从7-ADCA生产过程中环境友好的回收苯乙酸

采用新工艺回收7-氨基-3-脱乙酰氧基头孢烷酸(7-ADCA)生产中产生的苯乙酸,并对苯乙酸回收过程中产生的废硫酸进行处理。采用硫酸(9 8%)洗涤二氯甲烷,硫酸与二氯甲烷适宜的体积比为1∶50,洗涤好的二氯甲烷中含苯乙酸4%。蒸馏出二氯甲烷,趁热向熔融苯乙酸中加入母液,降温析出苯乙酸。晾干的苯乙酸为白色鳞片状,含量大于99%,苯乙酸母液补充部分清水套用到下一批析出苯乙酸。废浓硫酸用芬顿试剂氧化,控制80℃氧化4h,双氧水和七水硫酸亚铁重量比为6∶1,废浓硫酸COD下降85%以上。氧化结束后向废硫酸中加入铁粉和双氧水,制得聚合硫酸铁。     

对海水进行混凝除浊预处理研究

介绍采用聚合氯化铁（PFC）为絮凝剂，对海水进行混凝除浊预处理方法。考察了原海水pH、搅拌速度、反应时间及PFC用量对海水预处理效果的影响。结果表明，在原海水pH凋至9．0，搅拌速度控制在200min^-1，反应时间选择8—10min，PFC用量0．10μg／g的最佳条件下，能使处理后的海水浊度降至0．05mol／L，而联产的酸性废水中和剂浆料中Mg（OH）2含量在25％～45％，大肠菌群的去除率大于89．3％。     

用硫酸铁铵容量法测定钛精矿中二氧化钛含量

样品在过氧化钠熔融、水浸取及盐酸酸化后，在盐酸和硫酸介质中，隔绝空气，用金属铝将钛(Ⅳ)还原至钛(Ⅲ)，以硫氰酸盐为指示剂，用硫酸铁铵标准溶液滴定。     

氯化铁催化合成富马酸二甲酯

氯化铁可代替硫酸催化酯化反应，探讨了氯化铁催化合成富马酸二甲酯的条件，操作简便，反应温和，无腐蚀，减少了污染与产品纯化。     

ICAP-AES等离子体发射光谱法测定氧化铁粉的杂质元素

介绍了ICP等离子体光谱仪的原理及测定高纯氧化铁中铝、钙、硫、钾、镁、锰、钠和钛等8种杂质元素的分析方法，考察了样品的溶解方案及工作曲线的选择，确定了仪器最佳工作条件和方法检出限。     

中国软磁铁氧体用氧化铁

介绍了中国软磁铁氧体用氧化铁的发展历史和现状，预测了未来的市场需求，并探讨了氧化铁的应用技术问题。     

煤矸石制备聚合氯化铝铁钙与应用试验研究

以煤矸石为原料,经过高温焙烧、酸浸、聚合、熟化等过程制备了高效无机高分子絮凝剂聚合氯化铝铁钙（PAFCC）,通过单因素试验研究了PAFCC制备条件对浊度、COD和UV₂₅₄去除率的影响。试验结果表明,PAFCC最佳聚合条件为pH值2、聚合温度60℃、聚合时间5 h、在40℃下熟化28 h。混凝结果表明：制备的PAFCC对浊度有极好的去除效果,达95.70%,同时,对COD、UV₂₅₄也有一定的去除效果,去除率分别达到47.51%、45.98%。炼化废水混凝结果表明：PAFCC对浊度和总磷有较好的去除效果,同时,对COD及氨氮也有一定的去除效果,效果明显优于传统PAC。     

Ethylene and Nitric Oxide Involvement in the Regulation of Fe and P Deficiency Responses in Dicotyledonous Plants

Iron (Fe) and phosphorus (P) are two essential elements for plant growth. Both elements are abundant in soils but with poor availability for plants, which favor their acquisition by developing morphological and physiological responses in their roots. Although the regulation of the genes related to these responses is not totally known, ethylene (ET) and nitric oxide (NO) have been involved in the activation of both Fe-related and P-related genes. The common involvement of ET and NO suggests that they must act in conjunction with other specific signals, more closely related to each deficiency. Among the specific signals involved in the regulation of Fe- or P-related genes have been proposed Fe-peptides (or Fe ion itself) and microRNAs, like miR399 (P), moving through the phloem. These Fe- or P-related phloem signals could interact with ET/NO and confer specificity to the responses to each deficiency, avoiding the induction of the specific responses when ET/NO increase due to other nutrient deficiencies or stresses. Besides the specificity conferred by these signals, ET itself could confer specificity to the responses to Fe- or P-deficiency by acting through different signaling pathways in each case. Given the above considerations, there are preliminary results suggesting that ET could regulate different nutrient responses by acting both in conjunction with other signals and through different signaling pathways. Because of the close relationship among these two elements, a better knowledge of the physiological and molecular basis of their interaction is necessary to improve their nutrition and to avoid the problems associated with their misuse. As examples of this interaction, it is known that Fe chlorosis can be induced, under certain circumstances, by a P over- fertilization. On the other hand, Fe oxides can have a role in the immobilization of P in soils. Qualitative and quantitative assessment of the dynamic of known Fe- and P-related genes expression, selected ad hoc and involved in each of these deficiencies, would allow us to get a profound knowledge of the processes that regulate the responses to both deficiencies. The better knowledge of the regulation by ET of the responses to these deficiencies is necessary to properly understand the interactions between Fe and P. This will allow the obtention of more efficient varieties in the absorption of P and Fe, and the use of more rational management techniques for P and Fe fertilization. This will contribute to minimize the environmental impacts caused by the use of P and Fe fertilizers (Fe chelates) in agriculture and to adjust the costs for farmers, due to the high prices and/or scarcity of Fe and P fertilizers. This review aims to summarize the latest advances in the knowledge about Fe and P deficiency responses, analyzing the similarities and differences among them and considering the interactions among their main regulators, including some hormones (ethylene) and signaling substances (NO and GSNO) as well as other P- and Fe-related signals.