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Nazir Mohsin Sabah Aneeqa Sarwar Sana Yaseen Azeema Jurcut Anca 《Wireless Personal Communications》2021,119(4):3529-3552
Wireless Personal Communications - Present-day wireless methods are necessary to support a variety of higher-speed data communication facilities for its subscribers such as cloud-based video... 相似文献
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Braeken An Liyanage Madhusanka Jurcut Anca Delia 《Wireless Personal Communications》2019,106(2):345-364
Wireless Personal Communications - The Internet of Things (IoT) technologies interconnect a broad range of network devices, differing in terms of size, weight, functionality, and resource... 相似文献
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Nazir Mohsin Sabah Aneeqa Sarwar Sana Yaseen Azeema Jurcut Anca 《Wireless Personal Communications》2021,116(4):3553-3566
Wireless Personal Communications - Relying on the fact that, cascaded $$\alpha$$ – $$\mu$$ distribution provide simplified and excellent fitting to the measurement data compared to the other... 相似文献
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Laura Damian Cristian Cezar Login Carolina Solomon Cristina Belizna Svetlana Encica Laura Urian Ciprian Jurcut Bogdan Stancu Romana Vulturar 《International journal of molecular sciences》2022,23(13)
Inclusion body myositis (IBM) is an acquired, late-onset inflammatory myopathy, with both inflammatory and degenerative pathogenesis. Although idiopathic inflammatory myopathies may be associated with malignancies, IBM is generally not considered paraneoplastic. Many studies of malignancy in inflammatory myopathies did not include IBM patients. Indeed, IBM is often diagnosed only after around 5 years from onset, while paraneoplastic myositis is generally defined as the co-occurrence of malignancy and myopathy within 1 to 3 years of each other. Nevertheless, a significant association with large granular lymphocyte leukemia has been recently described in IBM, and there are reports of cancer-associated IBM. We review the pathogenic mechanisms supposed to be involved in IBM and outline the common mechanisms in IBM and malignancy, as well as the therapeutic perspectives. The terminally differentiated, CD8+ highly cytotoxic T cells expressing NK features are central in the pathogenesis of IBM and, paradoxically, play a role in some cancers as well. Interferon gamma plays a central role, mostly during the early stages of the disease. The secondary mitochondrial dysfunction, the autophagy and cell cycle dysregulation, and the crosstalk between metabolic and mitogenic pathways could be shared by IBM and cancer. There are intermingled subcellular mechanisms in IBM and neoplasia, and probably their co-existence is underestimated. The link between IBM and cancers deserves further interest, in order to search for efficient therapies in IBM and to improve muscle function, life quality, and survival in both diseases. 相似文献
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