IPv6在矿井工业以太网中的应用研究

介绍了IPv6产生的背景和发展现状,阐述了IPv6应用于矿井工业以太网的发展趋势和研究方向,指出对于所有基于矿井工业以太网的硬件和软件的开发都应带有双协议栈,随着IPv6技术的成熟和普及再研发出只有IPv6协议栈的适用于煤矿的网络设备,如支持IPv6的防爆网络设备、组态软件以及相应的数据采集驱动设备、控制硬件等。     

基于IPv6的无线高原冻土监测系统

设计了一种新型的高原冻土监测系统。采用ARM7内核的MCU进行控制,并基于IPv6协议利用IPv6技术和GPRS技术相结合的方式,建立了可自组网的无线传感器网络,实现了对指定区域环境参数的实时监测。系统使用了多个土壤水分传感器、温度传感器、二氧化碳传感器、氮气传感器对高原冻土环境进行全方位实时观测。各监测点将数据通过基于IPv6的无线网络传送给汇聚节点,汇聚节点再通过GPRS网络将数据传送给监控中心。监控中心可以对数据进行记录和分析,更好地指导环境保护和建设工作。     

Measurement of the heat of fusion of titanium and a titanium alloy (90Ti-6Al-4V) by a microsecond-resolution transient technique

A microsecond-resolution pulse heating technique was used for the measurement of the heat of fusion of titanium and a titanium alloy (90Ti-6Al-4V). The method is based on rapid (50- to 100-s) resistive self-heating of the specimen by a high-current pulse from a capacitor discharge system and measuring, as functions of time, current through the specimen, voltage across the specimen, and radiance of the specimen. Melting of the specimen is manifested by a plateau in the measured radiance. The time integral of the net power absorbed by the specimen during melting yields the heat of fusion. The values obtained for heat of fusion were 272 J · g^–1 (13.0 kJ · mol^–1) for titanium and 286 J · g^–1 for the alloy 90Ti-6Al-4V, with an estimated maximum uncertainty of ±6% in each value.Paper presented at the Second Workshop on Subsecond Thermophysics, September 20–21, 1990, Torino, Italy.     

IPv6网络安全的实现与测试

IPv4地址日趋枯竭,IPv6协议替代IPv4协议势在必行。与IPv4协议相比,IPv6协议在地址空间、路由聚合、服务质量和网络安全等方面带来了很大的发展和提升空间,但IPv6的网络安全仍然存在。本文对IPv6的防火墙和入侵检测系统进行了测试,并对其结果进行了分析。为正确部署防火墙和定位入侵检测系统提出了系列建议。     

Surface characterization of 6H-SiC (0001) substrates in indentation and abrasive machining

Surface characterization of 6H-SiC (0001) substrates in indentation and abrasive machining was carried out to investigate microfracture, residual damage, and surface roughness associated with material removal and surface generation. Brittle versus plastic deformation was studied using Vickers indention and nano-indentation. To characterize the abrasive machining response, the 6H-SiC (0001) substrates were ground using diamond wheels with grit sizes of 25, 15 and 7 μm, and then polished with diamond suspensions of 3 and 0.05 μm. It is found that in indentation, there was a scale effect for brittle versus plastic deformation in 6H-SiC substrates. Also, in grinding, the scales of fracture and surface roughness of the substrates decreased with a decrease in diamond grit size. However, in polishing, a reduction in grit size of diamond suspensions gave no significant improvement in surface roughness. Furthermore, the results showed that fracture-free 6H-SiC (0001) surfaces were generated in polishing with the existence of the residual crystal defects, which were associated with the origin of defects in single crystal growth.     

基于STM32的多功能液体检测系统

本系统以STM32F103RCT6单片机为核心控制芯片,可以一键自动测量容器内液体的重量和液位参数,并显示测量数据、判别液体种类。使用激光与浮板进行液位测量;使用压力传感器与24位AD转化模块HX711进行高精度的重量测量;利用pH值传感器、光电传感器、导电率传感器获得液体不同的物理特性,区分液体种类。系统模块构成主要包括单片机系统模块、电源模块、激光测距模块、称重模块、传感器模块、OLED显示模块和按键模块,具有多功能、测量准确、反应速度快的特点。     

Ti-22Nb-6Zr(at%)合金的超弹性和形状记忆效应

用弯曲法和拉伸法测定Ti-22Nb-6Zr（at％）合金的超弹性和形状记忆效应，研究固溶处理温度对Ti-22Nb-6Zr合金组织结构及性能的影响。结果表明：固溶处理后Ti-22Nb-6Zr合金的室温组织为单一的β相，晶粒尺寸随固溶处理温度升高而增大；合金的静态弹性模量小于30GPa；Ti-22Nb-6Zr合金具有良好的超弹性和一定的形状记忆效应。室温下变形，合金的超弹性和形状记忆效应随固溶处理温度升高而提高。900℃固溶处理后的合金在室温下拉伸变形，应变为5％时，总的最大回复应变达4．12％，其中超弹性回复应变为3．91％，记忆回复应变为0.21％。     

ATP-Independent Initiation during Cap-Independent Translation of m6A-Modified mRNA

The methylation of adenosine in the N⁶ position (m⁶A) is a widely used modification of eukaryotic mRNAs. Its importance for the regulation of mRNA translation was put forward recently, essentially due to the ability of methylated mRNA to be translated in conditions of inhibited cap-dependent translation initiation, e.g., under stress. However, the peculiarities of translation initiation on m⁶A-modified mRNAs are not fully known. In this study, we used toeprinting and translation in a cell-free system to confirm that m⁶A-modified mRNAs can be translated in conditions of suppressed cap-dependent translation. We show for the first time that m⁶A-modified mRNAs display not only decreased elongation, but also a lower efficiency of translation initiation. Additionally, we report relative resistance of m⁶A-mRNA translation initiation in the absence of ATP and inhibited eIF4A activity. Our novel findings indicate that the scanning of m⁶A-modified leader sequences is performed by a noncanonical mechanism.     

Interleukin 6 and Aneurysmal Subarachnoid Hemorrhage. A Narrative Review

Interleukin 6 (IL-6) is a prominent proinflammatory cytokine. Neuroinflammation in general, and IL-6 signaling in particular, appear to play a major role in the pathobiology and pathophysiology of aneurysm formation and aneurysmal subarachnoid hemorrhage (SAH). Most importantly, elevated IL-6 CSF (rather than serum) levels appear to correlate with delayed cerebral ischemia (DCI, “vasospasm”) and secondary (“vasospastic”) infarctions. IL-6 CSF levels may also reflect other forms of injury to the brain following SAH, i.e., early brain damage and septic complications of SAH and aneurysm treatment. This would explain why many researchers have found an association between IL-6 levels and patient outcomes. These findings clearly suggest CSF IL-6 as a candidate biomarker in SAH patients. However, at this point, discrepant findings in variable study settings, as well as timing and other issues, e.g., defining proper clinical endpoints (i.e., secondary clinical deterioration vs. angiographic vasospasm vs. secondary vasospastic infarct) do not allow for its routine use. It is also tempting to speculate about potential therapeutic measures targeting elevated IL-6 CSF levels and neuroinflammation in SAH patients. Corticosteroids and anti-platelet drugs are indeed used in many SAH cases (not necessarily with the intention to interfere with detrimental inflammatory signaling), however, no convincing benefit has been demonstrated yet. The lack of a robust clinical perspective against the background of a relatively large body of data linking IL-6 and neuroinflammation with the pathophysiology of SAH is somewhat disappointing. One underlying reason might be that most relevant studies only report correlative data. The specific molecular pathways behind elevated IL-6 levels in SAH patients and their various interactions still remain to be delineated. We are optimistic that future research in this field will result in a better understanding of the role of neuroinflammation in the pathophysiology of SAH, which in turn, will translate into the identification of suitable biomarkers and even potential therapeutic targets.