电子商务平台快速身份认证算法研究

提出一种快速身份认证方法。待验证用户提出证书申请后,利用门限的思想将证书颁发的任务分派到每个认证参与者身上,通过计算得到的认证参与者的子证书集合,完成身份认证,避免了所有认证工作都在认证中心进行计算造成的认证效率低的问题。实验证明,这种方法能够快速完成用户的身份认证,同时保证了认证的安全性,取得了满意的结果。     

基于图像变形的卡用户认知性(英文)

尽管许多人可能并没有意识到,各类卡已经被广泛应用到我们的日常生活当中。其中,信用卡,现金卡,驾驶执照,等等,其实与钱包同等重要。为了防止这些卡被非法使用,用户认证是必不可少的。最常用的用户认证方法就是要求用户提供某些个人信息。如果用户提供的信息与卡中记录的信息一致,我们就认为用户是合法的。然而如果个人信息被泄露,卡就有可能被非法使用。本论文的目的就是要提供一种新的基于图像变形的卡用户认证方法。其基本思路是把真正持卡人的脸部图像通过图像变形隐藏到某个覆盖数据下,并把恢复原图像所需要的钥匙信息分散保存到卡,终端,服务器等不同地方。在用户使用卡的时候,认证系统可以利用反变形技术恢复原图像,让受权人察知到用户的真伪,从而有效地保护持卡人的权益。     

WiFi网络实名认证的方法研究和实现

随着移动互联网络技术的发展和智能手机的广泛应用,人们对基于WiFi的移动互联业务的需求越来越广泛,许多行业及公共场所都在建设无线WiFi网络,而网络安全管理要求无线WiFi网络必须采用实名认证,其基本手段有用户名认证和手机号认证。本文论述了不依赖于特定协议和与WiFi设备厂商无关的认证方式及实现实名认证的方法和过程。     

一种基于JPEG 2000的数字图像内容认证算法*

针对JPEG 2000图像内容认证的需求,提出一种适用于JPEG 2000图像认证的半脆弱水印算法.首先,将原始图像分块按Hilbert扫描顺序排列;然后在原图像分块Hilbert序列中选取分块,再结合各分块临界噪声阈值矩阵JND阈值,完成小波域的水印自适应嵌入;最后,引入私钥对水印进行加密,提高系统的安全性.在认证端,从接收到的图像提取其中所隐藏信息,比较接收到的图像和隐藏信息,判断内容发生变化的位置.实验表明,该算法对窜改等破坏图像内容的恶意操作有精确的检测和定位能力.     

基于Ajax的轻量级身份认证*

采用HTTPS和传统远程脚本来实现身份认证存在一些缺点.在分析了Ajax技术特点后,提出了一种轻量级基于Ajax的适合中小企业Web应用开发的身份认证方法.同时设计了实现该方法的模型,并分析了该方法的安全性和特点.     

数据库加密系统中基于X.509证书的椭圆曲线加密身份认证机制*

身份认证问题是网络安全的重要研究课题,利用椭圆曲线密码系统相对于其他公钥密码体制具有密钥长度短、运算速度快、计算数据量小的特点,结合X.509证书构建一个新的、高效的、安全的身份认证方案.     

基于家族基因的网格信任模型

针对现有基于PKI (Public Key Infrastructure)的网格信任系统的不足,证书主体信息不明确, 认证过程复杂等缺陷提出了一种新颖的基于家族基因的网格信任模型。该模型解决了传统信任模型存在的问题,如采用将用户的全部身份信息放在用户的基因里方法解决了证书主体信息的不明确,用基因检测解决了认证过程的复杂,用基因指派解决了访问控制的繁琐,并且给出了网格家族、家族基因、基因指派、基因鉴别和信任等概念,并建立了模型的形式化描述,理论分析和实验结果表明这种模型是网格信任领域一种较好的解决方案。     

The Effects of Insulin-Like Growth Factor I and BTP-2 on Acute Lung Injury

Acute lung injury (ALI) afflicts approximately 200,000 patients annually and has a 40% mortality rate. The COVID-19 pandemic has massively increased the rate of ALI incidence. The pathogenesis of ALI involves tissue damage from invading microbes and, in severe cases, the overexpression of inflammatory cytokines such as tumor necrosis factor-α (TNF-α) and interleukin-1β (IL-1β). This study aimed to develop a therapy to normalize the excess production of inflammatory cytokines and promote tissue repair in the lipopolysaccharide (LPS)-induced ALI. Based on our previous studies, we tested the insulin-like growth factor I (IGF-I) and BTP-2 therapies. IGF-I was selected, because we and others have shown that elevated inflammatory cytokines suppress the expression of growth hormone receptors in the liver, leading to a decrease in the circulating IGF-I. IGF-I is a growth factor that increases vascular protection, enhances tissue repair, and decreases pro-inflammatory cytokines. It is also required to produce anti-inflammatory 1,25-dihydroxyvitamin D. BTP-2, an inhibitor of cytosolic calcium, was used to suppress the LPS-induced increase in cytosolic calcium, which otherwise leads to an increase in proinflammatory cytokines. We showed that LPS increased the expression of the primary inflammatory mediators such as toll like receptor-4 (TLR-4), IL-1β, interleukin-17 (IL-17), TNF-α, and interferon-γ (IFN-γ), which were normalized by the IGF-I + BTP-2 dual therapy in the lungs, along with improved vascular gene expression markers. The histologic lung injury score was markedly elevated by LPS and reduced to normal by the combination therapy. In conclusion, the LPS-induced increases in inflammatory cytokines, vascular injuries, and lung injuries were all improved by IGF-I + BTP-2 combination therapy.     

Real-Time Monitoring the Effect of Cytopathic Hypoxia on Retinal Pigment Epithelial Barrier Functionality Using Electric Cell-Substrate Impedance Sensing (ECIS) Biosensor Technology

Disruption of retinal pigment epithelial (RPE barrier integrity is a hallmark feature of various retinal blinding diseases, including diabetic macular edema and age-related macular degeneration, but the underlying causes and pathophysiology are not completely well-defined. One of the most conserved phenomena in biology is the progressive decline in mitochondrial function with aging leading to cytopathic hypoxia, where cells are unable to use oxygen for energy production. Therefore, this study aimed to thoroughly investigate the role of cytopathic hypoxia in compromising the barrier functionality of RPE cells. We used Electric Cell-Substrate Impedance Sensing (ECIS) system to monitor precisely in real time the barrier integrity of RPE cell line (ARPE-19) after treatment with various concentrations of cytopathic hypoxia-inducing agent, Cobalt(II) chloride (CoCl₂). We further investigated how the resistance across ARPE-19 cells changes across three separate parameters: R_b (the electrical resistance between ARPE-19 cells), α (the resistance between the ARPE-19 and its substrate), and C_m (the capacitance of the ARPE-19 cell membrane). The viability of the ARPE-19 cells and mitochondrial bioenergetics were quantified with 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyl-2H-tetrazolium bromide (MTT) assay and seahorse technology, respectively. ECIS measurement showed that CoCl₂ reduced the total impedance of ARPE-19 cells in a dose dependent manner across all tested frequencies. Specifically, the ECIS program’s modelling demonstrated that CoCl₂ affected R_b as it begins to drastically decrease earlier than α or C_m, although ARPE-19 cells’ viability was not compromised. Using seahorse technology, all three concentrations of CoCl₂ significantly impaired basal, maximal, and ATP-linked respirations of ARPE-19 cells but did not affect proton leak and non-mitochondrial bioenergetic. Concordantly, the expression of a major paracellular tight junction protein (ZO-1) was reduced significantly with CoCl_2-treatment in a dose-dependent manner. Our data demonstrate that the ARPE-19 cells have distinct dielectric properties in response to cytopathic hypoxia in which disruption of barrier integrity between ARPE-19 cells precedes any changes in cells’ viability, cell-substrate contacts, and cell membrane permeability. Such differences can be used in screening of selective agents that improve the assembly of RPE tight junction without compromising other RPE barrier parameters.     

基于MMS替代协议的变电站安全通信技术研究及应用

目前,绝大多数变电站基于IEC61850标准建设,站内通信协议主要包括制造报文规范(MMS)、面向通用对象的变电站事件(GOOSE)、采样测量值(SMV)等,在工程应用实践中逐渐暴露出一些不足与安全隐患.为此,基于MMS替代协议,重点介绍了其实现的功能与安全防护技术.在功能方面,MMS替代协议增加了多个服务,并优化了现有MMS协议的不足;在安全防护技术方面,MMS替代协议的应用层实现了基于SM2算法与调度证书的双向身份认证,杜绝未授权设备的非法接入、攻击,从协议源头杜绝设备连接的不安全性;MMS替代协议的传输层实现了基于SM2算法与调度证书的全链路加密、认证,确保传输数据报文防伪造、防抵赖、防窃取,保护数据报文的机密性、完整性.测试结果表明MMS替代协议在工程应用中具有一定的参考意义.