主余震序列作用下非线性单自由度体系的增量损伤分析

同时采用真实主余震序列和人工构造主余震序列作为输入,开展了非线性单自由度体系在主余震序列作用下的增量损伤分析。挑选75组真实主余震序列,并基于相同的主震记录,分别采用重复法、随机法和衰减法构造不同的人工主余震序列作为输入。分别采用理想弹塑性模型、考虑屈服后刚度的双线型模型和三线型模型,开展主余震序列作用下非线性单自由度体系的反应分析。以修正的Park-Ang损伤指数作为结构损伤指标,对主余震序列作用下的结构增量损伤进行研究,并对强度折减系数、恢复力模型及主余震序列人工构造方法对增量损伤的影响进行研究。结果表明：余震造成的结构增量损伤不可忽视。基于重复法和随机法合成的人工主余震序列相比真实主余震序列的余震强度偏大,因此会高估余震对结构造成的增量损伤。基于衰减法合成的人工主余震序列的余震强度偏小,会低估余震对结构造成的增量损伤。结构特性对增量损伤的影响很小,体现为不同强度折减系数及不同恢复力模型对结构增量损伤的影响均不明显。     

铜箔缺陷成因与危害

铜箔在生产过程中可能会产生各种缺陷,本文对铜箔常规缺陷成因和危害进行了分析总结,将有利于铜箔在生产过程中提高产品质量,同时指导客户正确使用铜箔;特别是对有轻微缺陷的铜箔如何克服使用。     

影响铝电解槽寿命的因素

电解槽寿命是铝行业要研究的重要课题。影响铝电解槽寿命有设计、内衬材料、筑炉质量、焙烧启动方式、启动后期管理、工艺控制和工艺操作等因素的综合作用。该文着重叙述破损机理、筑炉工艺的特点和质量把关、焙烧方式选择、破损槽的测定方法、正常工艺操作，探索延长槽寿命的途径。     

微透镜列阵的光敏热成形新方法

首次在国内用光敏玻璃热成形方法研制了一种新型的微透镜列阵，采用具有特定化学组分的光敏玻璃材料，在普通的紫外平行光束照射下进行感光和热处理，制得了孔径为４００μｍ的微透镜列阵。     

聚氨酯型光敏树脂结构与性能关系的研究

本文对聚氨酯型光敏树脂结构与性能的关系进行了系统研究，得出了光敏树脂性能与预聚体结构、活性稀释剂结构及用量等关系的规律性结果．     

Life calculations for materials under irregular nonproportional loading

Experimental data on low-cycle fatigue of 304 steel and a VT9 titanium alloy in deformation by complex loading histories, which are the sequence of blocks of cycles of different shapes in the space of total strains, cited in the literature, are analyzed to develop adequate models for life calculations. The four damage accumulation rules and the low-cycle fatigue deformation criterion were used as basic approaches. Life prediction models were compared. It is shown that the application of a modified nonlinear damage accumulation rule can improve life prediction results, with better outcomes obtained for the programs involving nonproportional cycles. __________ Translated from Problemy Prochnosti, No. 5, pp. 141–150, September–October, 2007.     

论横向垄断协议的规制

横向垄断协议作为垄断协议的一种,对竞争损害最为直接和严重,各国均纷纷立法予以禁止。我国现行的《反垄断法》虽然规定了横向垄断协议,但关于横向垄断协议的法律责任、反垄断执法机构设置的规定存在不足之处。我国应该设置一个专门的独立的反垄断执法机构,规定横向垄断协议的经济法责任,实行对横向垄断协议的双罚制。     

Photosensitive poly(benzoxazole) based on precursor from diphenyl isophthalate and bis(o-aminophenol)

A new synthetic method for the preparation of poly(benzoxazole) (PBO) precursor, poly(o-hydroxyamide) (7) from bis(o-aminophenol) (5) and diphenyl isophthalate (6) has been developed. Polymer 7 was prepared by the polycondensation of 5 and 6 in 1-methyl-2-pyrrolidinone (NMP) at 185-205 °C. Model reactions were carried out in detail to elucidate appropriate conditions for the formation of 2-hydroxybenzanilide (3) from o-aminophenol (1) and phenyl benzoate (2). The photosensitive (PBO) precursor based on polymer 7 containing a 22% of benzoxazole unit and 30 wt% 1-{1,1-bis[4-(2-diazo-1-(2H)naphthalenone-5-sulfonyloxy)phenyl]ethyl}-4-{1-[4-(2-diazo-1(2H)naphthalenone-5-sulfonyloxy)phenyl]methylethyl}benzene (S-DNQ) showed a sensitivity of 110 mJ cm⁻² and a contrast of 5.0 when it was exposed to 436 nm light followed by developing with a 2.38 wt% aqueous tetramethylammonium hydroxide solution at room temperature. A fine positive image featuring 8 μm line and space patterns was observed on the film of the photoresist exposed to 200 mJ cm⁻² of UV-light at 436 nm by the contact mode.     

含AgX的SiO2凝胶的变色机理

本研究用溶胶－凝胶法制备了含ＡｇＸ（Ｘ：Ｃｌ＾－、Ｂｒ＾－、Ｉ＾－）的变色凝胶及玻璃。利用Ｘ射线衍射，差热－失重分析，红外吸收光谱和紫外－可见光谱分析，对ＳｉＯ２凝胶的变色机理进行了研究。本研究为用溶胶－凝胶法制备光致变色玻璃提供了依据。     

A Dual Synergetic Nanoreactor for Managing Parkinson's Disease by Regulating Inflammation and Mitigating Oxidative Damage

Glial cell-dominated inflammatory microenvironment and neuronal damage due to oxidative stress are major impediments to the treatment of central nervous degenerative diseases. Herein, a neuroinflammatory regulatory nanoreactor is developed by encapsulating dihydroquercetin (Que) and Pt nanozymes (Ptzymes) in mannitol modified poly(lactic-co-glycolic acid) nanoparticles. The nanoreactor shows enhanced antioxidative activities compared with either Que or Ptzymes alone due to synergetic effects of the incorporated active agents. In cellular and animal models of Parkinson's disease (PD), the nanoreactor traverses the blood–brain barrier and accumulates in neurons and microglia, thereby mitigating oxidative damage of neurons, and promoting the polarization of microglia into the anti-inflammatory M2-phenotype to control PD progression. This work demonstrates the gains of combining anti-inflammation small molecule compound and antioxidant nanozyme in a synergetic nanoreactor to effectively prevent neuronal damage and suppress the inflammatory microenvironment, which holds great potential for managing the advances of PD.