浅谈“水源空调“在丹东市的推广使用

本文通过对“水源空调”的介绍,结合丹东市的地质情况,分析论证其在我市推广使用的可行性。     

刮板输送机接链用千斤拉的研究

针对刮板输送机的掐链、接链问题,研制出新型掐、接链用千斤拉,介绍了该装置的结构特点和工作原理,并对使用效果进行了分析。     

电气扰动引起的弯曲振动

分析了电气扰动引起机组弯曲振动的原因.认为电气扰动可以影响联轴器的同心度和转子的弯曲.本文还分析了这种振动的特点和处理措施.     

Synthesis and applications of fluoroalkyl end-capped cooligomers having fullerene and mixed fullerenes in the main chain

Fluoroalkanoyl peroxides reacted with FULLERENES [fullerene (C₆₀) and commercially available fullerenes (Nanom Mix^TR and Nanom Black^TR)] and radical polymerizable comonomers such as acrylic acid, N,N-dimethylacrylamide, N-(1,1-dimethyl-3-oxobutyl)acrylamide, and acryloylmorpholine to afford fluoroalkyl end-capped cooligomers having FULLERENES in the main chain under very mild conditions. Fluoroalkyl end-capped Nanom Mix and Nanom Black cooligomers thus obtained were found to exhibit a similar solubility to that of the corresponding fluoroalkyl end-capped cooligomers having fullerene in the main chain. These fluorinated FULLERENES cooligomers were found to form the nanometer size-controlled self-assembled cooligomeric aggregates in aqueous solutions. These fluoroalkyl end-capped FULLERENES cooligomers were more effective for solubilizing fullerene, Nanom Mix and Nanom Black into water, compared to those of the corresponding fluoroalkyl end-capped homooligomers having no FULLERENES in the main chain. Fluoroalkyl end-capped fullerene- and Nanom Mix-acrylic acid cooligomers were found to exhibit fluorescence spectra related to fullerene and Nanom Mix in cooligomers, respectively, in aqueous solutions. Additionally, these fluorinated fullerene- and Nanom Mix-acrylic acid cooligomers were able to increase chemiluminescence intensity related to luminol, effectively, compared to the corresponding fluorinated acrylic acid homooligomers.     

小型压裂技术的应用

在加砂压裂施工前进行小规模压裂，能使加砂压裂更加有效。通过对近两年来胜利油田应用小型压裂指导压裂设计的4口井实例的总结，说明了为确保加砂压裂施工的成功，尤其对端部脱砂压裂设计，小型压裂必不可少。同时介绍了Nolte方法和步骤，及小型压裂为加砂压裂设计提供的必要参数，讨论了参数解释的影响因素。为配合小型压裂工作，全面提高压裂技术水平，应选部分井进行岩石力学参数解释工作、适当延长压降测量时间，并测出瞬时停泵压力等。     

塔格糖制备方法研究进展

塔格糖是一种天然的低能量填充型甜味剂，并具有抑制高血糖、改善肠道菌群和不致龋齿等多种生理功效。目前主要通过生物和化学两种方法合成。     

Heterogeneous object modeling: A review

A review on the recent development in heterogeneous object modeling is provided in this paper. General problems and prevalent solutions to the modeling of heterogeneous objects are investigated. Vigorous heterogeneous object representations are roughly classified into three major categories: evaluated models, unevaluated models and composite models. We reveal their similarities and study their strengths and weaknesses in terms of the representational capacities, intuitiveness, exactness, compactness, efficiency and other criteria. Different design paradigms are briefly reviewed to provide an overview of the state-of-the-art in heterogeneous object design. Finally, open problems and possible future directions are discussed.     

基于Mega8单片机和AD9833的正弦波信号发生器

正弦波信号作为一种基准信号广泛应用于各种电路中，产生正弦波信号的方法有很多种，DDS（Direct Digital Frequency Synthesis直接数字频率舍成器）作为一种新型技术，具有相对带宽宽，频率转换时间短，频率分辨率高，控制灵活方便等优点，成为现代频率合成技术中的佼佼者。AD公司生产的AD9833是一款低功耗、可编程波形发生器，在Mega8单片机的控制下可生成0-12．5MHz的正弦波，还可以生成三角波和方波。经现场验证，基于Mega8单片机和AD9833的正弦波信号发生器，可以非常方便地生成各种频率的正弦波。     

Role of TRPA1 in Tissue Damage and Kidney Disease

TRPA1, a nonselective cation channel, is expressed in sensory afferent that innervates peripheral targets. Neuronal TRPA1 can promote tissue repair, remove harmful stimuli and induce protective responses via the release of neuropeptides after the activation of the channel by chemical, exogenous, or endogenous irritants in the injured tissue. However, chronic inflammation after repeated noxious stimuli may result in the development of several diseases. In addition to sensory neurons, TRPA1, activated by inflammatory agents from some non-neuronal cells in the injured area or disease, might promote or protect disease progression. Therefore, TRPA1 works as a molecular sentinel of tissue damage or as an inflammation gatekeeper. Most kidney damage cases are associated with inflammation. In this review, we summarised the role of TRPA1 in neurogenic or non-neurogenic inflammation and in kidney disease, especially the non-neuronal TRPA1. In in vivo animal studies, TRPA1 prevented sepsis-induced or Ang-II-induced and ischemia-reperfusion renal injury by maintaining mitochondrial haemostasis or via the downregulation of macrophage-mediated inflammation, respectively. Renal tubular epithelial TRPA1 acts as an oxidative stress sensor to mediate hypoxia–reoxygenation injury in vitro and ischaemia–reperfusion-induced kidney injury in vivo through MAPKs/NF-kB signalling. Acute kidney injury (AKI) patients with high renal tubular TRPA1 expression had low complete renal function recovery. In renal disease, TPRA1 plays different roles in different cell types accordingly. These findings depict the important role of TRPA1 and warrant further investigation.