Pressure effects on the low temperature state of the α-(BEDT-TTF)2MHg(SCN)4 organic conductor family

Systematic magnetotransport measurements as a function of hydrostatic pressure have been made on four members of the α-(BEDT-TTF)₂MHg(SCN)₄ organic conductor family (M = K, Tl, Rb, NH₄). Applied pressure above about 6 Kbar removes the density wave state for M = K, Tl, and Rb. For M = NH₄ the superconducting state is removed with pressure as dT_c/dP = − 0.25 K/Kbar. In all cases the Shubnikov de Haas oscillation frequency increases with pressure, including the β orbit (which involves the entire Brillouin zone), and new orbits involving very small fractions of the Fermi surface are formed.     

Voltage-pulse driven harmonic resonant rail drivers for low-power applications

We describe a new design technique for efficient harmonic resonant rail drivers. The proposed circuit implementation is coupled to a standard pulse source and uses only discrete passive components and no external dc power supply. It can thus be externally tuned to minimize the consumed power in the target IC. A new design technique based on current-fed voltage pulse-forming network theory is proposed to find the value of each discrete component for a target frequency and a given load capacitance. The proposed circuit topology can be used to generate any desired periodic 50% duty-cycle waveform by superimposing multiple harmonics of the desired waveform, however, this paper focuses on the generation of trapezoidal-wave clock signals. We have tested the driver with a capacitive load between 38.3 and 97.8 pF with clock frequency ranging between 0.8 and 15 MHz. The overall power dissipation for our second-order harmonic rail driver is 19% of fC/sub L/V/sup 2/ at 15 MHz and 97.8 pF load.     

A low-power microprocessor based on resonant energy

We describe AC-1, a CMOS microprocessor that derives most of its operating power from the clock signals rather than from dc supplies. Clock-powered circuit elements are selectively used to drive high-fan-out nodes. An inductor-based, all-resonant clock-power generator allows us to recover 85% of the clock-drive energy. The measured top frequency for the microprocessor was 58.8 MHz at 26.2 mW. The resulting overall decrease in dissipation ranges from four to five times at clock frequencies from 35 to 54 MHz. We also compare the performance of the processor to a reimplementation in static logic     

The design and implementation of a low-power clock-poweredmicroprocessor

We describe the design and implementation of a 16-bit clock-powered microprocessor that dissipates only 2.9 mW at 15.8 MHz based on laboratory measurements. Clock-powered logic (CPL) has been developed as a new approach for designing and building low-power VLSI systems that exploit the benefits of supply-voltage-scaled static CMOS and energy-recovery CMOS techniques. In CPL, the clock signals are a source of ac power for the other large on-chip capacitive loads. Clock amplitude and waveform shape combine to reduce power. By exploiting energy recovery and an energy-conserving clock driver, it is possible to build ultra-low-power CMOS processors with this approach. We compare the CPL approach with a conventional, fully dissipative approach for a processor with a similar ISA and VLSI architecture which was designed using the same set of VLSI CAD tools. The simulation results indicate that the CPL microprocessor would dissipate 40% less power than the conventional design     

Low-power digital systems based on adiabatic-switching principles

Adiabatic switching is an approach to low-power digital circuits that differs fundamentally from other practical low-power techniques. When adiabatic switching is used, the signal energies stored on circuit capacitances may be recycled instead of dissipated as heat. We describe the fundamental adiabatic amplifier circuit and analyze its performance. The dissipation of the adiabatic amplifier is compared to that of conventional switching circuits, both for the case of a fixed voltage swing and the case when the voltage swing can be scaled to reduce power dissipation. We show how combinational and sequential adiabatic-switching logic circuits may be constructed and describe the timing restrictions required for adiabatic operation. Small chip-building experiments have been performed to validate the techniques and to analyse the associated circuit overhead     

Angular dependence of the high field phase transition in α-(BEDT-TTF)2TlHg(SCN)4

We report results of a systematic study of the anomalous low temperature state (LTS) angular magnetoresistance (MR) in the quasi-two dimensional organic conductor α-(BEDT-TTF)₂TlHg(SCN)₄ in fields up to 30 tesla. The high field suppression of the LTS is found to be independent of angle, while the MR maximum, H_max increases with angle in conjunction with the well studied angular dependent magnetoresistance oscillations (ADMRO). The different magnetic breakdown probabilities above and below H_max are found to be the source of a novel high field change in the ADMRO shape.     

The metabotropic glutamate receptors of the vestibular organs

This research sought to test the presence and function of metabotropic excitatory amino acid receptors (mGluR) in the frog semicircular canal (SCC). The mGluR agonist +/- 1-aminocyclopentane-trans-1,3-dicarboxylate (ACPD) produced an increase in afferent firing rates of the ampullar nerve of the intact posterior canal. This increase was not due to a stimulation of cholinergic efferent terminals or the acetylcholine (ACh) receptor, since atropine, in concentrations which blocked the response to exogenous acetylcholine, did not affect the response to ACPD. Likewise, ACPD effects were not due to stimulation of postsynaptic NMDA receptors, since the NMDA antagonist D(-)-2-amino-5-phosphonopentanoate (AP-5) did not affect the response to ACPD, reinforcing the reported selectivity of ACPD for mGluRs. When the SCC was superfused with artificial perilymph known to inhibit hair cell transmitter release (i.e. low Ca-high Mg), ACPD failed to increase afferent firing. This suggests that the receptor activated by ACPD is located on the hair cell. Pharmacological evidence suggested that the mGluRs involved in afferent facilitation belong to Group I (i.e. subtypes 1 and 5). In fact, the Group III agonist AP-4 had no effect, and the ACPD facilitatory effect was blocked by the Group I mGluR antagonists (S)-4-carboxyphenylglycine (CPG) and (RS)-1-aminoindan-1,5-dicarboxylic acid (AIDA). Additional pharmacological evidence supported the presence of Group I mGluRs. Interestingly, the mGluR antagonists, AIDA and 4CPG, by themselves did not affect the resting firing rates of ampullar afferents. This may suggest that the mGluRs are not involved in resting activity but perhaps only in evoked activity (as suggested in Guth et al. (1991) Hear. Res. 56, 69-78). In addition, the mRNA for the mGluR1 has been detected in hair cells of both SCC, utricle, and saccule. In summary, the evidence points to an mGluR localized to the hair cell (i.e. an autoreceptor) which may be activated to produce a positive feedback augmentation of evoked but not resting transmitter release and thus affect afferent activity.