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W. O. SAXTON 《Journal of microscopy》1995,179(2):201-213
Least-squares fitting methods are described, currently the most accurate known, for determining the imaging aberrations and other important parameters. The data required are either the image displacement or the image diffractogram shape (or both), as a function of injected beam tilt; the analysis is largely common to the two cases. Explicit solutions are given for some simple cases (e.g. coma-free alignment on the basis of three images only) and some promising developments are considered; the relevant aberration theory is set out in detail. Experimental work reported in a companion paper has confirmed the presence of threefold astigmatism at significant levels, but has not shown any evidence of other higher order aberrations. 相似文献
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Dogukan A Aygen B Berilgen MS Dag S Bektas S Gunal AI 《Hemodialysis international. International Symposium on Home Hemodialysis》2006,10(2):168-169
We describe a 60-year-old woman who became comatose after a single dose of gabapentin for right-sided sciatalgia. The patient was improved by hemodialysis. Gabapentin toxicity should be considered when mental status changes develop in patients with renal failure after even a single dose. 相似文献
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利用Richards-Wolf矢量衍射积分公式,获得矢量偏振贝塞耳-高斯光束经具有初级慧差的高数值孔径系统聚焦后的三维光场复振幅函数,模拟了不同慧差系数下聚焦光场的纵向分布,以及焦平面和光轴上的光强。研究表明,初级慧差的存在导致矢量偏振贝塞耳-高斯光束的会聚光场发生偏移和变形,焦平面光强的分布和光轴上的光强峰值都受初级慧差和入射光偏振态的共同影响,偏振态和初级慧差不影响聚焦光场在光轴上的对称分布。 相似文献
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星间光通信中,波前畸变是影响信号光远场特性的主要因素。本文分析了球差和彗差对信号光远场分布、天线接收光功率的影响,引入了畸变衰减因子,给出了畸变衰减因子与波前畸变均方根值的关系。理论分析和数值仿真结果表明,球差和彗差畸变越大,对天线接收功率的影响越大。在天线对准的条件下,当彗差和球差畸变均方根值分别约为0.120λ和0.135λ时,天线的接收功率为无畸变时的1/2。彗差对天线接收功率的影响主要是峰值移动造成的,球差对天线接收功率的影响主要是次峰的增强造成的。 相似文献
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星间光通信中,波前畸变是影响信号光远场特性的主要因素。分析了球差和彗差对信号光远场分布、天线接收光功率的影响,引入了畸变衰减因子,给出了畸变衰减因子与波前畸变均方根(RMS)值的关系。理论分析和数值仿真结果表明,球差和彗差畸变越大,对天线接收功率的影响越大。在天线对准的条件下,当彗差和球差畸变均方根值分别约为0.120λ和0.135λ时,天线的接收功率为无畸变时的1/2。彗差对天线接收功率的影响主要是峰值移动造成的,球差对天线接收功率的影响主要是次峰的增强造成的。 相似文献
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Iyer H Sen M Prasad C Rupar CA Lindsay RM 《Hemodialysis international. International Symposium on Home Hemodialysis》2012,16(1):95-100
Urea cycle disorders are an important and treatable cause of hyperammonemia in the newborn and pediatric age group. Presentation in adolescence or adult life is rare and can manifest as frequent vomiting and behavioral changes. An inherited metabolic disorder should be considered in adults with obvious or occult encephalopathy. Failure to diagnose and treat rapidly may lead to irreversible neuronal damage. An improved understanding of the diagnosis and management of late-onset urea cycle disorders is needed to assist nephrologists in providing optimal care. This report describes the clinical characteristics of a young man with first presentation of hyperammonemia in adult life. 相似文献