基于开源平台的网络镜像部署方案

介绍了一种基于开源平台的网络镜像部署方案。方案基于C／S架构,客户端的安装采用了基于PXE的远程启动模式,因此不需要为客户端进行手动安装,从而可以使无系统客户机也能向服务端发起镜像部署请求。网络镜像部署的实现充分吸纳了UDPcast、Partclone以及tftpd32等优秀的已有开源项目,避免了重复的开发工作,编写的调度、UI模块解决了开源软件操作不友好的缺点。实际测试显示,该方案己具有良好的实用价值,所用的系统架构也值得推广到其他应用场合。     

基于GA-RBF模型的无线电波信号预测研究

随着通信行业的发展,无线电波信号的预测问题成为科学研究的重要课题。因此研究了一种基于遗传算法优化径向基神经网络参数的无线电波信号预测模型,通过实验表明,这种经过遗传算法优化的模型比单纯的RBF网络模型预测精度更高,运行更稳定。     

一种基于PXE技术的计算机硬件网络检测方法

给出了一种基于PXE远程启动技术的计算机硬件检测方法,可用于计算机实验室中进行远程计算机硬件检测,在一定程度上提高了实验室的管理效率。     

Fibroblast protein profile analysis highlights the role of oxidative stress and vitamin K recycling in the pathogenesis of pseudoxanthoma elasticum

Pseudoxanthoma elasticum (PXE) is a genetic disorder associated to mutations in the ABCC6 gene; however, the pathogenetic mechanisms leading to elastic fibre calcifications and to clinical manifestations are still unknown. Dermal fibroblasts, directly involved in the production of the extracellular milieu, have been isolated from healthy subjects and from patients affected by PXE, cultured in vitro and characterized for their ability to produce reactive oxygen species, for structural and functional properties of their cell membranes, for changes in their protein profile. Data demonstrate that oxidative stress has profound and endurable consequences on PXE fibroblast phenotype being responsible for: reduced levels of global DNA methylation, increased amount of carbonylated proteins and of lipid peroxidation products, altered structural properties of cell membranes, modified protein expression. Data shed new light on the pathogenetic pathways in PXE, by identifying a network of proteins affecting elastic fibre calcification through inefficient vitamin K recycling, and highlight the role of differentially expressed proteins as targets for validating the efficacy of future therapeutic strategies aiming to delay and/or revert the pathologic phenotype of PXE fibroblasts. Moreover, data open new perspectives for investigating PXE-like phenotypes in the absence of ABCC6 mutations.     

基于PXE技术的计算机实验室软件系统架设

PXE作为一种网络启动技术,目前得到了广泛的应用。本文介绍了PXE技术的工作原理,结合Symantec Ghost 10.0企业版功能,阐明利用PXE技术实现计算机实验室软件系统架设及维护更新的具体过程。     

基于PXE和ghost多播技术的网络克隆

该文介绍了利用GHOST7.5进行网络多播克隆的方法与技巧,论述了如何通过利用Ghost7.5企业版的网络远程功能,将DHCP服务和TFTP文件传输与PXE网络启动功能相结合,实现了局域网内计算机系统在无软驱和光驱情况下的网络多播克隆.     

基于WINDOWS 2000的WIN98 PXE网络无盘工作站

一部机器通过网络启动,它必须得到这些信息:一个identity;某个操作系统的映像文件;可以操作的文件系统。工作站接收到完整的启动文件后,开始执行文件中的启动程序,将执行点转向启动块的入口,启动工作站。这时利用TFTP进入OS引导方式。     

Ghost软件在网络中的应用

根椐当前操作系统庞大、安装时间长、计算机系统维护繁杂等特点，本文通过PXE网络硬盘克隆方法和GhostCast网络硬盘克隆方法详细的介绍了网络克隆软件ghost的使用方法和技巧。     

基于PXE协议的Linux自动安装原理分析与应用

为解决在局域网环境下快速大规模部署Linux服务器的现实需求问题,文章分析了Linux网络安装过程中所使用的相关技术原理,包括预引导执行环境、引导协议、动态主机配置协议、网络文件系统及KickStart等,利用以上技术在局域网环境下实现了RHEL AS4的自动化网络安装.该方法可以快速并发完成Linux服务器的部署工作,减少工作量,提高工作效率.     

Structural and Functional Characterization of the ABCC6 Transporter in Hepatic Cells: Role on PXE,Cancer Therapy and Drug Resistance

Pseudoxanthoma elasticum (PXE) is a complex autosomal recessive disease caused by mutations of ABCC6 transporter and characterized by ectopic mineralization of soft connective tissues. Compared to the other ABC transporters, very few studies are available to explain the structural components and working of a full ABCC6 transporter, which may provide some idea about its physiological role in humans. Some studies suggest that mutations of ABCC6 in the liver lead to a decrease in some circulating factor and indicate that PXE is a metabolic disease. It has been reported that ABCC6 mediates the efflux of ATP, which is hydrolyzed in PPi and AMP; in the extracellular milieu, PPi gives potent anti-mineralization effect, whereas AMP is hydrolyzed to Pi and adenosine which affects some cellular properties by modulating the purinergic pathway. Structural and functional studies have demonstrated that silencing or inhibition of ABCC6 with probenecid changed the expression of several genes and proteins such as NT5E and TNAP, as well as Lamin, and CDK1, which are involved in cell motility and cell cycle. Furthermore, a change in cytoskeleton rearrangement and decreased motility of HepG2 cells makes ABCC6 a potential target for anti-cancer therapy. Collectively, these findings suggested that ABCC6 transporter performs functions that modify both the external and internal compartments of the cells.