induce severe dysfunction of extracerebral organs. Cardiac dysfunction associated with TBI is common and well known as the brain–heart crosstalk, which broadly refers to different cardiac disorders such as cardiac arrhythmias, ischemia, hemodynamic insufficiency, and sudden cardiac death, which corresponds to acute disorders of brain function. TBI-related cardiac dysfunction can both worsen the brain damage and increase the risk of death. TBI-related cardiac disorders have been mainly treated symptomatically. However, the analysis of pathomechanisms of TBI-related cardiac dysfunction has highlighted an important role of melatonin in the prevention and treatment of such disorders. Melatonin is a neurohormone released by the pineal gland. It plays a crucial role in the coordination of the circadian rhythm. Additionally, melatonin possesses strong anti-inflammatory, antioxidative, and antiapoptotic properties and can modulate sympathetic and parasympathetic activities. Melatonin has a protective effect not only on the brain, by attenuating its injury, but on extracranial organs, including the heart. The aim of this study was to analyze the molecular activity of melatonin in terms of TBI-related cardiac disorders. Our article describes the benefits resulting from using melatonin as an adjuvant in protection and treatment of brain injury-induced cardiac dysfunction.